Inducible Expression of Double-Stranded RNA Reveals a Role for dFADD in the Regulation of the Antibacterial Response in Drosophila Adults

[1]  M. Belvin,et al.  The Drosophila immune response against Gram-negative bacteria is mediated by a peptidoglycan recognition protein , 2002, Nature.

[2]  M. Rämet,et al.  Functional genomic analysis of phagocytosis and identification of a Drosophila receptor for E. coli , 2002, Nature.

[3]  K. Anderson,et al.  Requirement for a Peptidoglycan Recognition Protein (PGRP) in Relish Activation and Antibacterial Immune Responses in Drosophila , 2002, Science.

[4]  J. Hoffmann,et al.  Drosophila innate immunity: an evolutionary perspective , 2002, Nature Immunology.

[5]  M. Meister,et al.  27 Methods for studying infection and immunity in Drosophila , 2002 .

[6]  S. Tauszig-Delamasure,et al.  Drosophila MyD88 is required for the response to fungal and Gram-positive bacterial infections , 2002, Nature Immunology.

[7]  R. Medzhitov,et al.  Drosophila MyD88 is an adapter in the Toll signaling pathway , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[8]  D. Zachary,et al.  Drosophila immune deficiency (IMD) is a death domain protein that activates antibacterial defense and can promote apoptosis. , 2001, Developmental cell.

[9]  M. Karin,et al.  Signal transduction by tumor necrosis factor and its relatives. , 2001, Trends in cell biology.

[10]  B. Lemaître,et al.  Mutations in the Drosophila dTAK1 gene reveal a conserved function for MAPKKKs in the control of rel/NF-kappaB-dependent innate immune responses. , 2001, Genes & development.

[11]  C. Kyriacou,et al.  Efficient and heritable functional knock-out of an adult phenotype in Drosophila using a GAL4-driven hairpin RNA incorporating a heterologous spacer. , 2001, Nucleic acids research.

[12]  B. Lemaître,et al.  Drosophila immunity: two paths to NF-κB , 2001 .

[13]  J. Mihály,et al.  The role of the Drosophila TAK homologue dTAK during development , 2001, Mechanisms of Development.

[14]  K. Anderson,et al.  The antibacterial arm of the drosophila innate immune response requires an IkappaB kinase. , 2001, Genes & development.

[15]  Xiaolu Yang,et al.  dFADD, a Novel Death Domain-containing Adapter Protein for theDrosophila Caspase DREDD* , 2000, The Journal of Biological Chemistry.

[16]  T. Maniatis,et al.  A Drosophila IkappaB kinase complex required for Relish cleavage and antibacterial immunity. , 2000, Genes & development.

[17]  R. Zhou,et al.  Role of Drosophila IKKγ in a Toll-independent antibacterial immune response , 2000, Nature Immunology.

[18]  Istvan Ando,et al.  Activation of the Drosophila NF‐κB factor Relish by rapid endoproteolytic cleavage , 2000, EMBO reports.

[19]  Antony Rodriguez,et al.  The Drosophila caspase Dredd is required to resist Gram‐negative bacterial infection , 2000, EMBO reports.

[20]  R. Carthew,et al.  Heritable gene silencing in Drosophila using double-stranded RNA , 2000, Nature Biotechnology.

[21]  M. Elrod-Erickson,et al.  Interactions between the cellular and humoral immune responses in Drosophila , 2000, Current Biology.

[22]  M. O’Connor,et al.  TAK1 Participates in c-Jun N-Terminal Kinase Signaling during Drosophila Development , 2000, Molecular and Cellular Biology.

[23]  J. Belote,et al.  Temperature‐dependent gene silencing by an expressed inverted repeat in Drosophila , 2000, Genesis.

[24]  I. Andó,et al.  Relish, a central factor in the control of humoral but not cellular immunity in Drosophila. , 1999, Molecular cell.

[25]  J. Campos-Ortega,et al.  Lethal of Scute requires overexpression of Daughterless to elicit ectopic neuronal development during embryogenesis in Drosophila , 1997, Mechanisms of Development.

[26]  M. Meister,et al.  A recessive mutation, immune deficiency (imd), defines two distinct control pathways in the Drosophila host defense. , 1995, Proceedings of the National Academy of Sciences of the United States of America.

[27]  N. Perrimon,et al.  Targeted gene expression as a means of altering cell fates and generating dominant phenotypes. , 1993, Development.