Expression of UCP2 in Wistar rats varies according to age and the severity of obesity
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C. Pheiffer | J. Louw | C. Muller | S. Ghoor | Oelfah Patel | C. Jacobs
[1] K. Adeli,et al. Selective Hepatic Insulin Resistance, VLDL Overproduction, and Hypertriglyceridemia , 2012, Arteriosclerosis, thrombosis, and vascular biology.
[2] E. Zapata,et al. High-sucrose diet increases ROS generation, FFA accumulation, UCP2 level, and proton leak in liver mitochondria. , 2011, American journal of physiology. Endocrinology and metabolism.
[3] J. Gagliardino,et al. Rat liver uncoupling protein 2: changes induced by a fructose-rich diet. , 2011, Life sciences.
[4] M. Harper,et al. Uncoupling proteins and the control of mitochondrial reactive oxygen species production. , 2011, Free radical biology & medicine.
[5] Yunfeng Zhao,et al. New Aspects of Mitochondrial Uncoupling Proteins (UCPs) and Their Roles in Tumorigenesis , 2011, International journal of molecular sciences.
[6] J. Yun,et al. Differential expression of liver proteins between obesity-prone and obesity-resistant rats in response to a high-fat diet , 2011, British Journal of Nutrition.
[7] H. Inoko,et al. Nutritional and hormonal regulation of uncoupling protein 2 , 2009, IUBMB life.
[8] A. Vidal-Puig,et al. AMPK: a metabolic gauge regulating whole-body energy homeostasis. , 2008, Trends in molecular medicine.
[9] S. Previs,et al. Diet-induced obesity alters protein synthesis: tissue-specific effects in fasted versus fed mice. , 2008, Metabolism: clinical and experimental.
[10] C. Thompson,et al. Uncoupling protein‐2 controls proliferation by promoting fatty acid oxidation and limiting glycolysis‐derived pyruvate utilization , 2008, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[11] C. Warden,et al. Nutrition & Metabolism BioMed Central Review Uncoupling , 2006 .
[12] M. Febbraio,et al. Chronic rosiglitazone treatment restores AMPKalpha2 activity in insulin-resistant rat skeletal muscle. , 2006, American journal of physiology. Endocrinology and metabolism.
[13] J. Zoll,et al. Mitochondrial tissue specificity of substrates utilization in rat cardiac and skeletal muscles , 2005, Journal of cellular physiology.
[14] D. Bolster,et al. Repression of protein synthesis and mTOR signaling in rat liver mediated by the AMPK activator aminoimidazole carboxamide ribonucleoside. , 2005, American journal of physiology. Endocrinology and metabolism.
[15] R. Bergman,et al. Molecular evidence supporting the portal theory: a causative link between visceral adiposity and hepatic insulin resistance. , 2005, American journal of physiology. Endocrinology and metabolism.
[16] D. V. van Raalte,et al. Peroxisome Proliferator-Activated Receptor (PPAR)-α: A Pharmacological Target with a Promising Future , 2004, Pharmaceutical Research.
[17] S. Miura,et al. Mechanism for Peroxisome Proliferator-activated Receptor-α Activator-induced Up-regulation of UCP2 mRNA in Rodent Hepatocytes* , 2002, The Journal of Biological Chemistry.
[18] G. Lewis,et al. Mechanisms of hepatic very low-density lipoprotein overproduction in insulin resistance. , 2001, Trends in cardiovascular medicine.
[19] P. Pennefather,et al. Increased uncoupling protein-2 levels in beta-cells are associated with impaired glucose-stimulated insulin secretion: mechanism of action. , 2001, Diabetes.
[20] P. Kopelman. Obesity as a medical problem , 2000, Nature.
[21] D. Rudy,et al. Mechanisms of Hepatic Very Low Density Lipoprotein Overproduction in Insulin Resistance , 2000, The Journal of Biological Chemistry.
[22] R. Faggioni,et al. Upregulation of uncoupling protein 2 mRNA in genetic obesity: lack of an essential role for leptin, hyperphagia, increased tissue lipid content, and TNF-alpha. , 2000, Biochimica et biophysica acta.
[23] A. Martí,et al. Up-regulation of muscle UCP2 gene expression by a new β3-adrenoceptor agonist, trecadrine, in obese (cafeteria) rodents, but down-regulation in lean animals , 2000, International Journal of Obesity.
[24] V. P. Chacko,et al. Obesity Induces Expression of Uncoupling Protein-2 in Hepatocytes and Promotes Liver ATP Depletion* , 1999, The Journal of Biological Chemistry.
[25] Margaret S. Wu,et al. Peroxisome Proliferator-Activated Receptors γ and α Mediate in Vivo Regulation of Uncoupling Protein (UCP-1, UCP-2, UCP-3) Gene Expression. , 1998, Endocrinology.
[26] O. Boss,et al. The uncoupling proteins, a review. , 1998, European journal of endocrinology.
[27] R. Seeley,et al. Elevated Free Fatty Acids Induce Uncoupling Protein 3 Expression in Muscle: A Potential Explanation for the Effect of Fasting , 1998, Diabetes.
[28] O. Boss,et al. Tissue‐dependent upregulation of rat uncoupling protein‐2 expression in response to fasting or cold , 1997, FEBS letters.
[29] T. Barber,et al. Protein synthesis is stimulated in nutritionally obese rats. , 1995, The Journal of nutrition.
[30] D. Breuillé,et al. Sepsis modifies the contribution of different organs to whole-body protein synthesis in rats. , 1994, Clinical science.
[31] S. Welle,et al. Increased protein turnover in obese women. , 1992, Metabolism: clinical and experimental.
[32] B. Caballero. The global epidemic of obesity: an overview. , 2007, Epidemiologic reviews.
[33] D. Moller,et al. Peroxisome proliferator-activated receptors gamma and alpha mediate in vivo regulation of uncoupling protein (UCP-1, UCP-2, UCP-3) gene expression. , 1998, Endocrinology.