Factors influencing the induction phase of skin sensitization.

[1]  I Kimber,et al.  Investigation of lymph node cell proliferation as a possible immunological correlate of contact sensitizing potential. , 1991, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

[2]  D. Basketter,et al.  Differential cytokine production following chronic exposure of mice to chemical respiratory and contact allergens. , 1995, Immunology.

[3]  I. Kimber,et al.  Cell and molecular biology of chemical allergy , 1997, Clinical reviews in allergy & immunology.

[4]  I Kimber,et al.  Vehicle effects on skin sensitizing potency of four chemicals: assessment using the local lymph node assay , 2001, International journal of cosmetic science.

[5]  G. Kraal,et al.  Persistent immune tolerance to nickel and chromium by oral administration prior to cutaneous sensitization. , 1992, The Journal of investigative dermatology.

[6]  I Kimber,et al.  Skin sensitisation, vehicle effects and the local lymph node assay. , 2001, Food and chemical toxicology : an international journal published for the British Industrial Biological Research Association.

[7]  C. Albanesi,et al.  The role of chemokines in allergic contact dermatitis , 2001, Archives of Dermatological Research.

[8]  J. Knop,et al.  Is cytokine expression responsible for differences between allergens and irritants? , 1996, American journal of contact dermatitis : official journal of the American Contact Dermatitis Society.

[9]  D. Basketter,et al.  Influence of sodium lauryl sulphate on 2,4-dinitrochlorobenzene-induced lymph node activation. , 1993, Toxicology.

[10]  J. Ashby,et al.  Mechanistic relationship among mutagenicity, skin sensitization, and skin carcinogenicity. , 1993, Environmental health perspectives.

[11]  Jean-Pierre Lepoittevin,et al.  Hapten-Protein Interactions , 1998 .

[12]  S. Sadhra,et al.  Oxidation of resin acids in colophony (rosin) and its implications for patch testing , 1998, Contact dermatitis.

[13]  J. Geier,et al.  Genotype and phenotype of N‐acetyltransferase 2 (NAT2) polymorphism in patients with contact allergy , 1998, Contact Dermatitis.

[14]  J. Bos,et al.  The 500 Dalton rule for the skin penetration of chemical compounds and drugs , 2000, Experimental dermatology.

[15]  A. Kligman.  The identification of contact allergens by human assay. II. Factors influencing the induction and measurement of allergic contact dermatitis. , 1966, The Journal of investigative dermatology.

[16]  R. Fairchild,et al.  T cell populations primed by hapten sensitization in contact sensitivity are distinguished by polarized patterns of cytokine production: interferon gamma-producing (Tc1) effector CD8+ T cells and interleukin (Il) 4/Il-10-producing (Th2) negative regulatory CD4+ T cells , 1996, The Journal of experimental medicine.

[17]  J. Kehren,et al.  Oral Administration of Hapten Inhibits In Vivo Induction of Specific Cytotoxic CD8+ T Cells Mediating Tissue Inflammation: A Role for Regulatory CD4+ T Cells1 , 2000, The Journal of Immunology.

[18]  I. Kimber,et al.  Use of the local lymph node assay for the estimation of relative contact allergenic potency , 2000, Contact dermatitis.

[19]  A. Kligman.  The identification of contact allergens by human assay. I. A critique of standard methods. , 1966, The Journal of investigative dermatology.

[20]  T. Menné,et al.  Hand eczema in nickel‐sensitive female twins , 1983, Contact dermatitis.

[21]  E. Padovan,et al.  T cell immune responses to haptens. Structural models for allergic and autoimmune reactions. , 1996, Toxicology.

[22]  J. Heylings,et al.  Influence of dibutyl phthalate on dermal sensitization to fluorescein isothiocyanate. , 1996, Fundamental and applied toxicology : official journal of the Society of Toxicology.

[23]  R. Flavell,et al.  Essential Role of Lymph Nodes in Contact Hypersensitivity Revealed in Lymphotoxin-α–Deficient Mice , 2001, The Journal of experimental medicine.

[24]  I. Kimber,et al.  Allergic contact dermatitis: the cellular effectors , 2002, Contact dermatitis.

[25]  J. Heylings,et al.  Sensitization to 2,4-dinitrochlorobenzene: influence of vehicle on absorption and lymph node activation. , 1996, Toxicology.

[26]  T. Shiohara,et al.  Repeated elicitation of contact hypersensitivity induces a shift in cutaneous cytokine milieu from a T helper cell type 1 to a T helper cell type 2 profile. , 1997, Journal of immunology.

[27]  D. Basketter,et al.  Irritant thresholds in subjects withcolophony allergy , 2000, Contact dermatitis.

[28]  J. Wilkinson,et al.  Reduced frequency of nickel allergy upon oral nickel contact at an early age , 1991, Clinical and experimental immunology.

[29]  T. Shiohara,et al.  Distinct in vivo and in vitro cytokine profiles of draining lymph node cells in acute and chronic phases of contact hypersensitivity: importance of a type 2 cytokine-rich cutaneous milieu for the development of an early-type response in the chronic phase. , 1999, Journal of immunology.

[30]  I Kimber,et al.  Contact allergenic potency: correlation of human and local lymph node assay data. , 2001, American journal of contact dermatitis : official journal of the American Contact Dermatitis Society.

[31]  R. Steinman,et al.  Murine epidermal Langerhans cells mature into potent immunostimulatory dendritic cells in vitro , 1985, The Journal of experimental medicine.

[32]  J. Kappler,et al.  Components of the Ligand for a Ni++ Reactive Human T Cell Clone , 2003, The Journal of experimental medicine.

[33]  T. Mosmann,et al.  The expanding universe of T-cell subsets: Th1, Th2 and more. , 1996, Immunology today.