A systematic review of associations between functional MRI activity and polygenic risk for schizophrenia and bipolar disorder
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Danai Dima | D. Dima | S. Ranlund | M. Kyriakopoulos | S. Williams | Zalina Dezhina | Marinos Kyriakopoulos | Zalina Dezhina | Siri Ranlund | Steve C R Williams
[1] Daniel R Weinberger,et al. Mapping DNA methylation across development, genotype, and schizophrenia in the human frontal cortex , 2015, Nature Neuroscience.
[2] Melissa J. Green,et al. The cognitive and neurophysiological basis of emotion dysregulation in bipolar disorder. , 2007, Journal of affective disorders.
[3] Benjamin S Aribisala,et al. Novel genetic loci associated with hippocampal volume , 2017, Nature Communications.
[4] Fabio Sambataro,et al. Nonlinear response of the anterior cingulate and prefrontal cortex in schizophrenia as a function of variable attentional control. , 2010, Cerebral cortex.
[5] D. Collier,et al. The impact of the Val158Met catechol-O-methyltransferase genotype on neural correlates of sad facial affect processing in patients with bipolar disorder and their relatives , 2010, Psychological Medicine.
[6] R. Shelton,et al. Brain structure, function, and neurochemistry in schizophrenia and bipolar disorder—a systematic review of the magnetic resonance neuroimaging literature , 2017, npj Schizophrenia.
[7] B. Kerner. Toward a Deeper Understanding of the Genetics of Bipolar Disorder , 2015, Front. Psychiatry.
[8] V. Menon,et al. Saliency, switching, attention and control: a network model of insula function , 2010, Brain Structure and Function.
[9] Kjell Elenius,et al. Emotion Recognition , 2009, Computers in the Human Interaction Loop.
[10] Gerome Breen,et al. The polygenic risk for bipolar disorder influences brain regional function relating to visual and default state processing of emotional information , 2016, NeuroImage: Clinical.
[11] Disorder Working Group. Large-scale genome-wide association analysis of bipolar disorder identifies a new susceptibility locus near ODZ4 , 2012, Nature Genetics.
[12] Simon C. Potter,et al. MiR-137-derived polygenic risk: effects on cognitive performance in patients with schizophrenia and controls , 2017, Translational Psychiatry.
[13] D. Collier,et al. The impact of the Val 158 Met catechol-O-methyltransferase genotype on neural correlates of sad facial affect processing in patients with bipolar disorder and their relatives , 2010 .
[14] Vince D. Calhoun,et al. Imaging Genetics and Genomics in Psychiatry: A Critical Review of Progress and Potential , 2017, Biological Psychiatry.
[15] C. Spencer,et al. Biological Insights From 108 Schizophrenia-Associated Genetic Loci , 2014, Nature.
[16] T. Wassink,et al. Prefrontal inefficiency is associated with polygenic risk for schizophrenia. , 2014, Schizophrenia bulletin.
[17] E. Leibenluft,et al. Facial emotion labeling deficits in children and adolescents at risk for bipolar disorder. , 2008, The American journal of psychiatry.
[18] Lars T. Westlye,et al. Altered Brain Activation during Emotional Face Processing in Relation to Both Diagnosis and Polygenic Risk of Bipolar Disorder , 2015, PloS one.
[19] S Cichon,et al. Effects of a genome-wide supported psychosis risk variant on neural activation during a theory-of-mind task , 2011, Molecular Psychiatry.
[20] L. Uddin. Salience processing and insular cortical function and dysfunction , 2014, Nature Reviews Neuroscience.
[21] Kalervo Kiianmaa,et al. Enhanced Extracellular Glutamate and Dopamine in the Ventral Pallidum of Alcohol-Preferring AA and Alcohol-Avoiding ANA Rats after Morphine , 2015, Front. Psychiatry.
[22] Scott R Sponheim,et al. Cumulative genetic risk and prefrontal activity in patients with schizophrenia. , 2013, Schizophrenia bulletin.
[23] E. Bullmore,et al. A quantitative meta-analysis of fMRI studies in bipolar disorder. , 2011, Bipolar disorders.
[24] Lars T Westlye,et al. Polygenic risk for schizophrenia associated with working memory-related prefrontal brain activation in patients with schizophrenia and healthy controls. , 2015, Schizophrenia bulletin.
[25] E. Bora,et al. Social cognition in schizophrenia in comparison to bipolar disorder: A meta-analysis , 2016, Schizophrenia Research.
