Retinoic acid and α-galactosylceramide regulate the expression of costimulatory receptors and transcription factors responsible for B cell activation and differentiation.

[1]  Dung-Tsa Chen,et al.  Therapeutic regulation of myeloid-derived suppressor cells and immune response to cancer vaccine in patients with extensive stage small cell lung cancer , 2013, Cancer Immunology, Immunotherapy.

[2]  R. Noelle,et al.  Critical Role for All-trans Retinoic Acid for Optimal Effector and Effector Memory CD8 T Cell Differentiation , 2013, The Journal of Immunology.

[3]  Yu-Chen Lin,et al.  Carbohydrate-based vaccines with a glycolipid adjuvant for breast cancer , 2013, Proceedings of the National Academy of Sciences.

[4]  D. Speiser,et al.  CD1d-antibody fusion proteins target iNKT cells to the tumor and trigger long-term therapeutic responses , 2012, Cancer Immunology, Immunotherapy.

[5]  A. Ross,et al.  All-Trans-Retinoic Acid and the Glycolipid α-Galactosylceramide Combined Reduce Breast Tumor Growth and Lung Metastasis in a 4T1 Murine Breast Tumor Model , 2012, Nutrition and cancer.

[6]  T. Mallevaey,et al.  Strategy of lipid recognition by invariant natural killer T cells: ‘one for all and all for one’ , 2012, Immunology.

[7]  K. Tarte,et al.  Characterization of a Transitional Preplasmablast Population in the Process of Human B Cell to Plasma Cell Differentiation , 2011, The Journal of Immunology.

[8]  J. Rossjohn,et al.  New ways to turn on NKT cells , 2011, The Journal of experimental medicine.

[9]  Shweta Jain,et al.  CD40 Signaling Synergizes with TLR-2 in the BCR Independent Activation of Resting B Cells , 2011, PloS one.

[10]  S. Nutt,et al.  Germinal center B and follicular helper T cells: siblings, cousins or just good friends? , 2011, Nature Immunology.

[11]  Irene Puga,et al.  Innate control of B cell responses. , 2011, Trends in immunology.

[12]  A. Ross,et al.  Retinoic Acid and α-Galactosylceramide Differentially Regulate B Cell Activation In Vitro and Augment Antibody Production In Vivo , 2011, Clinical and Vaccine Immunology.

[13]  R. Rickert,et al.  Emergence of the PI3-kinase pathway as a central modulator of normal and aberrant B cell differentiation. , 2011, Current opinion in immunology.

[14]  Yifan Ma,et al.  Vitamin A and retinoic acid in the regulation of B-cell development and antibody production. , 2011, Vitamins and hormones.

[15]  T. Tedder,et al.  B10 cells and regulatory B cells balance immune responses during inflammation, autoimmunity, and cancer , 2010, Annals of the New York Academy of Sciences.

[16]  S. Naderi,et al.  Regulation of B cell proliferation and differentiation by retinoic acid. , 2009, Seminars in immunology.

[17]  J. Mora,et al.  Role of retinoic acid in the imprinting of gut-homing IgA-secreting cells. , 2009, Seminars in immunology.

[18]  K. Rajewsky,et al.  Fas receptor expression in germinal-center B cells is essential for T and B lymphocyte homeostasis. , 2008, Immunity.

[19]  M. Huber,et al.  Enhanced B‐cell activation mediated by TLR4 and BCR crosstalk , 2008, European journal of immunology.

[20]  T. Tedder,et al.  Regulatory B cells as inhibitors of immune responses and inflammation , 2008, Immunological reviews.

[21]  G. Besra,et al.  B cell receptor-mediated uptake of CD1d-restricted antigen augments antibody responses by recruiting invariant NKT cell help in vivo , 2008, Proceedings of the National Academy of Sciences.

[22]  L. Gapin,et al.  CD1d-restricted iNKT cells, the 'Swiss-Army knife' of the immune system. , 2008, Current opinion in immunology.

[23]  M. Fujimoto,et al.  A regulatory B cell subset with a unique CD1dhiCD5+ phenotype controls T cell-dependent inflammatory responses. , 2008, Immunity.

[24]  F. Ronchi,et al.  Immune regulation by invariant NKT cells in autoimmunity. , 2008, Frontiers in bioscience : a journal and virtual library.

[25]  G. A. Lang,et al.  Glycolipid‐activated NKT cells support the induction of persistent plasma cell responses and antibody titers , 2008, European journal of immunology.

