Resolvin D1 activates the inflammation resolving response at splenic and ventricular site following myocardial infarction leading to improved ventricular function.
暂无分享,去创建一个
C. Serhan | S. Prabhu | M. Joshi | R. Colas | J. Dalli | V. Kain | K. Ingle | G. Halade | Jesmond Dalli
[1] M. Lindsey,et al. Obesity superimposed on aging magnifies inflammation and delays the resolving response after myocardial infarction. , 2015, American journal of physiology. Heart and circulatory physiology.
[2] J. Min,et al. Splenic metabolic activity predicts risk of future cardiovascular events: demonstration of a cardiosplenic axis in humans. , 2015, JACC. Cardiovascular imaging.
[3] C. Serhan,et al. Protective Actions of Aspirin-Triggered (17R) Resolvin D1 and Its Analogue, 17R-Hydroxy-19-Para-Fluorophenoxy-Resolvin D1 Methyl Ester, in C5a-Dependent IgG Immune Complex–Induced Inflammation and Lung Injury , 2014, The Journal of Immunology.
[4] Sumanth D. Prabhu,et al. Inflammation revisited: inflammation versus resolution of inflammation following myocardial infarction , 2014, Basic Research in Cardiology.
[5] J. Backs,et al. Resolvin D1 limits 5-lipoxygenase nuclear localization and leukotriene B4 synthesis by inhibiting a calcium-activated kinase pathway , 2014, Proceedings of the National Academy of Sciences.
[6] E. Thorp,et al. Phagocyte-myocyte interactions and consequences during hypoxic wound healing. , 2014, Cellular immunology.
[7] C. Serhan,et al. Identification and signature profiles for pro-resolving and inflammatory lipid mediators in human tissue. , 2014, American journal of physiology. Cell physiology.
[8] C. Serhan,et al. Cutting Edge: Parathyroid Hormone Facilitates Macrophage Efferocytosis in Bone Marrow via Proresolving Mediators Resolvin D1 and Resolvin D2 , 2014, The Journal of Immunology.
[9] Charles N. Serhan,et al. Pro-resolving lipid mediators are leads for resolution physiology , 2014, Nature.
[10] F. Carvalho,et al. Impaired resolution of inflammation in human chronic heart failure , 2014, European journal of clinical investigation.
[11] V. Arroyo,et al. Resolvin D1 primes the resolution process initiated by calorie restriction in obesity‐induced steatohepatitis , 2014, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[12] Sungeun Kim,et al. Metabolic Activity of the Spleen and Bone Marrow in Patients With Acute Myocardial Infarction Evaluated by 18F-Fluorodeoxyglucose Positron Emission Tomograpic Imaging , 2014, Circulation. Cardiovascular imaging.
[13] S. Prabhu,et al. Integrative Physiology Methods and Results: We Evaluated C57bl/6 Mice with Chronic Hf 8 Weeks after Coronary Ligation. as Compared with Sham-operated Controls, Hf Mice Exhibited: (1) Increased Proinflammatory Cd11b , 2022 .
[14] S. Prabhu,et al. Cardiac immune cell remodeling after myocardial infarction. , 2013, Journal of molecular and cellular cardiology.
[15] M. Lindsey,et al. Left ventricular remodeling: one small step for the extracellular matrix will translate to a giant leap for the myocardium. , 2013, Congestive heart failure.
[16] P. Nordbeck,et al. 5-Lipoxygenase facilitates healing after myocardial infarction , 2013, Basic Research in Cardiology.
[17] M. Perretti,et al. Proresolving and Tissue-Protective Actions of Annexin A1–Based Cleavage-Resistant Peptides Are Mediated by Formyl Peptide Receptor 2/Lipoxin A4 Receptor , 2013, The Journal of Immunology.
[18] J. Hensler,et al. Reduced BDNF attenuates inflammation and angiogenesis to improve survival and cardiac function following myocardial infarction in mice , 2013, American journal of physiology. Heart and circulatory physiology.
[19] Yunan Tang,et al. Proresolution Therapy for the Treatment of Delayed Healing of Diabetic Wounds , 2013, Diabetes.
