Protective effect of resin adsorption on septic plasma-induced tubular injury

[1]  S. Hollenberg,et al.  Cardiac dysfunction in severe sepsis and septic shock , 2009, Current opinion in critical care.

[2]  C. Coopersmith,et al.  CD4+ lymphocytes control gut epithelial apoptosis and mediate survival in sepsis , 2009, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[3]  G. Camussi,et al.  Macrophage stimulating protein may promote tubular regeneration after acute injury. , 2008, Journal of the American Society of Nephrology : JASN.

[4]  R. Bellomo,et al.  The future of extracorporeal support , 2008, Critical care medicine.

[5]  V. Ranieri,et al.  Circulating plasma factors induce tubular and glomerular alterations in septic burns patients , 2008, Critical care.

[6]  R. Bellomo,et al.  The histopathology of septic acute kidney injury: a systematic review , 2008, Critical care.

[7]  Luigi Biancone,et al.  Endothelial progenitor cell derived microvesicles activate an angiogenic program in endothelial cells by a horizontal transfer of mRNA. , 2007, Blood.

[8]  Ying Wang,et al.  The role of ICAM-1 in endotoxin-induced acute renal failure. , 2007, American journal of physiology. Renal physiology.

[9]  C. Ronco,et al.  Acute kidney injury in the intensive care unit: current trends in incidence and outcome , 2007, Critical care.

[10]  M. Bucher,et al.  Cytokine-mediated regulation of urea transporters during experimental endotoxemia. , 2007, American journal of physiology. Renal physiology.

[11]  F. Antunes,et al.  Prognostic utility of RIFLE for acute renal failure in patients with sepsis , 2007, Critical care.

[12]  A. Kurtz,et al.  Regulation of renal sodium transporters during severe inflammation. , 2007, Journal of the American Society of Nephrology : JASN.

[13]  R. Bellomo,et al.  Defining and classifying acute renal failure: from advocacy to consensus and validation of the RIFLE criteria , 2007, Intensive Care Medicine.

[14]  M. Bucher,et al.  Regulation of renal glucose transporters during severe inflammation. , 2007, American journal of physiology. Renal physiology.

[15]  R. Hotchkiss,et al.  Apoptosis and caspases regulate death and inflammation in sepsis , 2006, Nature Reviews Immunology.

[16]  W. Yew,et al.  Correlation of microalbuminuria and outcome in patients with extensive burns. , 2006, British journal of anaesthesia.

[17]  J. Kellum,et al.  RIFLE criteria provide robust assessment of kidney dysfunction and correlate with hospital mortality. , 2006, Critical care medicine.

[18]  R. Heyderman,et al.  Platelet and soluble CD40L in meningococcal sepsis , 2006, Intensive Care Medicine.

[19]  P. Jennings,et al.  Mechanisms of Neutrophil Transmigration Across Renal Proximal Tubular HK-2 Cells , 2006, Cellular Physiology and Biochemistry.

[20]  L. Gesualdo,et al.  CD40L proinflammatory and profibrotic effects on proximal tubular epithelial cells: role of NF-kappaB and lyn. , 2006, Journal of the American Society of Nephrology : JASN.

[21]  C. Probst,et al.  LEUKOCYTE-ENDOTHELIAL INTERACTIONS VIA ICAM-1 ARE DETRIMENTAL IN POLYMICROBIAL SEPSIS , 2006, Shock.

[22]  L. Gesualdo,et al.  CD40 ligand increases complement C3 secretion by proximal tubular epithelial cells. , 2005, Journal of the American Society of Nephrology : JASN.

[23]  S. Mizuno,et al.  Prevention of neutrophil extravasation by hepatocyte growth factor leads to attenuations of tubular apoptosis and renal dysfunction in mouse ischemic kidneys. , 2005, The American journal of pathology.

[24]  M. Fink,et al.  Epithelial barrier dysfunction: a unifying theme to explain the pathogenesis of multiple organ dysfunction at the cellular level. , 2005, Critical care clinics.

[25]  R. Quigg,et al.  Acute Renal Failure in Endotoxemia is Dependent on Caspase Activation , 2003 .

[26]  F. Haddy,et al.  Acute renal failure and sepsis. , 2004, The New England journal of medicine.

[27]  R. Bellomo,et al.  Acute renal failure – definition, outcome measures, animal models, fluid therapy and information technology needs: the Second International Consensus Conference of the Acute Dialysis Quality Initiative (ADQI) Group , 2004, Critical care.

[28]  Emil,et al.  Plasma cytokine levels predict mortality in patients with acute renal failure. , 2004, Kidney international.

[29]  John A Kellum,et al.  Hemoadsorption removes tumor necrosis factor, interleukin-6, and interleukin-10, reduces nuclear factor-&kgr;B DNA binding, and improves short-term survival in lethal endotoxemia* , 2004, Critical care medicine.

[30]  R. Quigg,et al.  Role of Toll-Like Receptor 4 in Endotoxin-Induced Acute Renal Failure 1 , 2004, The Journal of Immunology.

[31]  R. Bellomo,et al.  The pathogenesis of septic acute renal failure , 2003, Current opinion in critical care.

[32]  S. Ridley,et al.  Drugs in anaesthesia , 2003, Anaesthesia.

[33]  R. Bellomo,et al.  Interpreting the mechanisms of continuous renal replacement therapy in sepsis: the peak concentration hypothesis. , 2003, Artificial organs.

