BCL3-PVRL2-TOMM40 SNPs, gene-gene and gene-environment interactions on dyslipidemia

[1]  R. Krauss,et al.  Low-density lipoproteins cause atherosclerotic cardiovascular disease , 2018 .

[2]  Lale Tokgözoğlu,et al.  Low-density lipoproteins cause atherosclerotic cardiovascular disease. 1. Evidence from genetic, epidemiologic, and clinical studies. A consensus statement from the European Atherosclerosis Society Consensus Panel , 2017, European heart journal.

[3]  Jinzhen Wu,et al.  Association of the SPTLC3 rs364585 polymorphism and serum lipid profiles in two Chinese ethnic groups , 2017, Lipids in Health and Disease.

[4]  M. Munafo,et al.  G = E: What GWAS Can Tell Us about the Environment , 2016, PLoS genetics.

[5]  S. Pan,et al.  Association between the DOCK7, PCSK9 and GALNT2 Gene Polymorphisms and Serum Lipid levels , 2016, Scientific Reports.

[6]  S. Pan,et al.  Association of the variants and haplotypes in the DOCK7, PCSK9 and GALNT2 genes and the risk of hyperlipidaemia , 2015, Journal of cellular and molecular medicine.

[7]  De-zhai Yang,et al.  Prevalence of dyslipidemia and its risk factors in the Chinese Maonan and Han populations , 2016 .

[8]  E. Weiderpass,et al.  The High Prevalence of Low HDL-Cholesterol Levels and Dyslipidemia in Rural Populations in Northwestern China , 2015, PloS one.

[9]  Hui Li,et al.  Serum lipid profiles, the prevalence of dyslipidemia and the risk factors in two isolated Chinese minorities. , 2015, International journal of clinical and experimental medicine.

[10]  M. Arai,et al.  Association of genetic variants with dyslipidemia. , 2015, Molecular medicine reports.

[11]  R. Nsubuga,et al.  Prevalence of Dyslipidaemia and Associated Risk Factors in a Rural Population in South-Western Uganda: A Community Based Survey , 2015, PloS one.

[12]  Kota Matsui,et al.  Association of genetic variants with coronary artery disease and ischemic stroke in a longitudinal population-based genetic epidemiological study. , 2015, Biomedical reports.

[13]  X. Lou UGMDR: a unified conceptual framework for detection of multifactor interactions underlying complex traits , 2014, Heredity.

[14]  B. Zhang,et al.  Impact of cigarette smoking cessation on high-density lipoprotein functionality. , 2014, Circulation journal : official journal of the Japanese Circulation Society.

[15]  N. Mulia,et al.  U.S. trends in light, moderate, and heavy drinking episodes from 2000 to 2010. , 2014, Alcoholism, clinical and experimental research.

[16]  Jennifer G. Robinson,et al.  2013 ACC/AHA guideline on the treatment of blood cholesterol to reduce atherosclerotic cardiovascular risk in adults: a report of the American College of Cardiology/American Heart Association Task Force on Practice Guidelines. , 2014, Circulation.

[17]  O. Melander,et al.  Genetic Determinants of Long-Term Changes in Blood Lipid Concentrations: 10-Year Follow-Up of the GLACIER Study , 2014, PLoS genetics.

[18]  Wei Wang,et al.  Association of the variants in the BUD13-ZNF259 genes and the risk of hyperlipidaemia , 2014, Journal of cellular and molecular medicine.

[19]  Md Pgy Kathryn Beldowski 2013 ACC/AHA Guideline on the Treatment of Blood Cholesterol to Reduce Atherosclerotic Cardiovascular Risk in Adults , 2014 .

[20]  Fabian J Theis,et al.  Effects of smoking and smoking cessation on human serum metabolite profile: results from the KORA cohort study , 2013, BMC Medicine.

[21]  Jinzhen Wu,et al.  Interactions between the apolipoprotein a1/c3/a5 haplotypes and alcohol consumption on serum lipid levels. , 2013, Alcoholism, clinical and experimental research.

[22]  Jennifer G. Robinson,et al.  Reprint: 2013 ACC/AHA Guideline on the Treatment of Blood Cholesterol to Reduce Atherosclerotic Cardiovascular Risk in Adults. , 2013, Journal of the American Pharmacists Association : JAPhA.

[23]  Tom R. Gaunt,et al.  Large-scale gene-centric meta-analysis across 32 studies identifies multiple lipid loci. , 2012, American journal of human genetics.

[24]  E. Nakandakare,et al.  The role of dietary fatty acids in the pathology of metabolic syndrome. , 2012, The Journal of nutritional biochemistry.

