Mature T-cell and NK-cell lymphomas: updates on molecular genetic features
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[1] S. Ishikawa,et al. Clonal germinal center B cells function as a niche for T-cell lymphoma , 2022, Blood.
[2] A. Rosenwald,et al. The 5th edition of the World Health Organization Classification of Haematolymphoid Tumours: Lymphoid Neoplasms , 2022, Leukemia.
[3] W. Chan,et al. Genomic profiling identifies distinct genetic subtypes in extra-nodal natural killer/T-cell lymphoma , 2022, Leukemia.
[4] L. Staudt,et al. The International Consensus Classification of Mature Lymphoid Neoplasms: A Report from the Clinical Advisory Committee. , 2022, Blood.
[5] T. Noda,et al. Genomic determinants impacting the clinical outcome of mogamulizumab treatment for adult T-cell leukemia/lymphoma , 2022, Haematologica.
[6] P. Gaulard,et al. Nodal cytotoxic peripheral T-cell lymphoma occurs frequently in the clinical setting of immunodysregulation and is associated with recurrent epigenetic alterations , 2022, Modern Pathology.
[7] Peggy Wu,et al. T-Cell Lymphomas, Version 2.2022, NCCN Clinical Practice Guidelines in Oncology. , 2022, Journal of the National Comprehensive Cancer Network : JNCCN.
[8] K. Naresh,et al. Parallel evolution of two distinct lymphoid proliferations in clonal haematopoiesis , 2022, Histopathology.
[9] C. Ong,et al. Immune pathway upregulation and lower genomic instability distinguish EBV-positive nodal T/NK-cell lymphoma from ENKTL and PTCL-NOS , 2022, Haematologica.
[10] Hyunsung Kim,et al. The Pathologic and Genetic Characteristics of Extranodal NK/T-Cell Lymphoma , 2022, Life.
[11] S. Miyano,et al. Whole-genome landscape of adult T-cell leukemia/lymphoma. , 2021, Blood.
[12] Nicholas D. Ward,et al. Co-occurring rearrangements of DUSP22 and TP63 define a rare genetic subset of ALK-negative anaplastic large cell lymphoma with inferior survival outcomes , 2021, Leukemia & lymphoma.
[13] H. Iwasaki,et al. Clinical significance of TP53 mutations in adult T‐cell leukemia/lymphoma , 2021, British journal of haematology.
[14] Daniel‐Adriano Silva,et al. Super-enhancer-based identification of a BATF3/IL-2R−module reveals vulnerabilities in anaplastic large cell lymphoma , 2021, Nature Communications.
[15] F. Jardin,et al. Detection of gene fusion transcripts in Peripheral T-cell Lymphoma using a multiplexed targeted sequencing assay. , 2021, The Journal of molecular diagnostics : JMD.
[16] P. Gaulard,et al. Fusion transcripts FYN-TRAF3IP2 and KHDRBS1-LCK hijack T cell receptor signaling in peripheral T-cell lymphoma, not otherwise specified , 2021, Nature Communications.
[17] C. Ong,et al. A composite single-nucleotide polymorphism prediction signature for extranodal natural killer/T-cell lymphoma. , 2021, Blood.
[18] M. Arcila,et al. Next generation sequencing of breast implant‐associated anaplastic large cell lymphomas reveals a novel STAT3‐JAK2 fusion among other activating genetic alterations within the JAK‐STAT pathway , 2021, The breast journal.
[19] Š. Pospíšilová,et al. STAT3 and TP53 mutations associate with poor prognosis in anaplastic large cell lymphoma , 2020, Leukemia.
[20] G. Bhagat,et al. Combined Oral 5-Azacytidine and Romidepsin are Highly Effective in Patients with PTCL: A Multicenter Phase 2 Study. , 2020, Blood.
[21] V. Seshan,et al. T follicular helper phenotype predicts response to histone deacetylase inhibitors in relapsed/refractory peripheral T-cell lymphoma. , 2020, Blood advances.
