Unravelling the Molecular Determinants of Bee Sensitivity to Neonicotinoid Insecticides
暂无分享,去创建一个
Bartlomiej J. Troczka | R. Nauen | M. Williamson | T. Davies | L. Field | K. S. Singh | B. Lueke | C. Bass | C. Zimmer | J. Benting | G. Hertlein | R. A. Homem | B. Troczka | R. Reid | Cristina Manjón | M. Kohler | K. Beadle | Marion Zaworra | E. Randall | Laura Kor | Rafael A. Homem | Emma Randall
[1] Y. Nomura,et al. Molecular basis of selective resistance of the bumblebee BiNav1 sodium channel to tau-fluvalinate , 2017, Proceedings of the National Academy of Sciences.
[2] D. Cressey. The bitter battle over the world’s most popular insecticides , 2017, Nature.
[3] L. Field,et al. Induced thiacloprid insensitivity in honeybees (Apis mellifera L.) is associated with up‐regulation of detoxification genes , 2016, Insect molecular biology.
[4] Brian R Johnson,et al. Forager bees (Apis mellifera) highly express immune and detoxification genes in tissues associated with nectar processing , 2015, Scientific Reports.
[5] M. Berenbaum,et al. Task‐related differential expression of four cytochrome P450 genes in honeybee appendages , 2015, Insect molecular biology.
[6] M. Berenbaum,et al. Xenobiotic detoxification pathways in honey bees. , 2015, Current opinion in insect science.
[7] Erich Bornberg-Bauer,et al. The genomes of two key bumblebee species with primitive eusocial organization , 2015, Genome Biology.
[8] Reed M. Johnson. Honey bee toxicology. , 2015, Annual review of entomology.
[9] M. Schwab,et al. Cytochrome P450 enzymes in drug metabolism: regulation of gene expression, enzyme activities, and impact of genetic variation. , 2013, Pharmacology & therapeutics.
[10] Blair D. Siegfried,et al. Acaricide, Fungicide and Drug Interactions in Honey Bees (Apis mellifera) , 2013, PloS one.
[11] Isabel Gómez,et al. Evolution of Bacillus thuringiensis Cry toxins insecticidal activity , 2013, Microbial biotechnology.
[12] M. Paine,et al. Directionally selected cytochrome P450 alleles are driving the spread of pyrethroid resistance in the major malaria vector Anopheles funestus , 2012, Proceedings of the National Academy of Sciences.
[13] R. Scheiner,et al. Suitability of three common reference genes for quantitative real-time PCR in honey bees , 2012, Apidologie.
[14] M. Berenbaum,et al. CYP9Q-mediated detoxification of acaricides in the honey bee (Apis mellifera) , 2011, Proceedings of the National Academy of Sciences.
[15] Andrew J. Crossthwaite,et al. Mutation of a nicotinic acetylcholine receptor β subunit is associated with resistance to neonicotinoid insecticides in the aphid Myzus persicae , 2011, BMC Neuroscience.
[16] A. Cristino,et al. Validation of reference genes for gene expression studies in the honey bee, Apis mellifera, by quantitative real-time RT-PCR , 2008, Apidologie.
[17] Jeffrey G. Scott,et al. Is Apis mellifera more sensitive to insecticides than other insects? , 2010, Pest management science.
[18] S. R. Palli,et al. A brain-specific cytochrome P450 responsible for the majority of deltamethrin resistance in the QTC279 strain of Tribolium castaneum , 2010, Proceedings of the National Academy of Sciences.
[19] I. Pichová,et al. Selection of reference genes for real-time polymerase chain reaction analysis in tissues from Bombus terrestris and Bombus lucorum of different ages. , 2010, Analytical biochemistry.
[20] M. Gauthier. State of the art on insect nicotinic acetylcholine receptor function in learning and memory. , 2010, Advances in experimental medicine and biology.
[21] J. F. Kenneke,et al. Mechanistic investigation of the noncytochrome P450-mediated metabolism of triadimefon to triadimenol in hepatic microsomes. , 2008, Chemical research in toxicology.
[22] N. Perrimon,et al. Exploiting position effects and the gypsy retrovirus insulator to engineer precisely expressed transgenes , 2008, Nature Genetics.
[23] May R Berenbaum,et al. Molecular mechanisms of metabolic resistance to synthetic and natural xenobiotics. , 2007, Annual review of entomology.
[24] R. Feyereisen,et al. Evolution of insect P450. , 2006, Biochemical Society transactions.
[25] D. Boykin,et al. CYP4F Enzymes Are the Major Enzymes in Human Liver Microsomes That Catalyze the O-Demethylation of the Antiparasitic Prodrug DB289 [2,5-Bis(4-amidinophenyl)furan-bis-O-methylamidoxime] , 2006, Drug Metabolism and Disposition.
[26] M. Berenbaum,et al. A deficit of detoxification enzymes: pesticide sensitivity and environmental response in the honeybee , 2006, Insect molecular biology.
[27] D. Sattelle,et al. Role in the Selectivity of Neonicotinoids of Insect-Specific Basic Residues in Loop D of the Nicotinic Acetylcholine Receptor Agonist Binding Site , 2006, Molecular Pharmacology.
[28] J. Schenkman,et al. Spectral analyses of cytochromes P450. , 2006, Methods in molecular biology.
[29] J. Brunet,et al. In vivo metabolic fate of [14C]-acetamiprid in six biological compartments of the honeybee, Apis mellifera L. , 2005, Pest management science.
[30] L. Belzunces,et al. In vivo distribution and metabolisation of 14C-imidacloprid in different compartments of Apis mellifera L. , 2004, Pest management science.
[31] M. Williamson,et al. Identification of mutations conferring insecticide‐insensitive AChE in the cotton‐melon aphid, Aphis gossypii Glover , 2004, Insect molecular biology.
[32] J. Ambrose,et al. Mechanism for the differential toxicity of neonicotinoid insecticides in the honey bee, Apis mellife , 2004 .
[33] R. Schmuck. Ecotoxicological profile of the insecticide thiacloprid , 2001 .
[34] R. Nauen,et al. Toxicity and nicotinic acetylcholine receptor interaction of imidacloprid and its metabolites in Apis mellifera (Hymenoptera: Apidae). , 2001, Pest management science.
[35] M. Pfaffl,et al. A new mathematical model for relative quantification in real-time RT-PCR. , 2001, Nucleic acids research.