[25]  D. Prabhakaran,et al.  Implications of discoveries from genome-wide association studies in current cardiovascular practice. , 2011, World journal of cardiology.

[26]  A. Banerjee,et al.  Familial History of Stroke Is Associated With Acute Coronary Syndromes in Women , 2011, Circulation. Cardiovascular genetics.

[27]  Tom R. Gaunt,et al.  Gene-centric association signals for lipids and apolipoproteins identified via the HumanCVD BeadChip. , 2009, American journal of human genetics.

[28]  D. Arnett,et al.  Physical inactivity interacts with an endothelial lipase polymorphism to modulate high density lipoprotein cholesterol in the GOLDN study. , 2009, Atherosclerosis.

[29]  R. Hui,et al.  9p21 is a Shared Susceptibility Locus Strongly for Coronary Artery Disease and Weakly for Ischemic Stroke in Chinese Han Population , 2009, Circulation. Cardiovascular genetics.

[30]  J. Chu,et al.  The association between HLA-A, -B alleles and major histocompatibility complex class I polymorphic Alu insertions in four populations in China. , 2009, Tissue antigens.

[31]  F. Paccaud,et al.  Alcohol drinking and cardiovascular risk in a population with high mean alcohol consumption. , 2009, The American journal of cardiology.

[32]  S. Schinner,et al.  Alcohol consumption and blood lipids in elderly coronary patients , 2009 .

[33]  C. Erem,et al.  Prevalence of dyslipidemia and associated risk factors among Turkish adults: Trabzon lipid study , 2008, Endocrine.

[34]  Y. Rui-xing,et al.  Associations of diet and lifestyle with hyperlipidemia for middle-aged and elderly persons among the Guangxi Bai Ku Yao and Han populations. , 2008, Journal of the American Dietetic Association.

[35]  Xiaoxun Xie,et al.  Genetic relationships among four minorities in Guangxi revealed by analysis of 15 STRs. , 2007, Journal of genetics and genomics = Yi chuan xue bao.

[36]  W. Kraus,et al.  Inactivity, exercise training and detraining, and plasma lipoproteins. STRRIDE: a randomized, controlled study of exercise intensity and amount. , 2007, Journal of applied physiology.

[37]  M. Muramatsu,et al.  Polymorphisms of human leucocyte antigen genes in Maonan people in China , 2007, Tissue antigens.

[38]  S. Yusuf,et al.  Effect of potentially modifiable risk factors associated with myocardial infarction in 52 countries (the INTERHEART study): case-control study , 2004, The Lancet.

[39]  Sidney C. Smith,et al.  Atherosclerotic Vascular Disease Conference: Executive summary: Atherosclerotic Vascular Disease Conference proceeding for healthcare professionals from a special writing group of the American Heart Association. , 2004, Circulation.

[40]  M. Burns,et al.  Case-Control Study , 2020, Definitions.

[41]  T. Fukui,et al.  The effects of cessation from cigarette smoking on the lipid and lipoprotein profiles: a meta-analysis. , 2003, Preventive medicine.

[42]  P. Durrington Dyslipidaemia , 2003, The Lancet.

[43]  J. Mckenney,et al.  National Cholesterol Education Program Expert Panel on Detection, Evaluation, and Treatment of High Blood Cholesterol in Adults (Adult Treatment Panel III) , 2002 .

[44]  E. Schaefer,et al.  Dietary and plasma lipid, lipoprotein, and apolipoprotein profiles among elderly Hispanics and non-Hispanics and their association with diabetes. , 2002, The American journal of clinical nutrition.

[45]  S. Grundy,et al.  National Cholesterol Education Program Third Report of the National Cholesterol Education Program ( NCEP ) Expert Panel on Detection , Evaluation , and Treatment of High Blood Cholesterol in Adults ( Adult Treatment Panel III ) Final Report , 2022 .

[46]  M. Mayo,et al.  Differences among African American light, moderate, and heavy smokers. , 2001, Nicotine & tobacco research : official journal of the Society for Research on Nicotine and Tobacco.

[47]  J. Mckenney,et al.  Executive Summary of The Third Report of The National Cholesterol Education Program (NCEP) Expert Panel on Detection, Evaluation, And Treatment of High Blood Cholesterol In Adults (Adult Treatment Panel III). , 2001, JAMA.

[48]  C. Bouchard,et al.  Familial resemblance of plasma lipids, lipoproteins and postheparin lipoprotein and hepatic lipases in the HERITAGE Family Study. , 1997, Arteriosclerosis, thrombosis, and vascular biology.

[49]  G. Mcclearn,et al.  Genetic and environmental influences on serum lipid levels in twins. , 1993, The New England journal of medicine.