Physiological and pathological evidence of O-GlcNAcylation regulation during pregnancy related process.

[1]  Z. Kamenov,et al.  Placental Growth Factor and Pregnancy-Associated Plasma Protein-A as Potential Early Predictors of Gestational Diabetes Mellitus , 2023, Medicina.

[2]  Y. Wu,et al.  O-GlcNAcylation promotes cerebellum development and medulloblastoma oncogenesis via SHH signaling , 2022, Proceedings of the National Academy of Sciences of the United States of America.

[3]  T. Ezashi,et al.  The role of BMP4 signaling in trophoblast emergence from pluripotency , 2022, Cellular and Molecular Life Sciences.

[4]  Jiana Huang,et al.  NAT10 Maintains OGA mRNA Stability Through ac4C Modification in Regulating Oocyte Maturation , 2022, Frontiers in Endocrinology.

[5]  S. Renaud,et al.  How trophoblasts fuse: an in-depth look into placental syncytiotrophoblast formation , 2022, Cellular and Molecular Life Sciences.

[6]  Huabin Zhu,et al.  Disruption of O-GlcNAcylation Homeostasis Induced Ovarian Granulosa Cell Injury in Bovine , 2022, International journal of molecular sciences.

[7]  M. Cruz,et al.  O-GlcNAc transferase contributes to sex-specific placental deregulation in gestational diabetes , 2022, bioRxiv.

[8]  R. Morse,et al.  Nutrient sensitive protein O-GlcNAcylation modulates the transcriptome through epigenetic mechanisms during embryonic neurogenesis , 2022, Life Science Alliance.

[9]  C. Ansong,et al.  A microphysiological model of human trophoblast invasion during implantation , 2022, Nature Communications.

[10]  G. Hart,et al.  The Beginner’s Guide to O-GlcNAc: From Nutrient Sensitive Pathway Regulation to Its Impact on the Immune System , 2022, Frontiers in Immunology.

[11]  Emily S. Barrett,et al.  Extravillous Trophoblast Migration and Invasion: Impact of Environmental Chemicals and Pharmaceuticals. , 2021, Reproductive toxicology.

[12]  M. Westwood,et al.  Altered protein O-GlcNAcylation in placentas from mothers with diabetes causes aberrant endocytosis in placental trophoblast cells , 2021, Scientific Reports.

[13]  E. Dozio,et al.  Glycation and Glycosylation in Cardiovascular Remodeling: Focus on Advanced Glycation End Products and O-Linked Glycosylations as Glucose-Related Pathogenetic Factors and Disease Markers , 2021, Journal of clinical medicine.

[14]  Q. Shu,et al.  O-GlcNAc transferase Ogt regulates embryonic neuronal development through modulating Wnt/β-catenin signaling. , 2021, Human molecular genetics.

[15]  J. Reppetti,et al.  High Levels of Tumor Necrosis Factor-Alpha Reduce Placental Aquaporin 3 Expression and Impair in vitro Trophoblastic Cell Migration , 2021, Frontiers in Physiology.

[16]  E. Alejandro,et al.  Disruption of O-Linked N-Acetylglucosamine Signaling in Placenta Induces Insulin Sensitivity in Female Offspring , 2021, International journal of molecular sciences.

[17]  D. Freeman,et al.  Adipose tissue function in healthy pregnancy, gestational diabetes mellitus and pre-eclampsia , 2021, European Journal of Clinical Nutrition.

[18]  Linlin Sui,et al.  O-GlcNAc modification mediates aquaporin 3 to coordinate endometrial cell glycolysis and affects embryo implantation , 2021, Journal of advanced research.

[19]  T. Chou,et al.  Feedback Regulation of O-GlcNAc Transferase through Translation Control to Maintain Intracellular O-GlcNAc Homeostasis , 2021, International journal of molecular sciences.

[20]  Yanling Zhang,et al.  Quantitative chemoproteomics reveals O-GlcNAcylation of cystathionine γ-lyase (CSE) represses trophoblast syncytialization. , 2021, Cell chemical biology.

[21]  G. Hart,et al.  Excessive O-GlcNAcylation Causes Heart Failure and Sudden Death , 2021, Circulation.

[22]  D. Townson,et al.  Evidence and manipulation of O-GlcNAcylation in granulosa cells of bovine antral follicles , 2021, Biology of Reproduction.

