Fibrogenesis in chronic murine colitis is independent of innate lymphoid cells
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S. Vermeire | L. Boon | M. Ferrante | G. Van Assche | J. Ceuppens | C. Breynaert | J. Cremer | B. Creyns | G. De Hertogh
[1] S. Vermeire,et al. Fibrogenesis in Chronic DSS Colitis is Not Influenced by Neutralisation of Regulatory T Cells, of Major T Helper Cytokines or Absence of IL-13 , 2019, Scientific Reports.
[2] J. Mjösberg,et al. Distinct Alterations in the Composition of Mucosal Innate Lymphoid Cells in Newly Diagnosed and Established Crohn’s Disease and Ulcerative Colitis , 2018, Journal of Crohn's & colitis.
[3] K. Ishii,et al. Eosinophil depletion suppresses radiation-induced small intestinal fibrosis , 2018, Science Translational Medicine.
[4] A. Mortha,et al. Cytokine Networks between Innate Lymphoid Cells and Myeloid Cells , 2018, Front. Immunol..
[5] T. Kaisho,et al. Mast Cells Are Crucial for Induction of Group 2 Innate Lymphoid Cells and Clearance of Helminth Infections , 2017, Immunity.
[6] Junfeng Zhu,et al. Th1/Th2 Balance and Th17/Treg-Mediated Immunity in relation to Murine Resistance to Dextran Sulfate-Induced Colitis , 2017, Journal of immunology research.
[7] Minhu Chen,et al. CD177+ neutrophils as functionally activated neutrophils negatively regulate IBD , 2017, Gut.
[8] M. Hughes,et al. The orphan nuclear receptor RORα and group 3 innate lymphoid cells drive fibrosis in a mouse model of Crohn’s disease , 2016, Science Immunology.
[9] Gregory F. Sonnenberg. Transcriptionally defining ILC heterogeneity in humans , 2016, Nature Immunology.
[10] P. Fallon,et al. Macrophage and Innate Lymphoid Cell Interplay in the Genesis of Fibrosis , 2015, Front. Immunol..
[11] Kelley E. Capocelli,et al. Eosinophils and IL-33 Perpetuate Chronic Inflammation and Fibrosis in a Pediatric Population with Stricturing Crohn's Ileitis , 2015, Inflammatory bowel diseases.
[12] L. Boon,et al. Type 3 innate lymphoid cells maintain intestinal epithelial stem cells after tissue damage , 2015, The Journal of experimental medicine.
[13] I. Dotan,et al. ECM remodelling in IBD: innocent bystander or partner in crime? The emerging role of extracellular molecular events in sustaining intestinal inflammation , 2014, Gut.
[14] J. Goldblum,et al. Fibrosis in ulcerative colitis: mechanisms, features, and consequences of a neglected problem. , 2014, Inflammatory bowel diseases.
[15] J. Girard,et al. Central domain of IL-33 is cleaved by mast cell proteases for potent activation of group-2 innate lymphoid cells , 2014, Proceedings of the National Academy of Sciences.
[16] G. Rogler,et al. Results of the 4th scientific workshop of the ECCO (I): pathophysiology of intestinal fibrosis in IBD. , 2014, Journal of Crohn's & colitis.
[17] G. Corazza,et al. Absence of a role for interleukin‐13 in inflammatory bowel disease , 2014, European journal of immunology.
[18] B. Chassaing,et al. Dextran Sulfate Sodium (DSS)‐Induced Colitis in Mice , 2014, Current protocols in immunology.
[19] D. Voehringer,et al. IL-25 and type 2 innate lymphoid cells induce pulmonary fibrosis , 2013, Proceedings of the National Academy of Sciences.
[20] L. Fouser,et al. Lyn activity protects mice from DSS colitis and regulates the production of IL-22 from innate lymphoid cells , 2013, Mucosal Immunology.
[21] D. Voehringer,et al. Interleukin-33-dependent innate lymphoid cells mediate hepatic fibrosis. , 2013, Immunity.
[22] P. Rutgeerts,et al. Unique Gene Expression and MR T2 Relaxometry Patterns Define Chronic Murine Dextran Sodium Sulphate Colitis as a Model for Connective Tissue Changes in Human Crohn’s Disease , 2013, PloS one.
