Role of ITGAE in the development of autoimmune diabetes in non-obese diabetic mice.
暂无分享,去创建一个
P. Weinreb | A. Rajab | J. Buss | Q. Mi | Elizabeth S. Barrie | C. Adin | G. Hadley | M. Lodder | Jill Buss
[1] P. Meda. Protein-Mediated Interactions of Pancreatic Islet Cells , 2013, Scientifica.
[2] A. Cooke,et al. CD4 T cells and their antigens in the pathogenesis of autoimmune diabetes. , 2011, Current opinion in immunology.
[3] K. Ishii,et al. A New Subset of CD103+CD8α+ Dendritic Cells in the Small Intestine Expresses TLR3, TLR7, and TLR9 and Induces Th1 Response and CTL Activity , 2011, The Journal of Immunology.
[4] S. Moffatt-Bruce,et al. An Anti‐CD103 Immunotoxin Promotes Long‐Term Survival of Pancreatic Islet Allografts , 2009, American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons.
[5] R. Jefferis,et al. Recombinant antibody therapeutics: the impact of glycosylation on mechanisms of action. , 2009, Trends in pharmacological sciences.
[6] M. V. von Herrath,et al. CD103 is dispensable for anti-viral immunity and autoimmunity in a mouse model of virally-induced autoimmune diabetes. , 2009, Journal of autoimmunity.
[7] J. Bluestone,et al. Immunoregulatory Pathways Controlling Progression of Autoimmunity in NOD Mice , 2008, Annals of the New York Academy of Sciences.
[8] D. Männel,et al. Cutting Edge: Expression of TNFR2 Defines a Maximally Suppressive Subset of Mouse CD4+CD25+FoxP3+ T Regulatory Cells: Applicability to Tumor-Infiltrating T Regulatory Cells1 , 2008, The Journal of Immunology.
[9] Simon C. Potter,et al. Localization of type 1 diabetes susceptibility to the MHC class I genes HLA-B and HLA-A , 2007, Nature.
[10] Irene Jarchum,et al. In Vivo Cytotoxicity of Insulin-Specific CD8+ T-Cells in HLA-A*0201 Transgenic NOD Mice , 2007, Diabetes.
[11] Angelo J. Canty,et al. The Idd4 Locus Displays Sex-Specific Epistatic Effects on Type 1 Diabetes Susceptibility in Nonobese Diabetic Mice , 2006, Diabetes.
[12] David C Montefiori,et al. Evaluation of CD8+ T-cell and antibody responses following transient increased viraemia in rhesus macaques infected with live, attenuated simian immunodeficiency virus. , 2005, The Journal of general virology.
[13] F. Powrie,et al. Essential role for CD103 in the T cell–mediated regulation of experimental colitis , 2005, The Journal of experimental medicine.
[14] C. Drachenberg,et al. Critical Role for CD103+CD8+ Effectors in Promoting Tubular Injury following Allogeneic Renal Transplantation , 2005, The Journal of Immunology.
[15] C. Drachenberg,et al. TGF-β–dependent CD103 expression by CD8+ T cells promotes selective destruction of the host intestinal epithelium during graft-versus-host disease , 2005, The Journal of experimental medicine.
[16] K. Shitara,et al. The Absence of Fucose but Not the Presence of Galactose or Bisecting N-Acetylglucosamine of Human IgG1 Complex-type Oligosaccharides Shows the Critical Role of Enhancing Antibody-dependent Cellular Cytotoxicity* , 2003, The Journal of Biological Chemistry.
[17] D. Farber,et al. CD103 Expression Is Required for Destruction of Pancreatic Islet Allografts by CD8+ T Cells , 2002, The Journal of experimental medicine.
[18] Alf Hamann,et al. Expression of the integrin αEβ7 identifies unique subsets of CD25+ as well as CD25− regulatory T cells , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[19] D. Beier,et al. Mucosal T Lymphocyte Numbers Are Selectively Reduced in Integrin αE (CD103)-Deficient Mice , 1999, The Journal of Immunology.
[20] D. Beier,et al. Mucosal T lymphocyte numbers are selectively reduced in integrin alpha E (CD103)-deficient mice. , 1999, Journal of immunology.
[21] D. Roopenian,et al. Major histocompatibility complex class I-restricted T cells are required for all but the end stages of diabetes development in nonobese diabetic mice and use a prevalent T cell receptor alpha chain gene rearrangement. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[22] Bhagirath Singh,et al. The nonobese diabetic mouse as a model of autoimmune diabetes: immune dysregulation gets the NOD. , 1997, Immunity.