[23]  Y. Sadovsky,et al.  Placental trophoblast syncytialization potentiates macropinocytosis via mTOR signaling to adapt to reduced amino acid supply , 2021, Proceedings of the National Academy of Sciences.

[24]  C. Antoniali,et al.  Pregnancy decreases O-GlcNAc-modified proteins in systemic arteries of normotensive and spontaneously hypertensive rats. , 2020, Life sciences.

[25]  F. Rosario,et al.  Inhibition of mechanistic target of rapamycin signaling decreases levels of O-GlcNAc transferase and increases serotonin release in the human placenta. , 2020, Clinical science.

[26]  Caitlyn E. Bowman,et al.  Regulation of maternal–fetal metabolic communication , 2020, Cellular and Molecular Life Sciences.

[27]  S. Kimber,et al.  Protein O-GlcNAcylation Promotes Trophoblast Differentiation at Implantation , 2020, Cells.

[28]  Haitao Wu,et al.  Loss of O-GlcNAc transferase in neural stem cells impairs corticogenesis. , 2020, Biochemical and biophysical research communications.

[29]  Jianhua Zhang,et al.  Role of O-Linked N-acetylglucosamine (O-GlcNAc) Protein Modification in Cellular (Patho)Physiology. , 2020, Physiological reviews.

[30]  N. Freitag,et al.  Altered Glycosylation Contributes to Placental Dysfunction Upon Early Disruption of the NK Cell-DC Dynamics , 2020, Frontiers in Immunology.

[31]  Rosalind J Wright,et al.  Maternal stress in relation to sex-specific expression of placental genes involved in nutrient transport, oxygen tension, immune response, and the glucocorticoid barrier. , 2020, Placenta.

[32]  M. Moulton,et al.  O-GlcNAcylation Dampens Dpp/BMP Signaling to Ensure Proper Drosophila Embryonic Development. , 2020, Developmental cell.

[33]  R. Webb,et al.  O-GlcNAc impairs endothelial function in uterine arteries from virgin but not pregnant rats: The role of GSK3β. , 2020, European journal of pharmacology.

[34]  J. Reppetti,et al.  Intact caveolae are required for proper extravillous trophoblast migration and differentiation , 2020, Journal of cellular physiology.

[35]  U. Baumann,et al.  Loss of GFAT-1 feedback regulation activates the hexosamine pathway that modulates protein homeostasis , 2020, Nature Communications.

[36]  F. Cunha,et al.  Acute Increase in O-GlcNAc Improves Survival in Mice With LPS-Induced Systemic Inflammatory Response Syndrome , 2020, Frontiers in Physiology.

[37]  K. Nicolaides,et al.  Placental function and fetal weight are associated with maternal hemodynamic indices in uncomplicated pregnancies at 35-37 weeks gestation. , 2020, American journal of obstetrics and gynecology.

[38]  B. Yan,et al.  O‑GlcNAc modification influences endometrial receptivity by promoting endometrial cell proliferation, migration and invasion. , 2019, Oncology reports.

[39]  Huixia Yang,et al.  Preconception diabetes mellitus and adverse pregnancy outcomes in over 6.4 million women: A population-based cohort study in China , 2019, PLoS medicine.

[40]  T. Nagy,et al.  Hyperglycemia-Induced Aberrant Cell Proliferation; A Metabolic Challenge Mediated by Protein O-GlcNAc Modification , 2019, Cells.

[41]  Dong Hoon Lee,et al.  Metformin inhibits cervical cancer cell proliferation via decreased AMPK O-GlcNAcylation , 2019, Animal cells and systems.

[42]  J. James,et al.  Human placenta and trophoblast development: key molecular mechanisms and model systems , 2019, Cellular and Molecular Life Sciences.

[43]  C. Slawson,et al.  Disruption of O‐GlcNAc homeostasis during mammalian oocyte meiotic maturation impacts fertilization , 2019, Molecular reproduction and development.

[44]  G. Osol,et al.  Plasticity of the Maternal Vasculature During Pregnancy. , 2019, Annual review of physiology.

[45]  E. Alejandro,et al.  Nutrient sensor signaling pathways and cellular stress in fetal growth restriction. , 2019, Journal of molecular endocrinology.

[46]  V. V. Lima,et al.  O-GlcNAc Modification During Pregnancy: Focus on Placental Environment , 2018, Front. Physiol..

[47]  C. Slawson,et al.  Real Talk: The Inter-play Between the mTOR, AMPK, and Hexosamine Biosynthetic Pathways in Cell Signaling , 2018, Front. Endocrinol..

