Longitudinal analysis of serum cytokines in a Behcet's patient during 9 months of IVIG infusions: how does CXCL8 bridge the immune and neuroendocrine systems?

[1]  Rosanna Abbate,et al.  Neutrophil Activation Promotes Fibrinogen Oxidation and Thrombus Formation in Behçet Disease , 2016, Circulation.

[2]  M. Bozkurt,et al.  Serum Cytokine Levels in Behçet's Disease , 2015, Journal of clinical laboratory analysis.

[3]  F. D’Acquisto,et al.  ‘As above, so below’ examining the interplay between emotion and the immune system , 2014, Immunology.

[4]  P. Sfikakis,et al.  Anti-cytokine biologic treatment beyond anti-TNF in Behçet's disease. , 2014, Clinical and experimental rheumatology.

[5]  M. D’Elios,et al.  Behçet’s syndrome pathophysiology and potential therapeutic targets , 2014, Internal and Emergency Medicine.

[6]  P. Cacoub,et al.  Approaches to immunosuppression in Behçet's disease. , 2013, Immunotherapy.

[7]  H. Kikuchi,et al.  Changes in biomarkers focused on differences in disease course or treatment in patients with neuro-Behçet's disease. , 2012, Internal medicine.

[8]  T. Ergun,et al.  Neutrophil activation in Behçet's disease. , 2001, Clinical and experimental rheumatology.

[9]  C. Zouboulis,et al.  Adamantiades-Behçet’s Disease: Serum IL-8 Is a More Reliable Marker for Disease Activity than C-Reactive Protein and Erythrocyte Sedimentation Rate , 2000, Dermatology.

[10]  C. Zouboulis,et al.  Adamantiades-Behçet’s disease: interleukin-8 is increased in serum of patients with active oral and neurological manifestations and is secreted by small vessel endothelial cells , 2000, Archives of Dermatological Research.

[11]  A. Silman,et al.  [Criteria of diagnosis of Behcet's disease]. , 1975, La Tunisie medicale.