A study of cytotoxicity and genotoxicity of particulate matter (PM2.5) in human lung epithelial cells (A549)

[1]  P. Quax,et al.  Deficiency of the TLR4 analogue RP105 aggravates vein graft disease by inducing a pro-inflammatory response , 2016, Scientific Reports.

[2]  Occupational exposure to chemicals and oxidative toxic stress , 2015, Toxicology and Environmental Health Sciences.

[3]  Mi-Kyung Song,et al.  Gene expression profiling of human alveolar epithelial cells (A549 cells) exposed to atmospheric particulate matter 2.5 (PM2.5) collected from Seoul, Korea , 2014, Molecular & Cellular Toxicology.

[4]  B. Chauffert,et al.  The role of reactive oxygen species and subsequent DNA-damage response in the emergence of resistance towards resveratrol in colon cancer models , 2014, Cell Death and Disease.

[5]  M. Kleinman,et al.  Toxicity of low doses of ultrafine diesel exhaust particles on bovine brain microvessel endothelial cells , 2014, Molecular & Cellular Toxicology.

[6]  W. Dott,et al.  Pro-inflammatory effects and oxidative stress in lung macrophages and epithelial cells induced by ambient particulate matter. , 2013, Environmental pollution.

[7]  S. Tao,et al.  Emission characteristics for polycyclic aromatic hydrocarbons from solid fuels burned in domestic stoves in rural China. , 2013, Environmental science & technology.

[8]  T. Suuronen,et al.  Antagonistic crosstalk between NF-κB and SIRT1 in the regulation of inflammation and metabolic disorders. , 2013, Cellular signalling.

[9]  H. Lee,et al.  Extraction method for manipulation of water- and organic-soluble extracts of PM2.5 in Korean winter season and its chemical composition , 2013, Toxicology and Environmental Health Sciences.

[10]  M. Mattioli,et al.  Gene expression profiling of A549 cells exposed to Milan PM2.5. , 2012, Toxicology letters.

[11]  Mi-Kyung Song,et al.  Formation of a 3,4-diol-1,2-epoxide metabolite of benz[a]anthracene with cytotoxicity and genotoxicity in a human in vitro hepatocyte culture system. , 2012, Environmental toxicology and pharmacology.

[12]  S. Froehner,et al.  Health risk assessment of inhabitants exposed to PAHs particulate matter in air , 2011, Journal of environmental science and health. Part A, Toxic/hazardous substances & environmental engineering.

[13]  Y. Kim,et al.  Characteristics of the ambient particulate PAHs at Seoul, a mega city of Northeast Asia in comparison with the characteristics of a background site , 2011 .

[14]  A. Tabaku,et al.  Chronic obstructive pulmonary disease in iron-steel and ferrochrome industry workers. , 2010, Central European journal of public health.

[15]  M. Camatini,et al.  Seasonal variations in chemical composition and in vitro biological effects of fine PM from Milan. , 2010, Chemosphere.

[16]  D. Traversi,et al.  Chemical characterisation and cytotoxic effects in A549 cells of urban-air PM10 collected in Torino, Italy. , 2010, Environmental toxicology and pharmacology.

[17]  P. Schwarze,et al.  Differences in cytotoxicity versus pro-inflammatory potency of different PM fractions in human epithelial lung cells. , 2010, Toxicology in vitro : an international journal published in association with BIBRA.

[18]  G. Garçon,et al.  Oxidative damage induced in A549 cells by physically and chemically characterized air particulate matter (PM2.5) collected in Abidjan, Côte d'Ivoire , 2009, Journal of applied toxicology : JAT.

[19]  M. Kleinman,et al.  Particulate matter induced enhancement of inflammatory markers in the brains of apolipoprotein E knockout mice. , 2009, Journal of nanoscience and nanotechnology.

[20]  Tian Xia,et al.  The role of oxidative stress in ambient particulate matter-induced lung diseases and its implications in the toxicity of engineered nanoparticles. , 2008, Free radical biology & medicine.

[21]  Ying Guo,et al.  SIRT1 regulates apoptosis and Nanog expression in mouse embryonic stem cells by controlling p53 subcellular localization. , 2008, Cell stem cell.

[22]  Temporal determination of heavy metals in PM2.5 aerosols in a suburban site of Athens, Greece , 2007 .

[23]  P. Bromberg,et al.  COX-2 expression induced by diesel particles involves chromatin modification and degradation of HDAC1. , 2007, American journal of respiratory cell and molecular biology.

[24]  K. Ho,et al.  Characterizing ionic species in PM2.5 and PM10 in four Pearl River Delta cities, south China. , 2007, Journal of environmental sciences.

[25]  S. Soberanes,et al.  p53 mediates particulate matter-induced alveolar epithelial cell mitochondria-regulated apoptosis. , 2006, American journal of respiratory and critical care medicine.

[26]  P. Ostrosky-Wegman,et al.  Effect of chemical composition on the induction of DNA damage by urban airborne particulate matter , 2006, Environmental and molecular mutagenesis.

[27]  E Dybing,et al.  Release of inflammatory cytokines, cell toxicity and apoptosis in epithelial lung cells after exposure to ambient air particles of different size fractions. , 2004, Toxicology in vitro : an international journal published in association with BIBRA.

[28]  Tingming Shi,et al.  Soluble metals as well as the insoluble particle fraction are involved in cellular DNA damage induced by particulate matter , 2004, Molecular and Cellular Biochemistry.

[29]  A. Ashworth,et al.  CYLD is a deubiquitinating enzyme that negatively regulates NF-κB activation by TNFR family members , 2003, Nature.

[30]  J. Miranda,et al.  Proinflammatory and cytotoxic effects of Mexico City air pollution particulate matter in vitro are dependent on particle size and composition. , 2003, Environmental health perspectives.

[31]  Constantini Samara,et al.  Labile and bioaccessible fractions of heavy metals in the airborne particulate matter from urban and industrial areas , 2002 .

[32]  I. Rosas,et al.  Biologic effects induced in vitro by PM10 from three different zones of Mexico City. , 2002, Environmental health perspectives.

[33]  Ambient aerosol concentrations of elements resolved by size and by source: Contributions of some cytokine-active metals from coal- and oil-fired power plants , 2002 .

[34]  T. Hofmann,et al.  The pro- or anti-apoptotic function of NF-kappaB is determined by the nature of the apoptotic stimulus. , 2000, European journal of biochemistry.

[35]  K. Dreher,et al.  Particulate Matter Physicochemistry and Toxicology: In Search of Causality—A Critical Perspective , 2000, Inhalation toxicology.

[36]  C. Monn,et al.  Cytotoxicity and induction of proinflammatory cytokines from human monocytes exposed to fine (PM2.5) and coarse particles (PM10-2.5) in outdoor and indoor air. , 1999, Toxicology and applied pharmacology.