[48]  D. Hill Placental control of metabolic adaptations in the mother for an optimal pregnancy outcome. What goes wrong in gestational diabetes? , 2018, Placenta.

[49]  T. Napso,et al.  The Role of Placental Hormones in Mediating Maternal Adaptations to Support Pregnancy and Lactation , 2018, Front. Physiol..

[50]  C. Epperson,et al.  Placental H3K27me3 establishes female resilience to prenatal insults , 2018, Nature Communications.

[51]  V. V. Lima,et al.  O‐linked N‐acetyl‐glucosamine deposition in placental proteins varies according to maternal glycemic levels , 2018, Life sciences.

[52]  Reca Alejandra,et al.  The blocking of aquaporin-3 (AQP3) impairs extravillous trophoblast cell migration. , 2018, Biochemical and biophysical research communications.

[53]  G. Hart Nutrient regulation of signaling and transcription , 2018, The Journal of Biological Chemistry.

[54]  Rheure Alves-Lopes,et al.  Glycosylation with O-linked β-N-acetylglucosamine induces vascular dysfunction via production of superoxide anion/reactive oxygen species. , 2018, Canadian journal of physiology and pharmacology.

[55]  Mark R. Johnson,et al.  Progesterone, the maternal immune system and the onset of parturition in the mouse† , 2018, Biology of Reproduction.

[56]  N. Packer,et al.  Periconception onset diabetes is associated with embryopathy and fetal growth retardation, reproductive tract hyperglycosylation and impaired immune adaptation to pregnancy , 2018, Scientific Reports.

[57]  J. Brosens,et al.  The Glycosyltransferase EOGT Regulates Adropin Expression in Decidualizing Human Endometrium , 2018, Endocrinology.

[58]  B. Viollet,et al.  AMPK activation counteracts cardiac hypertrophy by reducing O-GlcNAcylation , 2018, Nature Communications.

[59]  Zi-jiang Chen,et al.  Downregulation of decidual SP1 and P300 is associated with severe preeclampsia. , 2017, Journal of molecular endocrinology.

[60]  B. Emerald,et al.  Higher O-GlcNAc Levels Are Associated with Defects in Progenitor Proliferation and Premature Neuronal Differentiation during in-Vitro Human Embryonic Cortical Neurogenesis , 2017, Front. Cell. Neurosci..

[61]  K. Moritz,et al.  Maternal growth restriction and stress exposure in rats differentially alters expression of components of the placental glucocorticoid barrier and nutrient transporters. , 2017, Placenta.

[62]  L. Wells,et al.  O-GlcNAc transferase regulates transcriptional activity of human Oct4 , 2017, Glycobiology.

[63]  Weiwei Song,et al.  Moderate mammalian target of rapamycin inhibition induces autophagy in HTR8/SVneo cells via O‐linked β‐N‐acetylglucosamine signaling , 2017, The journal of obstetrics and gynaecology research.

[64]  E. A. Reece,et al.  Impact of protein O-GlcNAcylation on neural tube malformation in diabetic embryopathy , 2017, Scientific Reports.

[65]  V. V. Lima,et al.  Increased O-Linked N-Acetylglucosamine Modification of NF-ΚB and Augmented Cytokine Production in the Placentas from Hyperglycemic Rats , 2017, Inflammation.

[66]  Jennifer J. Kohler,et al.  Chemical Modulation of Protein O-GlcNAcylation via OGT Inhibition Promotes Human Neural Cell Differentiation. , 2017, ACS chemical biology.

[67]  M. Nitert,et al.  Review: Placental transport and metabolism of energy substrates in maternal obesity and diabetes. , 2017, Placenta.

[68]  M. Pantaleon,et al.  Placental O-GlcNAc-transferase expression and interactions with the glucocorticoid receptor are sex specific and regulated by maternal corticosterone exposure in mice , 2017, Scientific Reports.

[69]  M. Paidas Novel Therapy for the Treatment of Early-Onset Preeclampsia , 2017, Clinical obstetrics and gynecology.

[70]  R. Dagda,et al.  Mitochondrial O-GlcNAc Transferase (mOGT) Regulates Mitochondrial Structure, Function, and Survival in HeLa Cells* , 2017, The Journal of Biological Chemistry.

[71]  Xiaodong Liu,et al.  The Roles of Mitochondria in Autophagic Cell Death. , 2016, Cancer biotherapy & radiopharmaceuticals.

