β-Glucans inhibit intracellular growth of Mycobacterium bovis BCG but not virulent Mycobacterium tuberculosis in human macrophages.

[1]  Y. Iwakura,et al.  Differential pathways regulating innate and adaptive antitumor immune responses by particulate and soluble yeast-derived β-glucans. , 2011, Blood.

[2]  Jun Ma,et al.  Activation of the innate immune receptor Dectin-1 upon formation of a “phagocytic synapse” , 2011, Nature.

[3]  J. Banchereau,et al.  Concomitant Activation and Antigen Uptake via Human Dectin-1 Results in Potent Antigen-Specific CD8+ T Cell Responses , 2010, The Journal of Immunology.

[4]  H. Wolff,et al.  (1,3)-β-Glucans Activate Both Dectin-1 and NLRP3 Inflammasome in Human Macrophages , 2010, The Journal of Immunology.

[5]  Stefano Dusi,et al.  Induction of Th1/Th17 immune response by Mycobacterium tuberculosis: role of dectin‐1, mannose receptor, and DC‐SIGN , 2009, Journal of leukocyte biology.

[6]  I. Fichtner,et al.  Oral administration of a soluble 1-3, 1-6 beta-glucan during prophylactic survivin peptide vaccination diminishes growth of a B cell lymphoma in mice. , 2009, International immunopharmacology.

[7]  D. Underhill,et al.  β‐glucan recognition by the innate immune system , 2009, Immunological reviews.

[8]  Sergio Grinstein,et al.  Antimicrobial mechanisms of phagocytes and bacterial evasion strategies , 2009, Nature Reviews Microbiology.

[9]  E. Stuyven,et al.  Effect of beta-glucans on an ETEC infection in piglets. , 2009, Veterinary immunology and immunopathology.

[10]  N. Gow,et al.  Pattern recognition: recent insights from Dectin-1 , 2009, Current opinion in immunology.

[11]  Gordon D. Brown,et al.  β‐Glucans and Dectin‐1 , 2008, Annals of the New York Academy of Sciences.

[12]  David L. Williams,et al.  β-Glucan Activates Microglia without Inducing Cytokine Production in Dectin-1-Dependent Manner , 2008, The Journal of Immunology.

[13]  J. Schorey,et al.  The pattern recognition receptor Dectin-1: from fungi to mycobacteria. , 2008, Current drug targets.

[14]  Jun Yan,et al.  Yeast glucan particles activate murine resident macrophages to secrete proinflammatory cytokines via MyD88- and Syk kinase-dependent pathways. , 2007, Clinical immunology.

[15]  M. Yadav,et al.  The beta-glucan receptor dectin-1 functions together with TLR2 to mediate macrophage activation by mycobacteria. , 2006, Blood.

[16]  Toshio Suzuki,et al.  Antitumor and antimetastatic activity of a novel water-soluble low molecular weight beta-1, 3-D-glucan (branch beta-1,6) isolated from Aureobasidium pullulans 1A1 strain black yeast. , 2006, Anticancer research.

[17]  Jennifer L. Martin,et al.  Surfactant Protein D Increases Fusion of Mycobacterium tuberculosis- Containing Phagosomes with Lysosomes in Human Macrophages , 2006, Infection and Immunity.

[18]  Y. Adachi,et al.  Mechanism of Enhanced Hematopoietic Response by Soluble β‐Glucan SCG in Cyclophosphamide‐Treated Mice , 2006, Microbiology and immunology.

[19]  D. Li,et al.  Effects of β-glucan extracted from Saccharomyces cerevisiae on growth performance, and immunological and somatotropic responses of pigs challenged with Escherichia coli lipopolysaccharide1 , 2006 .

[20]  J. Albina,et al.  The effect of PGG‐β‐glucan on neutrophil chemotaxis in vivo , 2006 .

[21]  A. Azad,et al.  The human macrophage mannose receptor directs Mycobacterium tuberculosis lipoarabinomannan-mediated phagosome biogenesis , 2005, The Journal of experimental medicine.

[22]  Jun Yan,et al.  Yeast whole glucan particle (WGP) β-glucan in conjunction with antitumour monoclonal antibodies to treat cancer , 2005, Expert opinion on biological therapy.

[23]  J. Zweier,et al.  Pulmonary Surfactant Protein A Inhibits Macrophage Reactive Oxygen Intermediate Production in Response to Stimuli by Reducing NADPH Oxidase Activity1 , 2004, The Journal of Immunology.

[24]  S. Gordon,et al.  Dectin-1 Mediates the Biological Effects of β-Glucans , 2003, The Journal of experimental medicine.

[25]  D. Voelker,et al.  Pulmonary Surfactant Protein A Up-Regulates Activity of the Mannose Receptor, a Pattern Recognition Receptor Expressed on Human Macrophages1 , 2002, The Journal of Immunology.

[26]  S. Gordon,et al.  The β-Glucan Receptor, Dectin-1, Is Predominantly Expressed on the Surface of Cells of the Monocyte/Macrophage and Neutrophil Lineages1 , 2002, The Journal of Immunology.

[27]  G. Hetland,et al.  beta-1,3-Glucan reduces growth of Mycobacterium tuberculosis in macrophage cultures. , 2002, FEMS immunology and medical microbiology.

[28]  B. Britigan,et al.  Gallium Disrupts Iron Metabolism of Mycobacteria Residing within Human Macrophages , 2000, Infection and Immunity.

[29]  N. Ohno,et al.  Protective effect of beta-glucan against systemic Streptococcus pneumoniae infection in mice. , 2000, FEMS immunology and medical microbiology.

[30]  D. Flaherty,et al.  Dihydrofluorescein diacetate is superior for detecting intracellular oxidants: comparison with 2',7'-dichlorodihydrofluorescein diacetate, 5(and 6)-carboxy-2',7'-dichlorodihydrofluorescein diacetate, and dihydrorhodamine 123. , 1999, Free radical biology & medicine.

[31]  T. Mayadas,et al.  The beta-glucan-binding lectin site of mouse CR3 (CD11b/CD18) and its function in generating a primed state of the receptor that mediates cytotoxic activation in response to iC3b-opsonized target cells. , 1999, Journal of immunology.

[32]  D. Melican,et al.  Enhanced clearance of a multiple antibiotic resistant Staphylococcus aureus in rats treated with PGG-glucan is associated with increased leukocyte counts and increased neutrophil oxidative burst activity. , 1998, International journal of immunopharmacology.

[33]  K. Lemerise,et al.  Activation of rat macrophages by Betafectin PGG‐glucan requires cross‐linking of membrane receptors distinct from complement receptor three (CR3) , 1998, Journal of leukocyte biology.

[34]  Hetland,et al.  Protective Effect of β‐Glucan Against Mycobacterium bovis, BCG Infection in BALB/c Mice , 1998, Scandinavian journal of immunology.

[35]  L. Schlesinger Macrophage phagocytosis of virulent but not attenuated strains of Mycobacterium tuberculosis is mediated by mannose receptors in addition to complement receptors. , 1993, Journal of immunology.

[36]  M. Horwitz,et al.  Phagocytosis of Mycobacterium tuberculosis is mediated by human monocyte complement receptors and complement component C3. , 1990, Journal of immunology.

[37]  M. Fenton,et al.  Receptor-Mediated Recognition of Mycobacterium tuberculosis by Host Cells , 2005 .

[38]  Stewart T. Cole,et al.  Tuberculosis and the tubercle bacillus. , 2005 .