[22] P. Campbell,et al. CDKN2A deletion is a frequent event associated with poor outcome in patients with peripheral T-cell lymphoma not otherwise specified (PTCL-NOS) , 2020, Haematologica.
[23] D. M. Miedema,et al. Chromosome 20 loss is characteristic for Breast implant-Associated Anaplastic Large Cell Lymphoma. , 2020, Blood.
[24] C. Gambacorti-Passerini,et al. Phase two study of crizotinib in patients with anaplastic lymphoma kinase (ALK)‐positive anaplastic large cell lymphoma relapsed/refractory to chemotherapy , 2020, American journal of hematology.
[25] C. Ong,et al. Avelumab for the treatment of relapsed or refractory extranodal NK/T-cell lymphoma: an open-label phase 2 study. , 2020, Blood.
[26] Dachuan Huang,et al. Whole-genome sequencing identifies responders to Pembrolizumab in relapse/refractory natural-killer/T cell lymphoma , 2020, Leukemia.
[27] S. Chiba,et al. Advances in understanding of angioimmunoblastic T-cell lymphoma , 2020, Leukemia.
[28] M. Georges,et al. Targeted deep sequencing reveals clonal and subclonal mutational signatures in Adult T-cell leukemia/lymphoma and defines an unfavorable indolent subtype , 2020, Leukemia.
[29] S. Kato,et al. Nodal EBV+ cytotoxic T-cell lymphoma: A literature review based on the 2017 WHO classification , 2020, Journal of clinical and experimental hematopathology : JCEH.
[30] D. de Jong,et al. Increased prevalence of BRCA1/2 mutations in women with macro-textured breast implants and anaplastic large cell lymphoma of the breast. , 2020, Blood.
[31] M. Arcila,et al. Clonal hematopoiesis in angioimmunoblastic T-cell lymphoma with divergent evolution to myeloid neoplasms. , 2020, Blood advances.
[32] A. Feldman,et al. Molecular profiling reveals a hypoxia signature in breast implant-associated anaplastic large cell lymphoma , 2020, Haematologica.
[33] Ali F. Alsulami,et al. Whole exome sequencing reveals NOTCH1 mutations in anaplastic large cell lymphoma and points to Notch both as a key pathway and a potential therapeutic target , 2020, Haematologica.
[34] Weili Zhao,et al. Genomic and Transcriptomic Characterization of Natural Killer T Cell Lymphoma. , 2020, Cancer cell.
[35] G. Follows,et al. Angioimmunoblastic T‐cell lymphoma contains multiple clonal T‐cell populations derived from a common TET2 mutant progenitor cell , 2019, The Journal of pathology.
[36] L. Staudt,et al. Reproducing the Molecular Subclassification of Peripheral T-cell Lymphoma-NOS by Immunohistochemistry. , 2019, Blood.
[37] L. Quintanilla‐Martinez,et al. Mutational profile and EBV strains of extranodal NK/T-cell lymphoma, nasal type in Latin America , 2019, Modern Pathology.
[38] K. Debackere,et al. FER and FES tyrosine kinase fusions in follicular T-cell lymphoma. , 2019, Blood.
[39] B. Ebert,et al. Clonal hematopoiesis in human aging and disease , 2019, Science.
[40] W. Park,et al. Immune subtyping of extranodal NK/T-cell lymphoma: a new biomarker and an immune shift during disease progression , 2019, Modern Pathology.
[41] S. Pileri,et al. Recurrent PDL1 expression and PDL1 (CD274) copy number alterations in breast implant-associated anaplastic large-cell lymphomas. , 2019, Human pathology.
[42] R. Gascoyne,et al. Identification of high‐risk DUSP22‐rearranged ALK‐negative anaplastic large cell lymphoma , 2019, British journal of haematology.
[43] Jesse S. Voss,et al. Recurrent MSC E116K mutations in ALK-negative anaplastic large cell lymphoma. , 2019, Blood.