[72]  A. Fowden,et al.  Placental metabolism: substrate requirements and the response to stress. , 2016, Reproduction in domestic animals = Zuchthygiene.

[73]  Satoshi Tanaka,et al.  Novel O-GlcNAcylation on Ser40 of canonical H2A isoforms specific to viviparity , 2016, Scientific Reports.

[74]  A. Aliverti,et al.  Respiratory physiology of pregnancy , 2015, Breathe.

[75]  S. Lehnert,et al.  The histone variant H2A.Z is dynamically expressed in the developing mouse placenta and in differentiating trophoblast stem cells. , 2015, Placenta.

[76]  A. Makino,et al.  O-GlcNAcase overexpression reverses coronary endothelial cell dysfunction in type 1 diabetic mice. , 2015, American journal of physiology. Cell physiology.

[77]  Eran Hadar,et al.  The International Federation of Gynecology and Obstetrics (FIGO) Initiative on gestational diabetes mellitus: A pragmatic guide for diagnosis, management, and care # , 2015, International journal of gynaecology and obstetrics: the official organ of the International Federation of Gynaecology and Obstetrics.

[78]  S. Fisher Why is placentation abnormal in preeclampsia? , 2015, American journal of obstetrics and gynecology.

[79]  S. Ryu,et al.  O-GlcNAc cycling enzymes control vascular development of the placenta by modulating the levels of HIF-1α. , 2015, Placenta.

[80]  J. Hanover,et al.  A little sugar goes a long way: The cell biology of O-GlcNAc , 2015, The Journal of cell biology.

[81]  R. Pijnenborg,et al.  The role of invasive trophoblast in implantation and placentation of primates , 2015, Philosophical Transactions of the Royal Society B: Biological Sciences.

[82]  O. Gavrilova,et al.  Conditional Knock-out Reveals a Requirement for O-Linked N-Acetylglucosaminase (O-GlcNAcase) in Metabolic Homeostasis* , 2015, The Journal of Biological Chemistry.

[83]  J. Hanover,et al.  X marks the spot: does it matter that O-GlcNAc transferase is an X-linked gene? , 2014, Biochemical and biophysical research communications.

[84]  G. Barton,et al.  Elevated O‐GlcNAc Levels Activate Epigenetically Repressed Genes and Delay Mouse ESC Differentiation Without Affecting Naïve to Primed Cell Transition , 2014, Stem cells.

[85]  D. Vocadlo,et al.  O-GlcNAc and neurodegeneration: biochemical mechanisms and potential roles in Alzheimer's disease and beyond. , 2014, Chemical Society reviews.

[86]  K. Adamo,et al.  Maternal–Fetal Nutrient Transport in Pregnancy Pathologies: The Role of the Placenta , 2014, International journal of molecular sciences.

[87]  R. Webb,et al.  Vascular O-GlcNAcylation augments reactivity to constrictor stimuli by prolonging phosphorylated levels of the myosin light chain , 2014, Brazilian journal of medical and biological research = Revista brasileira de pesquisas medicas e biologicas.

[88]  T. Powell,et al.  The Role of Placental Nutrient Sensing in Maternal-Fetal Resource Allocation1 , 2014, Biology of reproduction.

[89]  G. Hart,et al.  Nutrient regulation of signaling, transcription, and cell physiology by O-GlcNAcylation. , 2014, Cell metabolism.

[90]  T. Bale,et al.  Targeted placental deletion of OGT recapitulates the prenatal stress phenotype including hypothalamic mitochondrial dysfunction , 2014, Proceedings of the National Academy of Sciences.

[91]  D. Russell,et al.  Hyperglycaemic conditions perturb mouse oocyte in vitro developmental competence via beta-O-linked glycosylation of heat shock protein 90. , 2014, Human reproduction.

[92]  J. Shabanowitz,et al.  Cross-talk between Two Essential Nutrient-sensitive Enzymes , 2014, The Journal of Biological Chemistry.

[93]  Yong Sun,et al.  Activation of AKT by O-Linked N-Acetylglucosamine Induces Vascular Calcification in Diabetes Mellitus , 2014, Circulation research.

[94]  Junfeng Ma,et al.  Protein O-GlcNAcylation in diabetes and diabetic complications , 2013, Expert review of proteomics.

