CALCOCO2/NDP52 and SQSTM1/p62 differentially regulate coxsackievirus B3 propagation
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Honglin Luo | Huitao Liu | Y. Mohamud | Y. Xue | Haoyu Deng | Junyan Qu
[1] W. Jackson,et al. Enteroviruses Remodel Autophagic Trafficking through Regulation of Host SNARE Proteins to Promote Virus Replication and Cell Exit. , 2018, Cell reports.
[2] Honglin Luo,et al. Enteroviral Infection Inhibits Autophagic Flux via Disruption of the SNARE Complex to Enhance Viral Replication. , 2018, Cell reports.
[3] Honglin Luo,et al. N-Terminomics TAILS Identifies Host Cell Substrates of Poliovirus and Coxsackievirus B3 3C Proteinases That Modulate Virus Infection , 2018, Journal of Virology.
[4] J. Cui,et al. Tetherin Suppresses Type I Interferon Signaling by Targeting MAVS for NDP52-Mediated Selective Autophagic Degradation in Human Cells. , 2017, Molecular cell.
[5] A. Ballabio,et al. Molecular definitions of autophagy and related processes , 2017, The EMBO journal.
[6] L. Tafforeau,et al. Influenza virus protein PB1-F2 interacts with CALCOCO2 (NDP52) to modulate innate immune response. , 2017, The Journal of general virology.
[7] R. Mahieux,et al. Distinct Contributions of Autophagy Receptors in Measles Virus Replication , 2017, Viruses.
[8] J. Carette,et al. PLA2G16 represents a switch between entry and clearance of Picornaviridae , 2017, Nature.
[9] V. Chow,et al. Enterovirus 71 infection of motor neuron-like NSC-34 cells undergoes a non-lytic exit pathway , 2016, Scientific Reports.
[10] C. Behl,et al. Ubiquitin-Dependent And Independent Signals In Selective Autophagy. , 2016, Trends in cell biology.
[11] C. Coyne,et al. Unc93b Induces Apoptotic Cell Death and Is Cleaved by Host and Enteroviral Proteases , 2015, PloS one.
[12] J. Burman,et al. The ubiquitin kinase PINK1 recruits autophagy receptors to induce mitophagy , 2015, Nature.
[13] Honglin Luo,et al. Cytoplasmic translocation, aggregation, and cleavage of TDP-43 by enteroviral proteases modulate viral pathogenesis , 2015, Cell Death and Differentiation.
[14] E. Wimmer,et al. Phosphatidylserine Vesicles Enable Efficient En Bloc Transmission of Enteroviruses , 2015, Cell.
[15] P. Lőw,et al. The Role of the Selective Adaptor p62 and Ubiquitin-Like Proteins in Autophagy , 2014, BioMed research international.
[16] Honglin Luo,et al. Dominant-negative function of the C-terminal fragments of NBR1 and SQSTM1 generated during enteroviral infection , 2014, Cell Death and Differentiation.
[17] A. Segall,et al. Coxsackievirus B Exits the Host Cell in Shed Microvesicles Displaying Autophagosomal Markers , 2014, PLoS pathogens.
[18] M. Alirezaei,et al. In Vivo Ablation of Type I Interferon Receptor from Cardiomyocytes Delays Coxsackieviral Clearance and Accelerates Myocardial Disease , 2014, Journal of Virology.
[19] L. Platanias,et al. Beta Interferon Regulation of Glucose Metabolism Is PI3K/Akt Dependent and Important for Antiviral Activity against Coxsackievirus B3 , 2014, Journal of Virology.
[20] P. Fisher,et al. Enterovirus 2Apro Targets MDA5 and MAVS in Infected Cells , 2014, Journal of Virology.
[21] David A. Scott,et al. Genome engineering using the CRISPR-Cas9 system , 2013, Nature Protocols.
[22] J. Bergelson,et al. Echovirus 7 Entry into Polarized Caco-2 Intestinal Epithelial Cells Involves Core Components of the Autophagy Machinery , 2013, Journal of Virology.
[23] L. Zitvogel,et al. Autophagy and cellular immune responses. , 2013, Immunity.
[24] Honglin Luo,et al. Cleavage of sequestosome 1/p62 by an enteroviral protease results in disrupted selective autophagy and impaired NFKB signaling , 2013, Autophagy.
[25] M. Prevost,et al. Species‐specific impact of the autophagy machinery on Chikungunya virus infection , 2013, EMBO reports.
[26] S. Bloor,et al. LC3C, Bound Selectively by a Noncanonical LIR Motif in NDP52, Is Required for Antibacterial Autophagy , 2012, Molecular cell.
[27] M. Alirezaei,et al. Pancreatic acinar cell-specific autophagy disruption reduces coxsackievirus replication and pathogenesis in vivo. , 2012, Cell host & microbe.
[28] Honglin Luo,et al. Interplay between the cellular autophagy machinery and positive-stranded RNA viruses , 2012, Acta biochimica et biophysica Sinica.
[29] W. Jackson,et al. Human Rhinovirus 2 Induces the Autophagic Pathway and Replicates More Efficiently in Autophagic Cells , 2011, Journal of Virology.
[30] P. Kim,et al. The ubiquitin-binding adaptor proteins p62/SQSTM1 and NDP52 are recruited independently to bacteria-associated microdomains to target Salmonella to the autophagy pathway , 2011, Autophagy.
[31] Elizabeth Delorme-Axford,et al. The Coxsackievirus B 3Cpro Protease Cleaves MAVS and TRIF to Attenuate Host Type I Interferon and Apoptotic Signaling , 2011, PLoS pathogens.
[32] Kay Hofmann,et al. Selective autophagy: ubiquitin-mediated recognition and beyond , 2010, Nature Cell Biology.
[33] R. Sumpter,et al. Autophagy protects against Sindbis virus infection of the central nervous system. , 2010, Cell host & microbe.
[34] S. Bloor,et al. The TBK1 adaptor and autophagy receptor NDP52 restricts the proliferation of ubiquitin-coated bacteria , 2009, Nature Immunology.
[35] Ivan Dikic,et al. A role for ubiquitin in selective autophagy. , 2009, Molecular cell.
[36] B. McManus,et al. Autophagosome Supports Coxsackievirus B3 Replication in Host Cells , 2008, Journal of Virology.
[37] Zhijian J. Chen,et al. Identification and Characterization of MAVS, a Mitochondrial Antiviral Signaling Protein that Activates NF-κB and IRF3 , 2005, Cell.
[38] K. Kirkegaard,et al. Subversion of Cellular Autophagosomal Machinery by RNA Viruses , 2005, PLoS biology.
[39] L. Reed,et al. A SIMPLE METHOD OF ESTIMATING FIFTY PER CENT ENDPOINTS , 1938 .