[26] P. McGuire,et al. Ventral Striatal Activation During Reward Processing in Psychosis: A Neurofunctional Meta-Analysis. , 2015, JAMA psychiatry.
[27] Tianzi Jiang,et al. Polygenic risk for five psychiatric disorders and cross-disorder and disorder-specific neural connectivity in two independent populations , 2017, NeuroImage: Clinical.
[28] Paul M. Thompson,et al. Emerging Global Initiatives in Neurogenetics: The Enhancing Neuroimaging Genetics through Meta-analysis (ENIGMA) Consortium , 2017, Neuron.
[29] Peter F. Liddle,et al. Aberrant salience network (bilateral insula and anterior cingulate cortex) connectivity during information processing in schizophrenia , 2010, Schizophrenia Research.
[30] John G. Csernansky,et al. Prefrontal Cortex Function in Nonpsychotic Siblings of Individuals with Schizophrenia , 2008, Biological Psychiatry.
[31] Tilo Kircher,et al. Association of rs1006737 in CACNA1C with alterations in prefrontal activation and fronto‐hippocampal connectivity , 2014, Human brain mapping.
[32] G. Pergola,et al. A Polygenic Risk Score of glutamatergic SNPs associated with schizophrenia predicts attentional behavior and related brain activity in healthy humans , 2017, European Neuropsychopharmacology.
[33] Melissa J. Green,et al. Genome-wide association study identifies 30 Loci Associated with Bipolar Disorder , 2017, bioRxiv.
[34] Andreas Meyer-Lindenberg,et al. Imaging genetics: Progressing by leaps and bounds , 2010, NeuroImage.
[35] E. Bora,et al. Theory of mind impairment in schizophrenia: Meta-analysis , 2009, Schizophrenia Research.
[36] Nick Craddock,et al. Psychosis genetics: modeling the relationship between schizophrenia, bipolar disorder, and mixed (or "schizoaffective") psychoses. , 2009, Schizophrenia bulletin.
[37] Paul M. Thompson,et al. Neuroimaging genomics in psychiatry—a translational approach , 2017, Genome Medicine.
[38] J. Marchini,et al. Genome-wide association studies of brain structure and function 1 in the UK Biobank 2 , 2018 .
[39] Janice M. Fullerton,et al. Genome-wide association study reveals two new risk loci for bipolar disorder , 2014, Nature Communications.
[40] P. McGuffin,et al. Polygenic Heterogeneity: A Complex Model of Genetic Inheritance in Psychiatric Disorders , 2010, Biological Psychiatry.
[41] Mohamed Saoud,et al. Emotion recognition and genetic vulnerability to schizophrenia. , 2007, The British journal of psychiatry : the journal of mental science.
[42] M. Ruggeri,et al. Hippocampal Subfield Volumes in Patients With First-Episode Psychosis , 2018, Schizophrenia bulletin.
[43] K. Marwick,et al. Social cognition in schizophrenia: a review of face processing. , 2008, British medical bulletin.
[44] H. Luhmann,et al. Local circuits targeting parvalbumin-containing interneurons in layer IV of rat barrel cortex , 2009, Brain Structure and Function.
[45] Charan Ranganath,et al. The Cognitive Neuroscience of Memory Function and Dysfunction in Schizophrenia , 2008, Biological Psychiatry.
[46] V. Calhoun,et al. Working memory circuitry in schizophrenia shows widespread cortical inefficiency and compensation , 2010, Schizophrenia Research.
[47] Daniel R Weinberger,et al. Intermediate phenotypes in psychiatric disorders. , 2011, Current opinion in genetics & development.
[48] P. Jackson,et al. Social cognition in first-degree relatives of people with schizophrenia: A meta-analysis , 2013, Psychiatry Research.
[49] Mark E. Schmidt,et al. The Alzheimer's Disease Neuroimaging Initiative: Progress report and future plans , 2010, Alzheimer's & Dementia.
[50] Michael F. Green,et al. Social cognition in schizophrenia , 2015, Nature Reviews Neuroscience.
[51] Thomas E. Nichols,et al. Common genetic variants influence human subcortical brain structures , 2015, Nature.
[52] F. Dudbridge. Power and Predictive Accuracy of Polygenic Risk Scores , 2013, PLoS genetics.
[53] P. Shekelle,et al. Preferred reporting items for systematic review and meta-analysis protocols (PRISMA-P) 2015: elaboration and explanation , 2016, British Medical Journal.
[54] Daniel R. Weinberger,et al. Imaging genetics—days of future past , 2010, NeuroImage.