[20] Nguyen T. Nguyen,et al. Transgenic overexpression of matrix metalloproteinase-9 in macrophages attenuates the inflammatory response and improves left ventricular function post-myocardial infarction. , 2012, Journal of molecular and cellular cardiology.
[21] M. Perretti,et al. Resolvin D1 Limits Polymorphonuclear Leukocyte Recruitment to Inflammatory Loci: Receptor-Dependent Actions , 2012, Arteriosclerosis, thrombosis, and vascular biology.
[22] M. Spite,et al. Resolvins: anti-inflammatory and proresolving mediators derived from omega-3 polyunsaturated fatty acids. , 2012, Annual review of nutrition.
[23] C. Serhan,et al. Specialized Proresolving Mediators Enhance Human B Cell Differentiation to Antibody-Secreting Cells , 2012, The Journal of Immunology.
[24] Charles P. Lin,et al. Myocardial infarction accelerates atherosclerosis , 2012, Nature.
[25] Charles N. Serhan,et al. Infection Regulates Pro-Resolving Mediators that Lower Antibiotic Requirements , 2012, Nature.
[26] S. Serrano,et al. B–helper neutrophils stimulate immunoglobulin diversification and production in the marginal zone of the spleen , 2011, Nature Immunology.
[27] P. Libby,et al. Rapid monocyte kinetics in acute myocardial infarction are sustained by extramedullary monocytopoiesis , 2012, The Journal of experimental medicine.
[28] N. Frangogiannis,et al. Regulation of the inflammatory response in cardiac repair. , 2012, Circulation research.
[29] C. Serhan,et al. Emerging roles of resolvins in the resolution of inflammation and pain , 2011, Trends in Neurosciences.
[30] B. Maskrey,et al. Mechanisms of resolution of inflammation: a focus on cardiovascular disease. , 2011, Arteriosclerosis, thrombosis, and vascular biology.
[31] B. Chandrasekar,et al. Docosahexaenoic Acid-Enriched Fish Oil Attenuates Kidney Disease and Prolongs Median and Maximal Life Span of Autoimmune Lupus-Prone Mice , 2010, The Journal of Immunology.
[32] C. Serhan,et al. Resolvin D1 binds human phagocytes with evidence for proresolving receptors , 2010, Proceedings of the National Academy of Sciences.
[33] Fei Wang. The signaling mechanisms underlying cell polarity and chemotaxis. , 2009, Cold Spring Harbor perspectives in biology.
[34] J. Barkhausen,et al. Systematic analysis of functional and structural changes after coronary microembolization: a cardiac magnetic resonance imaging study. , 2009, JACC. Cardiovascular imaging.
[35] K. Chatila,et al. CD44 Is Critically Involved in Infarct Healing by Regulating the Inflammatory and Fibrotic Response1 , 2008, The Journal of Immunology.
[36] J. Uddin,et al. Resolvin D1 and Its Aspirin-triggered 17R Epimer , 2007, Journal of Biological Chemistry.
[37] John Savill,et al. Resolution of inflammation: the beginning programs the end , 2005, Nature Immunology.
[38] Dennis W. Hair,et al. Light scattering studies of stereocomplex formation of stereoregular poly(methyl methacrylate) in solutions , 2005 .
[39] M. Reilly,et al. Patients Exposed to Rofecoxib and Celecoxib Have Different Odds of Nonfatal Myocardial Infarction , 2005, Annals of Internal Medicine.
[40] T. Huynh,et al. Inhibition of COX pathway in experimental myocardial infarction. , 2004, Journal of molecular and cellular cardiology.
[41] J. McMurray,et al. Targeted Anticytokine Therapy in Patients With Chronic Heart Failure: Results of the Randomized Etanercept Worldwide Evaluation (RENEWAL) , 2004, Circulation.
[42] T. Grosser,et al. Role of Prostacyclin in the Cardiovascular Response to Thromboxane A2 , 2002, Science.
[43] J. Kamps,et al. Selective transfer of a lipophilic prodrug of 5-fluorodeoxyuridine from immunoliposomes to colon cancer cells. , 1999, Biochimica et biophysica acta.
[44] A. Mantovani,et al. Modulation of granulocyte survival and programmed cell death by cytokines and bacterial products. , 1992, Blood.