Doxorubicin induces mitochondrial permeability transition and contractile dysfunction in the human myocardium.
暂无分享,去创建一个
S. Lancel | R. Neviere | T. Modine | S. Préau | G. Fayad | D. Montaigne | X. Maréchal | Riadh Baccouch
[1] P. Pasdois,et al. The role of the mitochondrial permeability transition pore in heart disease. , 2009, Biochimica et biophysica acta.
[2] P. Oliveira,et al. Doxorubicin increases the susceptibility of brain mitochondria to Ca(2+)-induced permeability transition and oxidative damage. , 2008, Free radical biology & medicine.
[3] L. Matsubara,et al. Anthracycline-induced cardiotoxicity. , 2008, Cardiovascular & hematological agents in medicinal chemistry.
[4] R. Margreiter,et al. Analysis of mitochondrial function in situ in permeabilized muscle fibers, tissues and cells , 2008, Nature Protocols.
[5] K. Wallace. Adriamycin-induced interference with cardiac mitochondrial calcium homeostasis , 2007, Cardiovascular Toxicology.
[6] P. Bernardi,et al. The mitochondrial permeability transition pore. , 2007, Novartis Foundation symposium.
[7] T. Wallimann,et al. New insights into doxorubicin-induced cardiotoxicity: the critical role of cellular energetics. , 2006, Journal of molecular and cellular cardiology.
[8] R. Favory,et al. Myocardial dysfunction and potential cardiac hypoxia in rats induced by carbon monoxide inhalation. , 2006, American journal of respiratory and critical care medicine.
[9] R. Favory,et al. Inhibition of mitochondrial permeability transition prevents sepsis-induced myocardial dysfunction and mortality. , 2006, Journal of the American College of Cardiology.
[10] K. Wallace,et al. Adriamycin-induced oxidative mitochondrial cardiotoxicity , 2006, Cell Biology and Toxicology.
[11] T. Wallimann,et al. Acute toxicity of doxorubicin on isolated perfused heart: response of kinases regulating energy supply. , 2005, American journal of physiology. Heart and circulatory physiology.
[12] P. Oliveira,et al. Are the antioxidant properties of carvedilol important for the protection of cardiac mitochondria? , 2005, Current vascular pharmacology.
[13] L. Gianni,et al. Anthracyclines: Molecular Advances and Pharmacologic Developments in Antitumor Activity and Cardiotoxicity , 2004, Pharmacological Reviews.
[14] S. Javadov,et al. Mitochondrial permeability transition pore opening during myocardial reperfusion--a target for cardioprotection. , 2004, Cardiovascular research.
[15] E. Voest,et al. Doxorubicin and mechanical performance of cardiac trabeculae after acute and chronic treatment: a review. , 2001, European journal of pharmacology.
[16] Shaoyu Zhou,et al. Cumulative and irreversible cardiac mitochondrial dysfunction induced by doxorubicin. , 2001, Cancer research.
[17] A. Harken,et al. Tumor necrosis factor-alpha and interleukin-1beta synergistically depress human myocardial function. , 1999, Critical care medicine.
[18] Hung-Yi Wu,et al. Molecular Mechanisms of Doxorubicin-induced Cardiomyopathy , 1997, The Journal of Biological Chemistry.
[19] K. Wallace,et al. Dose-dependent increase in sensitivity to calcium-induced mitochondrial dysfunction and cardiomyocyte cell injury by doxorubicin. , 1996, Journal of molecular and cellular cardiology.
[20] A. Allen,et al. The cardiotoxicity of chemotherapeutic drugs. , 1992, Seminars in oncology.
[21] U. Andersson,et al. Effects of FK506 and cyclosporin A on cytokine production studied in vitro at a single-cell level. , 1992, Immunology.
[22] G. Krishna,et al. Daunomycin inhibits the uptake of adenine, amino acids, and glucose into cardiac myocytes. , 1987, Toxicology and applied pharmacology.