[44] S. Crotty. T Follicular Helper Cell Biology: A Decade of Discovery and Diseases. , 2019, Immunity.
[45] T. Miyazaki,et al. Molecular heterogeneity in peripheral T-cell lymphoma, not otherwise specified revealed by comprehensive genetic profiling , 2019, Leukemia.
[46] S. Pileri,et al. Whole exome sequencing reveals mutations in FAT1 tumor suppressor gene clinically impacting on peripheral T-cell lymphoma not otherwise specified , 2019, Modern Pathology.
[47] L. Staudt,et al. Genetic drivers of oncogenic pathways in molecular subgroups of peripheral T-cell lymphoma. , 2019, Blood.
[48] O. Dereure,et al. Systemic, primary cutaneous, and breast implant-associated ALK-negative anaplastic large-cell lymphomas present similar biologic features despite distinct clinical behavior , 2019, Virchows Archiv.
[49] P. Gaulard,et al. The pathological features of angioimmunoblastic T-cell lymphomas with IDH2R172 mutations , 2019, Modern Pathology.
[50] A. Jeyasekharan,et al. Molecular pathogenic pathways in extranodal NK/T cell lymphoma , 2019, Journal of Hematology & Oncology.
[51] P. Gaulard,et al. Frequent structural variations involving programmed death ligands in Epstein-Barr virus-associated lymphomas , 2019, Leukemia.
[52] Bo W. Han,et al. Genomic and transcriptomic landscapes of Epstein-Barr virus in extranodal natural killer T-cell lymphoma , 2018, Leukemia.
[53] S. Fox,et al. Frequent activating STAT3 mutations and novel recurrent genomic abnormalities detected in breast implant-associated anaplastic large cell lymphoma , 2018, Oncotarget.
[54] M. Piris,et al. DUSP22-rearranged anaplastic lymphomas are characterized by specific morphological features and a lack of cytotoxic and JAK/STAT surrogate markers , 2018, Haematologica.
[55] K. Pradhan,et al. North American ATLL has a distinct mutational and transcriptional profile and responds to epigenetic therapies. , 2018, Blood.
[56] J. Cerhan,et al. Molecular profiling reveals immunogenic cues in anaplastic large cell lymphomas with DUSP22 rearrangements. , 2018, Blood.
[57] R. D. Hatton,et al. Differential IL-2 expression defines developmental fates of follicular versus nonfollicular helper T cells , 2018, Science.
[58] G. Bhagat,et al. Transcriptional analysis distinguishes breast implant-associated anaplastic large cell lymphoma from other peripheral T-cell lymphomas , 2018, Modern Pathology.
[59] L. Medeiros,et al. Breast implant-associated anaplastic large cell lymphoma: a review , 2018, Modern Pathology.
[60] J. Aster,et al. RhoA G17V is sufficient to induce autoimmunity and promotes T-cell lymphomagenesis in mice. , 2018, Blood.
[61] S. Iida,et al. CCR4 mutations associated with superior outcome of adult T-cell leukemia/lymphoma under mogamulizumab treatment. , 2018, Blood.
[62] A. Feldman,et al. Genetic subtyping of breast implant-associated anaplastic large cell lymphoma. , 2018, Blood.
[63] P. Murray,et al. Epstein-Barr Virus-Induced Epigenetic Pathogenesis of Viral-Associated Lymphoepithelioma-Like Carcinomas and Natural Killer/T-Cell Lymphomas , 2018, Pathogens.
[64] Preti Jain,et al. Targeted next generation sequencing of breast implant‐associated anaplastic large cell lymphoma reveals mutations in JAK/STAT signalling pathway genes, TP53 and DNMT3A , 2018, British journal of haematology.
[65] A. Ferrando,et al. RHOA G17V Induces T Follicular Helper Cell Specification and Promotes Lymphomagenesis. , 2018, Cancer cell.
[66] J. Jen,et al. Targetable fusions of the FRK tyrosine kinase in ALK-negative anaplastic large cell lymphoma , 2017, Leukemia.