[23] D. Proud,et al. Role of IL-17A and neutrophils in fibrosis in experimental hypersensitivity pneumonitis. , 2013, The Journal of allergy and clinical immunology.
[24] M. Oukka,et al. IL-23R+ innate lymphoid cells induce colitis via interleukin-22-dependent mechanism , 2013, Mucosal Immunology.
[25] F. Bushman,et al. Innate lymphoid cells regulate CD4+ T cell responses to intestinal commensal bacteria , 2013, Nature.
[26] C. Buskens,et al. Human type 1 innate lymphoid cells accumulate in inflamed mucosal tissues , 2013, Nature Immunology.
[27] Eric Vivier,et al. Innate lymphoid cells — a proposal for uniform nomenclature , 2013, Nature Reviews Immunology.
[28] Amanda B. Waddell,et al. Eosinophils in infection and intestinal immunity , 2013, Current opinion in gastroenterology.
[29] C. Hofmann,et al. IL‐33 attenuates development and perpetuation of chronic intestinal inflammation , 2012, Inflammatory bowel diseases.
[30] B. Malissen,et al. Resident and pro-inflammatory macrophages in the colon represent alternative context-dependent fates of the same Ly6Chi monocyte precursors , 2012, Mucosal Immunology.
[31] Kevin W Eliceiri,et al. NIH Image to ImageJ: 25 years of image analysis , 2012, Nature Methods.
[32] Marjolein J. W. de Bruijn,et al. Pulmonary innate lymphoid cells are major producers of IL‐5 and IL‐13 in murine models of allergic asthma , 2012, European journal of immunology.
[33] T. Abe,et al. IL-33 is a crucial amplifier of innate rather than acquired immunity , 2010, Proceedings of the National Academy of Sciences.
[34] F. Powrie,et al. Innate lymphoid cells drive IL-23 dependent innate intestinal pathology , 2010, Nature.
[35] A. Spinelli,et al. Intestinal fibrosis in Crohn's disease: medical treatment or surgery? , 2010, Current drug targets.
[36] M. Ballmaier,et al. Common γ-Chain-Dependent Signals Confer Selective Survival of Eosinophils in the Murine Small Intestine1 , 2009, The Journal of Immunology.
[37] C. Hunter,et al. IL-27 Regulates Homeostasis of the Intestinal CD4+ Effector T Cell Pool and Limits Intestinal Inflammation in a Murine Model of Colitis1 , 2009, The Journal of Immunology.
[38] S. Gordon,et al. The role of macrophages in inflammatory bowel diseases , 2009, Expert Reviews in Molecular Medicine.
[39] J. Wedemeyer,et al. Role of gastrointestinal eosinophils in inflammatory bowel disease and intestinal tumours. , 2008, Best practice & research. Clinical gastroenterology.
[40] I. Williams,et al. Colonic neutrophils in inflammatory bowel disease: double-edged swords of the innate immune system with protective and destructive capacity. , 2007, Gastroenterology.
[41] J. Hagood,et al. Thy‐1 as a regulator of cell‐cell and cell‐matrix interactions in axon regeneration, apoptosis, adhesion, migration, cancer, and fibrosis , 2006, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[42] Young Ho Suh,et al. Involvement of lymphocytes in dextran sulfate sodium-induced experimental colitis. , 2006, World journal of gastroenterology.
[43] S Al-Haddad,et al. The role of eosinophils in inflammatory bowel disease , 2005, Gut.
[44] M. Pines,et al. Mast Cells Involvement in the Inflammation and Fibrosis Development of the TNBS-induced Rat Model of Colitis , 2002, Scandinavian journal of gastroenterology.
[45] Susumu Tonegawa,et al. RAG-1-deficient mice have no mature B and T lymphocytes , 1992, Cell.
[46] SURGERY IN ULCERATIVE COLITIS , 1964 .
[47] J. F. Woessner,et al. The determination of hydroxyproline in tissue and protein samples containing small proportions of this imino acid. , 1961, Archives of biochemistry and biophysics.
[48] F. Magro,et al. Fibrosis in Ulcerative Colitis , 2018 .
[49] G. Rogler,et al. Mechanisms, Management, and Treatment of Fibrosis in Patients With Inflammatory Bowel Diseases. , 2017, Gastroenterology.
[50] H. Spits,et al. The expanding family of innate lymphoid cells: regulators and effectors of immunity and tissue remodeling , 2011, Nature Immunology.