[26]  G. A. Lang,et al.  Requirement for CD1d expression by B cells to stimulate NKT cell-enhanced antibody production. , 2008, Blood.

[27]  James M. Wettenhall,et al.  Identification of Pax5 Target Genes in Early B Cell Differentiation1 , 2008, The Journal of Immunology.

[28]  T. Yamazaki,et al.  Regulation of B-cell development by BCAP and CD19 through their binding to phosphoinositide 3-kinase. , 2008, Blood.

[29]  A. Baxter,et al.  Interactions between B-Lymphocytes and Type 1 NKT Cells in Autoimmune Diabetes , 2008, Journal of immunotoxicology.

[30]  S. Nutt,et al.  The regulation of the B‐cell gene expression programme by Pax5 , 2008, Immunology and cell biology.

[31]  C. Webb,et al.  Retinoids Accelerate B Lineage Lymphoid Differentiation1 , 2008, The Journal of Immunology.

[32]  F. Alt,et al.  S-S synapsis during class switch recombination is promoted by distantly located transcriptional elements and activation-induced deaminase. , 2007, Immunity.

[33]  A. Ross,et al.  Retinoic Acid Regulates CD1d Gene Expression at the Transcriptional Level in Human and Rodent Monocytic Cells , 2007, Experimental biology and medicine.

[34]  S. Pillai,et al.  Naive recirculating B cells mature simultaneously in the spleen and bone marrow. , 2007, Blood.

[35]  Roger Sciammas,et al.  Graded expression of interferon regulatory factor-4 coordinates isotype switching with plasma cell differentiation. , 2006, Immunity.

[36]  K. Morikawa,et al.  All-trans-retinoic acid accelerates the differentiation of human B lymphocytes maturing into plasma cells. , 2005, International immunopharmacology.

[37]  A. Ross,et al.  Vitamin A and immune function: retinoic acid modulates population dynamics in antigen receptor and CD38-stimulated splenic B cells. , 2005, Proceedings of the National Academy of Sciences of the United States of America.

[38]  N. Engedal,et al.  Survival of activated human T lymphocytes is promoted by retinoic acid via induction of IL-2. , 2004, International immunology.

[39]  K. Calame,et al.  Transcription factors controlling the beginning and end of B-cell differentiation. , 2003, Current opinion in genetics & development.

[40]  H. Kagechika,et al.  Retinoic acids exert direct effects on T cells to suppress Th1 development and enhance Th2 development via retinoic acid receptors. , 2003, International immunology.

[41]  Kuo-I Lin,et al.  Blimp-1-Dependent Repression of Pax-5 Is Required for Differentiation of B Cells to Immunoglobulin M-Secreting Plasma Cells , 2002, Molecular and Cellular Biology.

[42]  M. Schebesta,et al.  Transcriptional control of B-cell development. , 2002, Current opinion in immunology.

[43]  V. Singh,et al.  Distinct Role of CD80 and CD86 in the Regulation of the Activation of B Cell and B Cell Lymphoma* , 2002, The Journal of Biological Chemistry.

[44]  A. Ross,et al.  All‐trans‐retinoic acid and polyriboinosinic : polyribocytidylic acid in combination potentiate specific antibody production and cell‐mediated immunity , 2001, Immunology.

[45]  G. Kelsoe,et al.  Spontaneous formation of germinal centers in autoimmune mice , 2001, Journal of leukocyte biology.

[46]  Abraham Kupfer,et al.  TCR-Induced Transmembrane Signaling by Peptide/MHC Class II Via Associated Ig-α/β Dimers , 2001 .

[47]  J. Kappler,et al.  TCR-induced transmembrane signaling by peptide/MHC class II via associated Ig-alpha/beta dimers. , 2001, Science.

[48]  S. Gerondakis,et al.  Rel Induces Interferon Regulatory Factor 4 (IRF-4) Expression in Lymphocytes , 2000, The Journal of experimental medicine.

[49]  J. Delabie,et al.  Marginal-zone B cells in the human lymph node and spleen show somatic hypermutations and display clonal expansion. , 1999, Blood.

[50]  M. Inaoki,et al.  The CD19-CD21 complex regulates signal transduction thresholds governing humoral immunity and autoimmunity. , 1997, Immunity.

[51]  T. Mak,et al.  Requirement for the Transcription Factor LSIRF/IRF4 for Mature B and T Lymphocyte Function , 1997, Science.

[52]  S. Sato,et al.  The CD19 signal transduction molecule is a response regulator of B-lymphocyte differentiation. , 1995, Proceedings of the National Academy of Sciences of the United States of America.