[67] P. Gaulard,et al. Activation of RHOA–VAV1 signaling in angioimmunoblastic T-cell lymphoma , 2017, Leukemia.
[68] Dachuan Huang,et al. Oncogenic activation of the STAT3 pathway drives PD-L1 expression in natural killer/T-cell lymphoma. , 2017, Blood.
[69] O. Nureki,et al. Prognostic relevance of integrated genetic profiling in adult T-cell leukemia/lymphoma. , 2015, Blood.
[70] C. Ong,et al. Epstein-Barr virus-associated primary nodal T/NK-cell lymphoma shows a distinct molecular signature and copy number changes , 2017, Haematologica.
[71] D. Dunson,et al. Genetic and Functional Drivers of Diffuse Large B Cell Lymphoma , 2017, Cell.
[72] M. Matsuoka,et al. Human T-cell leukaemia virus type 1: parasitism and pathogenesis , 2017, Philosophical Transactions of the Royal Society B: Biological Sciences.
[73] A. Feldman,et al. DUSP22 AND TP63 REARRANGEMENTS PREDICT OUTCOME OF ALK‐NEGATIVE ANAPLASTIC LARGE CELL LYMPHOMA: A DANISH COHORT STUDY , 2017, Blood.
[74] P. Gaulard,et al. Integrative clinicopathological and molecular analyses of angioimmunoblastic T-cell lymphoma and other nodal lymphomas of follicular helper T-cell origin , 2017, Haematologica.
[75] I. Lossos,et al. Activating mutations and translocations in the guanine exchange factor VAV1 in peripheral T-cell lymphomas , 2017, Proceedings of the National Academy of Sciences.
[76] S. Miyano,et al. Identification of cell-type-specific mutations in nodal T-cell lymphomas , 2017, Blood Cancer Journal.
[77] P. Gaulard,et al. The IDH2 R172K mutation associated with angioimmunoblastic T-cell lymphoma produces 2HG in T cells and impacts lymphoid development , 2016, Proceedings of the National Academy of Sciences.
[78] Wen-Qi Jiang,et al. PD-L1 is upregulated by EBV-driven LMP1 through NF-κB pathway and correlates with poor prognosis in natural killer/T-cell lymphoma , 2016, Journal of Hematology & Oncology.
[79] Ryan D. Morin,et al. Activating mutations in genes related to TCR signaling in angioimmunoblastic and other follicular helper T-cell-derived lymphomas. , 2016, Blood.
[80] Sarah H. Johnson,et al. Integrated mate-pair and RNA sequencing identifies novel, targetable gene fusions in peripheral T-cell lymphoma. , 2016, Blood.
[81] Gisela Mir Arnau,et al. Whole exome sequencing reveals activating JAK1 and STAT3 mutations in breast implant-associated anaplastic large cell lymphoma anaplastic large cell lymphoma , 2016, Haematologica.
[82] P. Kim,et al. Frequent CTLA4-CD28 gene fusion in diverse types of T-cell lymphoma , 2016, Haematologica.
[83] Satoru Miyano,et al. Aberrant PD-L1 expression through 3′-UTR disruption in multiple cancers , 2016, Nature.
[84] Y. Ko,et al. Nasal-type NK/T-cell lymphomas are more frequently T rather than NK lineage based on T-cell receptor gene, RNA, and protein studies: lineage does not predict clinical behavior , 2016, Modern Pathology.
[85] P. Gaulard,et al. DNMT3AR882H mutant and Tet2 inactivation cooperate in the deregulation of DNA methylation control to induce lymphoid malignancies in mice , 2016, Leukemia.
[86] P. Gaulard,et al. Breast implant-associated anaplastic large cell lymphoma: two distinct clinicopathological variants with different outcomes. , 2016, Annals of oncology : official journal of the European Society for Medical Oncology.
[87] R. Rabadán,et al. Identification of a new subclass of ALK-negative ALCL expressing aberrant levels of ERBB4 transcripts. , 2016, Blood.