[55] P. Elliott,et al. UK Biobank: An Open Access Resource for Identifying the Causes of a Wide Range of Complex Diseases of Middle and Old Age , 2015, PLoS medicine.
[56] Klaus P. Ebmeier,et al. Magnetic resonance imaging studies in bipolar disorder and schizophrenia: meta-analysis. , 2009, The British journal of psychiatry : the journal of mental science.
[57] Gerome Breen,et al. Polygenic risk scores in imaging genetics: Usefulness and applications , 2015, Journal of psychopharmacology.
[58] M. Owen,et al. Associations between polygenic risk for schizophrenia and brain function during probabilistic learning in healthy individuals , 2015, Human brain mapping.
[59] M. Hill,et al. Transcriptional consequences of schizophrenia candidate miR-137 manipulation in human neural progenitor cells , 2014, Schizophrenia Research.
[60] I. L. Nieuwenhuis,et al. The role of the ventromedial prefrontal cortex in memory consolidation , 2011, Behavioural Brain Research.
[61] S. Djurovic,et al. Polygenic dissection of diagnosis and clinical dimensions of bipolar disorder and schizophrenia , 2013, Molecular Psychiatry.
[62] Tyrone D. Cannon,et al. Common genetic determinants of schizophrenia and bipolar disorder in Swedish families: a population-based study , 2009, The Lancet.
[63] Patrick F Sullivan,et al. The Psychiatric GWAS Consortium: Big Science Comes to Psychiatry , 2010, Neuron.
[64] R. Murray,et al. Heritability estimates for psychotic disorders: the Maudsley twin psychosis series. , 1999, Archives of general psychiatry.
[65] Thomas E. Nichols,et al. Scanning the horizon: towards transparent and reproducible neuroimaging research , 2016, Nature Reviews Neuroscience.
[66] P. Visscher,et al. 10 Years of GWAS Discovery: Biology, Function, and Translation. , 2017, American journal of human genetics.
[67] A W Toga,et al. Working memory and DLPFC inefficiency in schizophrenia: the FBIRN study. , 2009, Schizophrenia bulletin.
[68] S. Cichon,et al. Evaluating Historical Candidate Genes for Schizophrenia , 2015, Molecular Psychiatry.
[69] A. Slachevsky,et al. Executive Functioning in Schizophrenia , 2013, Front. Psychiatry.
[70] A. Farmer,et al. Evaluation of the validity and utility of a transdiagnostic psychosis dimension encompassing schizophrenia and bipolar disorder. , 2016, The British journal of psychiatry : the journal of mental science.
[71] V. Calhoun,et al. Cross-Tissue Exploration of Genetic and Epigenetic Effects on Brain Gray Matter in Schizophrenia , 2017, Schizophrenia bulletin.
[72] J Radua,et al. The impact of CACNA1C allelic variation on effective connectivity during emotional processing in bipolar disorder , 2013, Molecular Psychiatry.
[73] J. Mill,et al. Methylation quantitative trait loci in the developing brain and their enrichment in schizophrenia-associated genomic regions , 2015, Nature neuroscience.
[74] T. Goldberg,et al. Episodic Memory in Schizophrenia , 2009, Neuropsychology Review.
[75] Lloyd T. Elliott,et al. The genetic basis of human brain structure and function: 1,262 genome-wide associations found from 3,144 GWAS of multimodal brain imaging phenotypes from 9,707 UK Biobank participants , 2017, bioRxiv.
[76] Andrew K. Martin,et al. Theory of mind and the social brain: implications for understanding the genetic basis of schizophrenia , 2014, Genes, brain, and behavior.
[77] Gerome Breen,et al. Psychiatric Genomics: An Update and an Agenda , 2017, bioRxiv.
[78] Marisa O. Hollinshead,et al. Identification of common variants associated with human hippocampal and intracranial volumes , 2012, Nature Genetics.
[79] J. Kaprio,et al. High concordance of bipolar I disorder in a nationwide sample of twins. , 2004, The American journal of psychiatry.
[80] R. Murray,et al. Pattern of neural responses to verbal fluency shows diagnostic specificity for schizophrenia and bipolar disorder , 2011, BMC psychiatry.
[81] Manuel A. R. Ferreira,et al. Large-scale genome-wide association analysis of bipolar disorder identifies a new susceptibility locus near ODZ4 , 2011, Nature Genetics.
[82] Ann K. Shinn,et al. Default mode network abnormalities in bipolar disorder and schizophrenia , 2010, Psychiatry Research: Neuroimaging.