[95]  Dan Liu,et al.  Ten-Eleven Translocation 1 (Tet1) Is Regulated by O-Linked N-Acetylglucosamine Transferase (Ogt) for Target Gene Repression in Mouse Embryonic Stem Cells* , 2013, The Journal of Biological Chemistry.

[96]  T. Bale,et al.  O-GlcNAc transferase (OGT) as a placental biomarker of maternal stress and reprogramming of CNS gene transcription in development , 2013, Proceedings of the National Academy of Sciences.

[97]  I. Cameron,et al.  Metabolic Induction and Early Responses of Mouse Blastocyst Developmental Programming following Maternal Low Protein Diet Affecting Life-Long Health , 2012, PloS one.

[98]  S. Oparil,et al.  Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction. , 2012, American journal of physiology. Heart and circulatory physiology.

[99]  T. Bale,et al.  Prenatal programing: At the intersection of maternal stress and immune activation , 2012, Hormones and Behavior.

[100]  U. Elkayam,et al.  Physiologic changes during normal pregnancy and delivery. , 2012, Cardiology clinics.

[101]  M. Longtine,et al.  Survival by self-destruction: a role for autophagy in the placenta? , 2012, Placenta.

[102]  Kathryn M. Spitler,et al.  O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter? , 2012, Clinical science.

[103]  D. Steinberg,et al.  Cell Free Expression of hif1α and p21 in Maternal Peripheral Blood as a Marker for Preeclampsia and Fetal Growth Restriction , 2012, PloS one.

[104]  B. Sibai,et al.  Maternal mortality from preeclampsia/eclampsia. , 2012, Seminars in perinatology.

[105]  Seong-Yun Jeong,et al.  Glucosamine increases vascular contraction through activation of RhoA/Rho kinase pathway in isolated rat aorta. , 2011, BMB reports.

[106]  G. Hart,et al.  Cross talk between O-GlcNAcylation and phosphorylation: roles in signaling, transcription, and chronic disease. , 2011, Annual review of biochemistry.

[107]  Z. Fortes,et al.  O-GlcNAcylation contributes to the vascular effects of ET-1 via activation of the RhoA/Rho-kinase pathway. , 2011, Cardiovascular research.

[108]  M. Pantaleon,et al.  Toxic Effects of Hyperglycemia Are Mediated by the Hexosamine Signaling Pathway and O-Linked Glycosylation in Early Mouse Embryos1 , 2010, Biology of reproduction.

[109]  J. Hanover,et al.  The hexosamine signaling pathway: O-GlcNAc cycling in feast or famine. , 2010, Biochimica et biophysica acta.

[110]  G. Hart,et al.  The intersections between O-GlcNAcylation and phosphorylation: implications for multiple signaling pathways , 2010, Journal of Cell Science.

[111]  J. Dennis,et al.  Metabolism, Cell Surface Organization, and Disease , 2009, Cell.

[112]  Z. Fortes,et al.  Impaired Vasodilator Activity in Deoxycorticosterone Acetate-Salt Hypertension Is Associated With Increased Protein O-GlcNAcylation , 2009, Hypertension.

[113]  Peng-Hui Wang,et al.  Placentation abnormalities in the pathophysiology of preeclampsia , 2009, Expert review of molecular diagnostics.

[114]  E. Kang,et al.  NFκB activation is associated with its O-GlcNAcylation state under hyperglycemic conditions , 2008, Proceedings of the National Academy of Sciences.

[115]  G. Hart,et al.  Regulation of the O-Linked β-N-Acetylglucosamine Transferase by Insulin Signaling* , 2008, Journal of Biological Chemistry.

[116]  S. Oparil,et al.  Increased protein O-GlcNAc modification inhibits inflammatory and neointimal responses to acute endoluminal arterial injury. , 2008, American journal of physiology. Heart and circulatory physiology.

[117]  P. Catalano,et al.  Metabolic changes in pregnancy. , 2007, Clinical obstetrics and gynecology.

[118]  E. Seely,et al.  Chronic hypertension in pregnancy. , 2007, Circulation.

[119]  J. Hanover,et al.  The Hexosamine Signaling Pathway: Deciphering the "O-GlcNAc Code" , 2005, Science's STKE.

[120]  S. Fisher,et al.  Abnormal placentation and the syndrome of preeclampsia. , 2004, Seminars in nephrology.

[121]  R. Ness,et al.  Evidence for the functional activity of hypoxia-inducible transcription factors overexpressed in preeclamptic placentae. , 2004, Placenta.

