Survival of Mycobacterium tuberculosis in Host Macrophages Involves Resistance to Apoptosis Dependent upon Induction of Antiapoptotic Bcl-2 Family Member Mcl-11
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[1] T. Mustafa,et al. Increased Bcl‐2 and Reduced Bax Expression in Infected Macrophages in Slowly Progressive Primary Murine Mycobacterium tuberculosis Infection , 2002, Scandinavian journal of immunology.
[2] Limin Zheng,et al. Mycobacterium tuberculosis Promotes Apoptosis in Human Neutrophils by Activating Caspase-3 and Altering Expression of Bax/Bcl-xL Via an Oxygen-Dependent Pathway1 , 2002, The Journal of Immunology.
[3] D. Kumararatne,et al. ATP-mediated killing of intracellular mycobacteria by macrophages is a P2X(7)-dependent process inducing bacterial death by phagosome-lysosome fusion. , 2002, Journal of immunology.
[4] S. Catz,et al. Transcriptional regulation of bcl-2 by nuclear factor κB and its significance in prostate cancer , 2001, Oncogene.
[5] N. Reiner,et al. 1α,25-Dihydroxyvitamin D3-induced Monocyte Antimycobacterial Activity Is Regulated by Phosphatidylinositol 3-Kinase and Mediated by the NADPH-dependent Phagocyte Oxidase* , 2001, The Journal of Biological Chemistry.
[6] D. Kumararatne,et al. ATP-Mediated Killing of Intracellular Mycobacteria by Macrophages Is a P2X7-Dependent Process Inducing Bacterial Death by Phagosome-Lysosome Fusion1 , 2001, The Journal of Immunology.
[7] D. Kusner,et al. ATP Stimulates Human Macrophages to Kill Intracellular Virulent Mycobacterium tuberculosis Via Calcium-Dependent Phagosome-Lysosome Fusion1 , 2001, The Journal of Immunology.
[8] S. Lightman,et al. ATP-Mediated Killing of Mycobacterium bovis Bacille Calmette-Guérin Within Human Macrophages Is Calcium Dependent and Associated with the Acidification of Mycobacteria-Containing Phagosomes1 , 2001, The Journal of Immunology.
[9] A. Orlofsky,et al. Requirement of A1-a for Bacillus Calmette-Guérin-Mediated Protection of Macrophages Against Nitric Oxide-Induced Apoptosis1 , 2001, The Journal of Immunology.
[10] S. Cory,et al. Life-or-death decisions by the Bcl-2 protein family. , 2001, Trends in biochemical sciences.
[11] C. Thompson,et al. The role of the Bcl-2 family in the regulation of outer mitochondrial membrane permeability , 2000, Cell Death and Differentiation.
[12] S. Leuenroth,et al. The loss of Mcl‐1 expression in human polymorphonuclear leukocytes promotes apoptosis , 2000, Journal of leukocyte biology.
[13] C. Montesano,et al. Mycobacterium tuberculosis-induced apoptosis in monocytes/macrophages: early membrane modifications and intracellular mycobacterial viability. , 2000, The Journal of infectious diseases.
[14] Y. Tsujimoto,et al. BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[15] Hardy Kornfeld,et al. Virulent Mycobacterium tuberculosis Strains Evade Apoptosis of Infected Alveolar Macrophages1 , 2000, The Journal of Immunology.
[16] R. Appelberg,et al. Macrophage Control of Mycobacterial Growth Induced by Picolinic Acid Is Dependent on Host Cell Apoptosis1 , 2000, The Journal of Immunology.
[17] R. Stokes,et al. The receptor-mediated uptake, survival, replication, and drug sensitivity of Mycobacterium tuberculosis within the macrophage-like cell line THP-1: a comparison with human monocyte-derived macrophages. , 1999, Cellular immunology.
[18] A. Zychlinsky,et al. The induction of apoptosis by bacterial pathogens. , 1999, Annual review of microbiology.
[19] V. Colizzi,et al. The role of macrophage cell death in tuberculosis , 1999, Cell Death and Differentiation.
[20] T. Ganz,et al. An antimicrobial activity of cytolytic T cells mediated by granulysin. , 1998, Science.
[21] J. Keane,et al. Pathogenic Mycobacterium tuberculosis evades apoptosis of host macrophages by release of TNF-R2, resulting in inactivation of TNF-alpha. , 1998, Journal of immunology.
[22] H. Macdonald,et al. Fas ligand-induced apoptosis of infected human macrophages reduces the viability of intracellular Mycobacterium tuberculosis. , 1998, Journal of immunology.
[23] J. Ernst,et al. Macrophage Receptors for Mycobacterium tuberculosis , 1998, Infection and Immunity.
[24] M. Arock,et al. Human eosinophils express bcl-2 family proteins: modulation of Mcl-1 expression by IFN-gamma. , 1998, American journal of respiratory cell and molecular biology.
[25] M. Fenton. Macrophages and tuberculosis , 1998, Current opinion in hematology.
[26] W. Rom,et al. Copyright © 1997, American Society for Microbiology Effects of Mycobacteria on Regulation of Apoptosis in , 1997 .
[27] A. Sette,et al. Differential effects of cytolytic T cell subsets on intracellular infection. , 1997, Science.
[28] R. Arbeit,et al. Programmed cell death of Mycobacterium avium serovar 4-infected human macrophages prevents the mycobacteria from spreading and induces mycobacterial growth inhibition by freshly added, uninfected macrophages. , 1997, Journal of immunology.
[29] J. Keane,et al. Infection by Mycobacterium tuberculosis promotes human alveolar macrophage apoptosis , 1997, Infection and immunity.
[30] M. Piacentini,et al. APOPTOSIS OF HUMAN MONOCYTES/MACROPHAGES IN MYCOBACTERIUM TUBERCULOSIS INFECTION , 1997, The Journal of pathology.
[31] M. Ernst,et al. Effect of in vitro infection of human monocytes with low numbers of Mycobacterium tuberculosis bacteria on monocyte apoptosis , 1996, Infection and immunity.
[32] E. Smeland,et al. Expression of the Bcl-2 homologue Mcl-1 correlates with survival of peripheral blood B lymphocytes. , 1996, Cancer research.
[33] N. Reiner,et al. Attenuation of gamma interferon-induced tyrosine phosphorylation in mononuclear phagocytes infected with Leishmania donovani: selective inhibition of signaling through Janus kinases and Stat1 , 1995, Infection and immunity.
[34] G. Kaplan,et al. Apoptosis, but not necrosis, of infected monocytes is coupled with killing of intracellular bacillus Calmette-Guerin , 1994, The Journal of experimental medicine.
[35] N. Reiner,et al. CD14-dependent activation of protein kinase C and mitogen-activated protein kinases (p42 and p44) in human monocytes treated with bacterial lipopolysaccharide. , 1994, Journal of immunology.
[36] M. Raviglione,et al. Global tuberculosis incidence and mortality during 1990-2000. , 1994, Bulletin of the World Health Organization.
[37] R. Craig,et al. MCL1, a gene expressed in programmed myeloid cell differentiation, has sequence similarity to BCL2. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[38] A. J. Crowle,et al. Inhibition by 1,25(OH)2-vitamin D3 of the multiplication of virulent tubercle bacilli in cultured human macrophages , 1987, Infection and immunity.
[39] J. S. Beck,et al. Cell death in granulomata: the role of apoptosis. , 1987, Journal of clinical pathology.
[40] U. K. Laemmli,et al. Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4 , 1970, Nature.