Females with paired occurrence of cancers in the UADT and genital region have a higher frequency of either Glutathione S-transferase M1/T1 null genotype

[1]  W. Foulkes,et al.  Meta- and pooled analyses of GSTM1, GSTT1, GSTP1, and CYP1A1 genotypes and risk of head and neck cancer. , 2003, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.

[2]  M. Plummer,et al.  Smoking and cervical cancer: pooled analysis of the IARC multi-centric case–control study , 2003, Cancer Causes & Control.

[3]  S. Franceschi,et al.  Human papillomavirus and risk factors for cervical cancer in Chennai, India: A case‐control study , 2003, International journal of cancer.

[4]  A. Hirvonen,et al.  Molecular epidemiology of sporadic breast cancer. The role of polymorphic genes involved in oestrogen biosynthesis and metabolism. , 2003, Mutation research.

[5]  R. Herrero.  Chapter 7: Human papillomavirus and cancer of the upper aerodigestive tract. , 2003, Journal of the National Cancer Institute. Monographs.

[6]  F. X. Bosch,et al.  Chapter 1: Human papillomavirus and cervical cancer--burden and assessment of causality. , 2003, Journal of the National Cancer Institute. Monographs.

[7]  S. Franceschi,et al.  Role of paan chewing and dietary habits in cervical carcinoma in Chennai, India , 2003, British Journal of Cancer.

[8]  L. Ward,et al.  The null genotype of glutathione s-transferase M1 and T1 locus increases the risk for thyroid cancer. , 2002, Cancer epidemiology, biomarkers & prevention : a publication of the American Association for Cancer Research, cosponsored by the American Society of Preventive Oncology.

[9]  S. Buch,et al.  Polymorphism at GSTM1, GSTM3 and GSTT1 gene loci and susceptibility to oral cancer in an Indian population. , 2002, Carcinogenesis.

[10]  S. Franceschi,et al.  Oral cancer in southern India: The influence of smoking, drinking, paan‐chewing and oral hygiene , 2002, International journal of cancer.

[11]  G Thomas,et al.  Genetic polymorphism of CYP1A1, GSTM1 and GSTT1 genes in Indian oral cancer. , 2001, Oral oncology.

[12]  Yongwen Jiang,et al.  Second primary cancers after anogenital, skin, oral, esophageal and rectal cancers: Etiological links? , 2001, International journal of cancer.

[13]  L. Marchand,et al.  CYP1A1, GSTM1, and GSTT1 polymorphisms and the risk of cervical squamous intraepithelial lesions in a multiethnic population. , 2001, Gynecologic oncology.

[14]  S. Namkoong,et al.  Combined analysis of germline polymorphisms of p53, GSTM1, GSTT1, CYP1a1, and CYP2e1 , 2000, Cancer.

[15]  N. Weiss,et al.  Glutathione S-transferase M1 genotypes and the risk of squamous carcinoma of the cervix: a population-based case-control study. , 1999, American journal of epidemiology.

[16]  M. Spitz,et al.  Glutathione‐S‐transferase polymorphisms and risk of squamous‐cell carcinoma of the head and neck , 1999, International journal of cancer.

[17]  C. Chen,et al.  Decreased expression of glutathione S-transferase M1 in HPV16-transfected human cervical keratinocytes in culture. , 1999, Carcinogenesis.

[18]  M. Yano,et al.  Genetic polymorphisms of drug‐metabolizing enzymes and susceptibility to head‐and‐neck squamous‐cell carcinoma , 1999, International journal of cancer.

[19]  M. Boon,et al.  Transience of cervical HPV infection in sexually active, young women with normal cervicovaginal cytology. , 1995, British Journal of Cancer.

[20]  T. Bjørge,et al.  Second primary cancers in patients with carcinoma in situ of the uterine cervix. The norwegian experience 1970–1992 , 1995, International journal of cancer.

[21]  P. Balaram,et al.  Human papillomaviruses in 91 oral cancers from indian betel quid chewers—high prevalence and multiplicity of infections , 1995, International journal of cancer.

[22]  A. Fryer,et al.  Theta class glutathione S-transferase GSTT1 genotypes and susceptibility to cervical neoplasia: interactions with GSTM1, CYP2D6 and smoking. , 1994, Carcinogenesis.

[23]  C. Wheeler,et al.  Determinants of Genital Human Papillomavirus Infection Among Cytologically Normal Women Attending the University of New Mexico Student Health Center , 1993, Sexually transmitted diseases.

[24]  M. Spitz,et al.  Association between malignancies of the upper aerodigestive tract and uterine cervix , 1992, Head & neck.

[25]  R. Biggar,et al.  Second primary cancers following anal and cervical carcinoma: evidence of shared etiologic factors. , 1992, American journal of epidemiology.

[26]  E. Franco,et al.  Multiple cancers of the upper aero-digestive tract: the challenge of risk factor identification. , 1991, Cancer letters.

[27]  L J Peters,et al.  Prevention of second primary tumors with isotretinoin in squamous-cell carcinoma of the head and neck. , 1990, The New England journal of medicine.

[28]  R. Sarin,et al.  Glutathione S-transferase M1 or T1 null genotype as a risk factor for developing multiple primary neoplasms in the upper aero-digestive tract, in Indian males using tobacco. , 2004, Oral oncology.

[29]  N. S. Murthy,et al.  Polymorphisms at GSTM1 and GSTT1 gene loci and susceptibility to cervical cancer in Indian population. , 2004, Neoplasma.

[30]  C. Matthias,et al.  [First results on the influence of polymorphisms at glutathione S-transferase, cytochrome P450, and tumor necrosis factor gene loci on the development of multiple head and neck cancer]. , 2003, Laryngo- rhino- otologie.

[31]  S. Tyring,et al.  Polymorphisms for chemical metabolizing genes and risk for cervical neoplasia , 2003, Environmental and molecular mutagenesis.

[32]  R. Herrero.  Human papillomavirus and cancer of the upper aerodigestive tract , 2003 .

[33]  J. Hayes,et al.  The glutathione S-transferase supergene family: regulation of GST and the contribution of the isoenzymes to cancer chemoprotection and drug resistance. , 1995, Critical reviews in biochemistry and molecular biology.

[34]  J. Higginson,et al.  International Agency for Research on Cancer. , 1968, WHO chronicle.