[23] S. Bartlett,et al. The epithelial cell-specific integrin, CD103 (alpha E integrin), defines a novel subset of alloreactive CD8+ CTL. , 1997, Journal of immunology.
[24] E. Leiter,et al. Initiation of autoimmune diabetes in NOD/Lt mice is MHC class I-dependent. , 1997, Journal of immunology.
[25] C. Benoist,et al. The role of CD8+ T cells in the initiation of insulin‐dependent diabetes mellitus , 1996, European journal of immunology.
[26] R Weissleder,et al. MR of carcinoma-specific monoclonal antibody conjugated to monocrystalline iron oxide nanoparticles: the potential for noninvasive diagnosis. , 1996, AJNR. American journal of neuroradiology.
[27] C. Fathman,et al. Islet-Infiltrating Lymphocytes from Prediabetic NOD Mice Rapidly Transfer Diabetes to NOD-scid/scid mice , 1995, Diabetes.
[28] P. Karecla,et al. Recognition of E‐cadherin on epithelial cells by the mucosal T cell integrin αM290β7 (αEβ7) , 1995 .
[29] D. Rimm,et al. Adhesion between epithelial cells and T lymphocytes mediated by E-cadherin and the αEβ7 integrin , 1994, Nature.
[30] E. Neuwelt,et al. Effect of blood-brain barrier disruption on intact and fragmented monoclonal antibody localization in intracerebral lung carcinoma xenografts. , 1994, Journal of nuclear medicine : official publication, Society of Nuclear Medicine.
[31] L. Orci,et al. Expression of neural cell adhesion molecule (N-CAM) in rat islets and its role in islet cell type segregation. , 1994, Journal of cell science.
[32] E. Leiter,et al. Major Histocompatibility Complex Class I-Deficient NOD-B2mnull Mice are Diabetes and Insulitis Resistant , 1994, Diabetes.
[33] R. Jaenisch,et al. β2-Microglobulin–Deficient NOD Mice Do Not Develop Insulitis or Diabetes , 1994, Diabetes.
[34] L. Lefrançois,et al. Developmental expression of the αIELβ7 integrin on T cell receptor γδ and T cell receptor αβ T cells , 1994 .
[35] C. Benoist,et al. Major histocompatibility complex class I molecules are required for the development of insulitis in non‐obese diabetic mice , 1993, European journal of immunology.
[36] M. Matsumoto,et al. Transfer of autoimmune diabetes from diabetic NOD mice to NOD athymic nude mice: the roles of T cell subsets in the pathogenesis. , 1993, Cellular immunology.
[37] E. Leiter,et al. Adoptive Transfer of Diabetes Into Immunodeficient NOD-scid/scid Mice: Relative Contributions of CD4+ and CD8+ T-Cells From Diabetic Versus Prediabetic NOD.NON-Thy-1a Donors , 1993, Diabetes.
[38] T. Morioka,et al. Evaluation of the central nervous function in resuscitated comatose patients by multilevel evoked potentials. , 1992, Resuscitation.
[39] P. Kilshaw,et al. A unique surface antigen on intraepithelial lymphocytes in the mouse. , 1988, Immunology letters.
[40] C. Fathman,et al. Immunotherapy of the nonobese diabetic mouse: treatment with an antibody to T-helper lymphocytes. , 1988, Science.
[41] L. Wicker,et al. Both the Lyt-2+ and L3T4+ T cell subsets are required for the transfer of diabetes in nonobese diabetic mice. , 1988, Journal of immunology.
[42] C. Boitard,et al. Syngeneic transfer of autoimmune diabetes from diabetic NOD mice to healthy neonates. Requirement for both L3T4+ and Lyt-2+ T cells , 1987, The Journal of experimental medicine.
[43] Y. Tochino,et al. Breeding of a non-obese, diabetic strain of mice. , 1980, Jikken dobutsu. Experimental animals.
[44] P. Santamaria,et al. CD8+ T cells in type 1 diabetes. , 2008, Advances in immunology.
[45] J. Binley,et al. Evaluation of CD 8 + T-cell and antibody responses following transient increased viraemia in rhesus macaques infected with live , attenuated simian immunodeficiency virus , 2005 .
[46] D. Farber,et al. CD 103 Expression Is Required for Destruction of Pancreatic Islet Allografts by CD 8 T Cells , 2002 .