[122]  G. Hart,et al.  Dynamic O-GlcNAc Modification of Nucleocytoplasmic Proteins in Response to Stress , 2004, Journal of Biological Chemistry.

[123]  G. Hart,et al.  O-GlcNAc a sensor of cellular state: the role of nucleocytoplasmic glycosylation in modulating cellular function in response to nutrition and stress. , 2004, Biochimica et biophysica acta.

[124]  Haiyang Tang,et al.  Inhibition of the activating signals in NK92 cells by recombinant GST-sHLA-G1α chain , 2004, Cell Research.

[125]  R. Soundararajan,et al.  Trophoblast 'pseudo-tumorigenesis': Significance and contributory factors , 2004, Reproductive biology and endocrinology : RB&E.

[126]  G. Hart,et al.  Ogt-Dependent X-Chromosome-Linked Protein Glycosylation Is a Requisite Modification in Somatic Cell Function and Embryo Viability , 2004, Molecular and Cellular Biology.

[127]  Xiaoyong Yang,et al.  O-GlcNAc Modification Is an Endogenous Inhibitor of the Proteasome , 2003, Cell.

[128]  S. Taler,et al.  Hypertension in pregnancy. , 2002, Journal of the American Society of Hypertension : JASH.

[129]  J. Winter,et al.  Adriana and Luisa Castellucci Award lecture 2001. Hypoxia inducible factor-1: oxygen regulation of trophoblast differentiation in normal and pre-eclamptic pregnancies--a review. , 2002, Placenta.

[130]  G. Hart,et al.  The O-GlcNAc transferase gene resides on the X chromosome and is essential for embryonic stem cell viability and mouse ontogeny. , 2000, Proceedings of the National Academy of Sciences of the United States of America.

[131]  D. Crane,et al.  The Journal of Clinical Endocrinology & Metabolism Printed in U.S.A. Copyright © 2000 by The Endocrine Society Placental Growth Hormone (GH), GH-Binding Protein, and Insulin-Like Growth Factor Axis in Normal, Growth- Retarded, and Diabetic Pregnancies: Co , 2022 .

[132]  B. Imperiali,et al.  Effect of N-linked glycosylation on glycopeptide and glycoprotein structure. , 1999, Current opinion in chemical biology.

[133]  J. E. Kudlow,et al.  O glycosylation of an Sp1-derived peptide blocks known Sp1 protein interactions , 1997, Molecular and cellular biology.

[134]  I. Han,et al.  Reduced O glycosylation of Sp1 is associated with increased proteasome susceptibility , 1997, Molecular and cellular biology.

[135]  C. Redman Pre-eclampsia and the placenta , 1991 .

[136]  G. Hart,et al.  Enzymatic addition of O-GlcNAc to nuclear and cytoplasmic proteins. Identification of a uridine diphospho-N-acetylglucosamine:peptide beta-N-acetylglucosaminyltransferase. , 1990, The Journal of biological chemistry.

[137]  G. Hart,et al.  Topography and polypeptide distribution of terminal N-acetylglucosamine residues on the surfaces of intact lymphocytes. Evidence for O-linked GlcNAc. , 1984, The Journal of biological chemistry.

[138]  G. Meschia,et al.  Principal substrates of fetal metabolism. , 1978, Physiological reviews.

[139]  M. Sasaki,et al.  Preferential inactivation of the paternally derived X chromosome in the extraembryonic membranes of the mouse , 1975, Nature.

[140]  M. Lyon Gene Action in the X-chromosome of the Mouse (Mus musculus L.) , 1961, Nature.

[141]  Q. Fu,et al.  Hemodynamic and Electrocardiographic Aspects of Uncomplicated Singleton Pregnancy. , 2018, Advances in experimental medicine and biology.

[142]  V. Regitz-Zagrosek,et al.  New frontiers in heart hypertrophy during pregnancy. , 2012, American journal of cardiovascular disease.

[143]  T. Zorn,et al.  Extracellular matrix of the mouse endometrium during decidualization. , 1991, Memorias do Instituto Oswaldo Cruz.

[144]  H. Ljunggren,et al.  In search of the 'missing self': MHC molecules and NK cell recognition. , 1990, Immunology today.

[145]  Virgil F. Fairbanks,et al.  THE NORMAL HUMAN FEMALE AS A MOSAIC OF X-CHROMOSOME ACTIVITY: STUDIES USING THE GENE FOR G-6-PD-DEFICIENCY AS A MARKER , 1962 .