[34]  J. Bonventre,et al.  Recent advances in the pathophysiology of ischemic acute renal failure. , 2003, Journal of the American Society of Nephrology : JASN.

[35]  Jeffrey A. Gold,et al.  CD40 Contributes to Lethality in Acute Sepsis: In Vivo Role for CD40 in Innate Immunity , 2003, Infection and Immunity.

[36]  T. Szakmany,et al.  Microalbuminuria in severe sepsis: timing is difficult, "normal" levels are uncertain , 2003, Intensive Care Medicine.

[37]  Anirban Datta,et al.  Epithelial polarity and tubulogenesis in vitro. , 2003, Trends in cell biology.

[38]  M. Fink,et al.  Proinflammatory Cytokines Cause No·-Dependent and -Independent Changes in Expression and Localization of Tight Junction Proteins in Intestinal Epithelial Cells , 2003, Shock.

[39]  T. Szakmany,et al.  Microalbuminuria does not reflect increased systemic capillary permeability in septic shock , 2003, Intensive Care Medicine.

[40]  D. Cha,et al.  Inflammatory Cytokines and Lipopolysaccharide Induce Fas-Mediated Apoptosis in Renal Tubular Cells , 2002, Nephron.

[41]  R. Quigg,et al.  Acute Renal Failure in Endotoxemia Is Caused by TNF Acting Directly on TNF Receptor-1 in Kidney1 , 2002, The Journal of Immunology.

[42]  Rinaldo Bellomo,et al.  A pilot study of coupled plasma filtration with adsorption in septic shock* , 2002, Critical care medicine.

[43]  J. Picot,et al.  Coexpressed nitric oxide synthase and apical beta(1) integrins influence tubule cell adhesion after cytokine-induced injury. , 2001, Journal of the American Society of Nephrology : JASN.

[44]  G. Camussi,et al.  Hemofiltration reduces the serum priming activity on neutrophil chemiluminescence in septic patients. , 2001, Kidney international.

[45]  H Birn,et al.  Megalin and cubilin: synergistic endocytic receptors in renal proximal tubule. , 2001, American journal of physiology. Renal physiology.

[46]  M. Daha,et al.  Immunological Function of Tubular Epithelial Cells: The Functional Implications of CD40 Expression , 2000, Nephron Experimental Nephrology.

[47]  M. Braga,et al.  Coupled plasma filtration‐adsorption in a rabbit model of endotoxic shock , 2000, Critical care medicine.

[48]  H. Vorum,et al.  Megalin knockout mice as an animal model of low molecular weight proteinuria. , 1999, The American journal of pathology.

[49]  A. Kribben,et al.  Pathophysiology of acute renal failure. , 1999, JN. Journal of Nephrology (Milano. 1992).

[50]  Glynne Pa,et al.  Inflammatory cytokines induce apoptotic and necrotic cell shedding from human proximal tubular epithelial cell monolayers , 1999 .

[51]  J. De Sutter,et al.  Cytokine removal during continuous hemofiltration in septic patients. , 1999, Journal of the American Society of Nephrology : JASN.

[52]  G. Camussi,et al.  HIV-1 kills renal tubular epithelial cells in vitro by triggering an apoptotic pathway involving caspase activation and Fas upregulation. , 1998, The Journal of clinical investigation.

[53]  John P. Johnson,et al.  Diffusive vs. convective therapy: effects on mediators of inflammation in patient with severe systemic inflammatory response syndrome. , 1998, Critical care medicine.

[54]  C. Ronco,et al.  Removal of cytokines and activated complement components in an experimental model of continuous plasma filtration coupled with sorbent adsorption. , 1998, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

[55]  J. Peters,et al.  Hemofiltration increases IL-6 clearance in early systemic inflammatory response syndrome but does not alter IL-6 and TNFα plasma concentrations , 1997, Intensive Care Medicine.

[56]  G. Camussi,et al.  Plasmin promotes an endothelium-dependent adhesion of neutrophils. Involvement of platelet activating factor and P-selectin. , 1996, Circulation.

[57]  G. Camussi,et al.  Porins and lipopolysaccharide stimulate platelet activating factor synthesis by human mesangial cells. , 1992, Kidney international.

[58]  J. E. Carceller.  American College of Chest Physicians/Society of Critical Care Medicine Consensus Conference: Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis , 1992, Critical care medicine.

[59]  W. Knaus,et al.  Definitions for sepsis and organ failure and guidelines for the use of innovative therapies in sepsis. The ACCP/SCCM Consensus Conference Committee. American College of Chest Physicians/Society of Critical Care Medicine. 1992. , 2009, Chest.

[60]  C. Ronco,et al.  which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Protective effect of resin adsorption on septic plasma-induced tubular injury , 2009 .

[61]  H. Ward,et al.  Tight junction biology and kidney dysfunction. , 2006, American journal of physiology. Renal physiology.

[62]  V. Pettilä,et al.  [Sepsis and kidney]. , 2006, Duodecim; laaketieteellinen aikakauskirja.

[63]  M. Fink,et al.  Increased iNOS activity is essential for hepatic epithelial tight junction dysfunction in endotoxemic mice. , 2004, American journal of physiology. Gastrointestinal and liver physiology.

[64]  M. Fink,et al.  Increased iNOS activity is essential for pulmonary epithelial tight junction dysfunction in endotoxemic mice. , 2004, American journal of physiology. Lung cellular and molecular physiology.

[65]  Evans,et al.  Inflammatory cytokines induce apoptotic and necrotic cell shedding from human proximal tubular epithelial cell monolayers , 1999, Kidney international.