[88] H. Aburatani,et al. Integrated molecular analysis of adult T cell leukemia/lymphoma , 2015, Nature Genetics.
[89] T. Molina,et al. Angioimmunoblastic T-cell lymphoma is the most common T-cell lymphoma in two distinct French information data sets , 2015, Haematologica.
[90] A. Rosenwald,et al. Convergent mutations and kinase fusions lead to oncogenic STAT3 activation in anaplastic large cell lymphoma. , 2015, Cancer cell.
[91] W. Choi,et al. Receptor-type tyrosine-protein phosphatase κ directly targets STAT3 activation for tumor suppression in nasal NK/T-cell lymphoma. , 2015, Blood.
[92] L. Staudt,et al. IDH2R172 mutations define a unique subgroup of patients with angioimmunoblastic T-cell lymphoma. , 2014, Blood.
[93] L. Staudt,et al. Gain-of-function CCR4 mutations in adult T cell leukemia/lymphoma , 2014, The Journal of experimental medicine.
[94] K. Elenitoba-Johnson,et al. A novel recurrent NPM1-TYK2 gene fusion in cutaneous CD30-positive lymphoproliferative disorders. , 2014, Blood.
[95] H. Aburatani,et al. Reduced TET2 function leads to T-cell lymphoma with follicular helper T-cell-like features in mice , 2014, Blood Cancer Journal.
[96] W. Wilson,et al. ALK-negative anaplastic large cell lymphoma is a genetically heterogeneous disease with widely disparate clinical outcomes. , 2014, Blood.
[97] Min Kyung Sung,et al. A recurrent inactivating mutation in RHOA GTPase in angioimmunoblastic T cell lymphoma , 2014, Nature Genetics.
[98] O. Nureki,et al. Somatic RHOA mutation in angioimmunoblastic T cell lymphoma , 2014, Nature Genetics.
[99] P. Gaulard,et al. JAK3 deregulation by activating mutations confers invasive growth advantage in extranodal nasal-type natural killer cell lymphoma , 2014, Leukemia.
[100] I. Lossos,et al. Recurrent mutations in epigenetic regulators, RHOA and FYN kinase in peripheral T cell lymphomas , 2014, Nature Genetics.
[101] N. Harris,et al. A targeted mutational landscape of angioimmunoblastic T-cell lymphoma. , 2012, Blood.
[102] L. Staudt,et al. Gene expression signatures delineate biological and prognostic subgroups in peripheral T-cell lymphoma. , 2012, Blood.
[103] A. Rosenwald,et al. PRDM1/BLIMP1 is commonly inactivated in anaplastic large T-cell lymphoma. , 2013, Blood.
[104] W. Chan,et al. MicroRNA expression profiling identifies molecular signatures associated with anaplastic large cell lymphoma. , 2013, Blood.
[105] B. Nathwani,et al. Classification of non-Hodgkin lymphoma in Central and South America: a review of 1028 cases. , 2012, Blood.
[106] David I. Smith,et al. Genome-wide analysis reveals recurrent structural abnormalities of TP63 and other p53-related genes in peripheral T-cell lymphomas. , 2012, Blood.
[107] Francesco Bertoni,et al. Identification of a 3-gene model as a powerful diagnostic tool for the recognition of ALK-negative anaplastic large-cell lymphoma. , 2012, Blood.
[108] Ryan D. Morin,et al. TBL1XR1/TP63: a novel recurrent gene fusion in B-cell non-Hodgkin lymphoma. , 2012, Blood.
[109] W. Chan,et al. IDH2 mutations are frequent in angioimmunoblastic T-cell lymphoma. , 2012, Blood.
[110] K. Karube,et al. Identification of FOXO3 and PRDM1 as tumor-suppressor gene candidates in NK-cell neoplasms by genomic and functional analyses. , 2011, Blood.
[111] P. Opolon,et al. TET2 inactivation results in pleiotropic hematopoietic abnormalities in mouse and is a recurrent event during human lymphomagenesis. , 2011, Cancer cell.