[83] V. Menon,et al. A critical role for the right fronto-insular cortex in switching between central-executive and default-mode networks , 2008, Proceedings of the National Academy of Sciences.
[84] R. Kahn,et al. Diminishing striatal activation across adolescent development during reward anticipation in offspring of schizophrenia patients , 2016, Schizophrenia Research.
[85] P. Shekelle,et al. Preferred reporting items for systematic review and meta-analysis protocols (PRISMA-P) 2015: elaboration and explanation , 2015, BMJ : British Medical Journal.
[86] M. Lepage,et al. Episodic memory-related activation in schizophrenia: meta-analysis , 2005, British Journal of Psychiatry.
[87] Anders D. Børglum,et al. Genome-wide association study identifies five new schizophrenia loci , 2011, Nature Genetics.
[88] M. Owen,et al. Genetic Risk for Schizophrenia: Convergence on Synaptic Pathways Involved in Plasticity , 2015, Biological Psychiatry.
[89] S. Lawrie,et al. The influence of polygenic risk for bipolar disorder on neural activation assessed using fMRI , 2012, Translational Psychiatry.
[90] A. Meyer-Lindenberg,et al. Neuroimaging Intermediate Phenotypes of Executive Control Dysfunction in Schizophrenia. , 2016, Biological psychiatry. Cognitive neuroscience and neuroimaging.
[91] L. Altshuler,et al. Emotion processing and regulation in bipolar disorder: a review , 2012, Bipolar disorders.
[92] F Alfaro Almagro. The genetic basis of human brain structure and function: 1,262 genome-wide associations found from 3,144 GWAS of multimodal brain imaging phenotypes from 9,707 UK Biobank participants , 2017 .
[93] M Takeda,et al. Imaging Genetics and Psychiatric Disorders , 2015, Current molecular medicine.
[94] P. Visscher,et al. Common polygenic variation contributes to risk of schizophrenia and bipolar disorder , 2009, Nature.
[95] J. Kwon,et al. Decreased neural response for facial emotion processing in subjects with high genetic load for schizophrenia , 2016, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[96] D. Glahn,et al. Beyond hypofrontality: A quantitative meta‐analysis of functional neuroimaging studies of working memory in schizophrenia , 2005, Human brain mapping.
[97] M. Daly,et al. Identification of risk loci with shared effects on five major psychiatric disorders: a genome-wide analysis , 2013, The Lancet.
[98] Kenneth Hugdahl,et al. Neuroimage: Clinical Impact of Glutamate Levels on Neuronal Response and Cognitive Abilities in Schizophrenia , 2022 .
[99] Peter Kirsch,et al. Hippocampal and Frontolimbic Function as Intermediate Phenotype for Psychosis: Evidence from Healthy Relatives and a Common Risk Variant in CACNA1C , 2014, Biological Psychiatry.
[100] P. Pattison,et al. Emotion recognition via facial expression and affective prosody in schizophrenia: a methodological review. , 2002, Clinical psychology review.
[101] P. Shekelle,et al. Preferred reporting items for systematic review and meta-analysis protocols (PRISMA-P) 2015 statement , 2015, Systematic Reviews.
[102] Daniel H. Geschwind,et al. Genetics and genomics of psychiatric disease , 2015, Science.
[103] Heather C. Whalley,et al. Impact of cross-disorder polygenic risk on frontal brain activation with specific effect of schizophrenia risk , 2015, Schizophrenia Research.
[104] Thomas W. Mühleisen,et al. Further Evidence for the Impact of a Genome-Wide-Supported Psychosis Risk Variant in ZNF804A on the Theory of Mind Network , 2014, Neuropsychopharmacology.
[105] Jack Euesden,et al. PRSice: Polygenic Risk Score software , 2014, Bioinform..
[106] K Charlet,et al. Functional neuroimaging effects of recently discovered genetic risk loci for schizophrenia and polygenic risk profile in five RDoC subdomains , 2017, Translational Psychiatry.
[107] P. Sullivan,et al. Transcriptional targets of the schizophrenia risk gene MIR137 , 2014, Translational Psychiatry.
[108] Nora C. Vetter,et al. Polygenic Risk of Psychosis and Ventral Striatal Activation During Reward Processing in Healthy Adolescents. , 2016, JAMA psychiatry.
[109] M. Phillips,et al. The functional neuroanatomy of bipolar disorder: a consensus model. , 2012, Bipolar disorders.