Inhibition of COMT induces dopamine-dependent natriuresis and inhibition of proximal tubular Na+,K+-ATPase.

[1]  O. Kuchel,et al.  Dopamine deficiency--its potential contribution to chronic renal failure complicating hypertension. , 1995, Hypertension research : official journal of the Japanese Society of Hypertension.

[2]  M. Nishimura,et al.  Diagnostic significance of dopamine estimation using plasma and urine in patients with adrenal and renal insufficiency, renal transplantation and hypertension. , 1995, Hypertension research : official journal of the Japanese Society of Hypertension.

[3]  I. Ulmanen,et al.  Distribution of catechol-O-methyltransferase enzyme in rat tissues. , 1994, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

[4]  A. Aperia,et al.  Catechol-O-methyltransferase mRNA in the kidney and its appearance during ontogeny. , 1993, Kidney international.

[5]  A. Aperia,et al.  Dopamine regulation of renal Na+,K(+)-ATPase activity is lacking in Dahl salt-sensitive rats. , 1993, Hypertension.

[6]  M. Lee.  Dopamine and the kidney: ten years on. , 1993, Clinical science.

[7]  M. H. Fernandes,et al.  Type A and B monoamine oxidase activities in the human and rat kidney. , 1992, Acta physiologica Scandinavica.

[8]  M. H. Fernandes,et al.  Deamination of newly‐formed dopamine in rat renal tissues , 1991, British journal of pharmacology.

[9]  K. Shimamoto,et al.  Suppressed dopaminergic activity and water-sodium handling in the kidneys at the prehypertensive stage of essential hypertension. , 1990, Journal of autonomic pharmacology.

[10]  M. H. Fernandes,et al.  Effects of MAO-A and MAO-B selective inhibitors Ro 41-1049 and Ro 19-6327 on the deamination of newly formed dopamine in the rat kidney. , 1990, The Journal of pharmacology and experimental therapeutics.

[11]  A. Aperia,et al.  Amino acid stimulation of Na,K-ATPase activity in rat proximal tubule after high-protein diet. , 1990, Acta physiologica Scandinavica.

[12]  A. Aperia,et al.  Renal function in different forms of renovascular hypertension in rats. , 1989, Acta physiologica Scandinavica.

[13]  S. Kaakkola,et al.  Inhibition of catechol-O-methyltransferase activity by two novel disubstituted catechols in the rat. , 1988, European journal of pharmacology.

[14]  T. Hökfelt,et al.  Proximal tubule Na+-K+-ATPase activity is inhibited during high-salt diet: evidence for DA-mediated effect. , 1988, The American journal of physiology.

[15]  M. Lee,et al.  Domperidone treatment in man inhibits the fall in plasma renin activity induced by intravenous gamma-L-glutamyl-L-dopa. , 1986, British journal of clinical pharmacology.

[16]  I. Kopin,et al.  Catecholamine metabolism: basic aspects and clinical significance. , 1985, Pharmacological reviews.

[17]  E. Nissinen,et al.  Determination of catechol-O-methyltransferase activity by high-performance liquid chromatography with electrochemical detection. , 1984, Analytical biochemistry.

[18]  M. Lee.  Dopamine and the kidney. , 1982, Clinical science.

[19]  M. Raxworthy,et al.  Substrate stereospecificity and selectivity of catechol-O-methyltransferase for DOPA, DOPA derivatives and alpha-substituted catecholamines. , 1982, Biochemical pharmacology.

[20]  R. Carey,et al.  Decreased plasma and urinary dopamine during dietary sodium depletion in man. , 1981, The Journal of clinical endocrinology and metabolism.

[21]  A. Baines,et al.  Production of urine free dopamine from DOPA; a micropuncture study. , 1980, Life sciences.

[22]  M. Orłowski,et al.  gamma-Glutamyl dopa: a kidney-specific dopamine precursor. , 1978, The Journal of pharmacology and experimental therapeutics.

[23]  S. Ball,et al.  The effect of carbidopa administration on urinary sodium excretion in man. Is dopamine an intrarenal natriuretic hormone? , 1977, British journal of clinical pharmacology.

[24]  C. Marsden,et al.  Catechol-O-methyl transferase: pharmacological aspects and physiological role. , 1975, Pharmacological reviews.

[25]  W. Lovenberg,et al.  Effects of dietary sodium and of acute saline infusion on the interrelationship between dopamine excretion and adrenergic activity in man. , 1974, The Journal of clinical investigation.

[26]  A. Barbeau,et al.  Relationship between the adrenergic nervous system and renin during adaptation to upright posture: a possible role for 3,4-dihydroxyphenethylamine (dopamine). , 1972, Clinical science.

[27]  C. Clemente,et al.  DISTRIBUTION OF CATECHOL‐O‐METHYL TRANSFERASE IN THE NERVOUS SYSTEM AND OTHER TISSUES , 1959 .

[28]  M. H. Fernandes,et al.  Role of monoamine oxidase and catechol-O-methyltransferase in the metabolism of renal dopamine. , 1994, Journal of neural transmission. Supplementum.

[29]  S. Kaakkola,et al.  Characteristics of catechol O-methyl-transferase (COMT) and properties of selective COMT inhibitors. , 1992, Progress in drug research. Fortschritte der Arzneimittelforschung. Progres des recherches pharmaceutiques.

[30]  T. Saruta,et al.  Aromatic L-amino acid decarboxylase activity along the rat nephron. , 1990, The American journal of physiology.

[31]  A. Aperia,et al.  Dopamine causes inhibition of Na+-K+-ATPase activity in rat proximal convoluted tubule segments. , 1987, The American journal of physiology.

[32]  J. Hagège,et al.  Proximal tubule dopamine histofluorescence in renal slices incubated with L-dopa. , 1985, Kidney international.