[112] A. Feldman,et al. Specificity of IRF4 translocations for primary cutaneous anaplastic large cell lymphoma: a multicenter study of 204 skin biopsies , 2011, Modern Pathology.
[113] M. Salto‐Tellez,et al. Activated oncogenic pathways and therapeutic targets in extranodal nasal‐type NK/T cell lymphoma revealed by gene expression profiling , 2011, The Journal of pathology.
[114] David I. Smith,et al. Discovery of recurrent t(6;7)(p25.3;q32.3) translocations in ALK-negative anaplastic large cell lymphomas by massively parallel genomic sequencing. , 2011, Blood.
[115] R. Greil,et al. Identification of differential and functionally active miRNAs in both anaplastic lymphoma kinase (ALK)+ and ALK− anaplastic large-cell lymphoma , 2010, Proceedings of the National Academy of Sciences.
[116] P. Gaulard,et al. Gene expression profiling identifies emerging oncogenic pathways operating in extranodal NK/T-cell lymphoma, nasal type. , 2010, Blood.
[117] W. Chan,et al. Molecular signatures to improve diagnosis in peripheral T-cell lymphoma and prognostication in angioimmunoblastic T-cell lymphoma. , 2010, Blood.
[118] R. deLeeuw,et al. Genomic analyses reveal global functional alterations that promote tumor growth and novel tumor suppressor genes in natural killer-cell malignancies , 2009, Leukemia.
[119] M. Wasik,et al. Oncogenic kinase NPM/ALK induces through STAT3 expression of immunosuppressive protein CD274 (PD-L1, B7-H1) , 2008, Proceedings of the National Academy of Sciences.
[120] Stefano A Pileri,et al. ALK- anaplastic large-cell lymphoma is clinically and immunophenotypically different from both ALK+ ALCL and peripheral T-cell lymphoma, not otherwise specified: report from the International Peripheral T-Cell Lymphoma Project. , 2008, Blood.
[121] R. Gascoyne,et al. Genomic profiling reveals different genetic aberrations in systemic ALK‐positive and ALK‐negative anaplastic large cell lymphomas , 2008, British journal of haematology.
[122] Christian Gisselbrecht,et al. The gene expression profile of nodal peripheral T-cell lymphoma demonstrates a molecular link between angioimmunoblastic T-cell lymphoma (AITL) and follicular helper T (TFH) cells. , 2007, Blood.
[123] R. Siebert,et al. Novel t(5;9)(q33;q22) fuses ITK to SYK in unspecified peripheral T-cell lymphoma , 2006, Leukemia.
[124] H. Tagawa,et al. Genome‐wide array‐based comparative genomic hybridization of natural killer cell lymphoma/leukemia: Different genomic alteration patterns of aggressive NK‐cell leukemia and extranodal Nk/T‐cell lymphoma, nasal type , 2005, Genes, chromosomes & cancer.
[125] D. Levy,et al. Stat3 is required for ALK-mediated lymphomagenesis and provides a possible therapeutic target , 2005, Nature Medicine.
[126] G. Delsol,et al. Oncogenic protein tyrosine kinases , 2004, Cellular and Molecular Life Sciences CMLS.
[127] D. Levy,et al. Anaplastic lymphoma kinase (ALK) activates Stat3 and protects hematopoietic cells from cell death , 2002, Oncogene.
[128] K. Ohshima,et al. Nasal lymphomas in Japan: a high prevalence of Epstein-Barr virus type A and deletion within the latent membrane protein gene. , 1999, Leukemia & lymphoma.
[129] H. Satoh,et al. Characterization of the transforming activity of p80, a hyperphosphorylated protein in a Ki-1 lymphoma cell line with chromosomal translocation t(2;5). , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[130] D N Shapiro,et al. Fusion of a kinase gene, ALK, to a nucleolar protein gene, NPM, in non-Hodgkin's lymphoma. , 1994, Science.