Downregulated miR-45 Inhibits the G1-S Phase Transition by Targeting Bmi-1 in Breast Cancer
暂无分享,去创建一个
Shu Wu | Lan-Zeng Wang | Xi Wang | Jun-ling Liu | Liang Yu | Xiangnan Liu | Hong-mei Wu | Fangyong Lei
[1] J. Cuzick,et al. Update on breast cancer risk prediction and prevention , 2015, Current opinion in obstetrics & gynecology.
[2] Changjiang Qin,et al. XRCC2 Promotes Colorectal Cancer Cell Growth, Regulates Cell Cycle Progression, and Apoptosis , 2014, Medicine.
[3] A. Levine,et al. MicroRNAs, miR-154, miR-299-5p, miR-376a, miR-376c, miR-377, miR-381, miR-487b, miR-485-3p, miR-495 and miR-654-3p, mapped to the 14q32.31 locus, regulate proliferation, apoptosis, migration and invasion in metastatic prostate cancer cells , 2014, Oncogene.
[4] Xueyun Zhong,et al. MicroRNA‐221 targeting PI3‐K/Akt signaling axis induces cell proliferation and BCNU resistance in human glioblastoma , 2014, Neuropathology : official journal of the Japanese Society of Neuropathology.
[5] K. Zen,et al. MicroRNA-495 induces breast cancer cell migration by targeting JAM-A , 2014, Protein & Cell.
[6] Wei R. Chen,et al. Mir-208 promotes cell proliferation by repressing SOX6 expression in human esophageal squamous cell carcinoma , 2014, Journal of Translational Medicine.
[7] Liqiang Song,et al. miR‐495 Enhances the Sensitivity of Non‐Small Cell Lung Cancer Cells to Platinum by Modulation of Copper‐Transporting P‐type Adenosine Triphosphatase A (ATP7A) , 2014, Journal of cellular biochemistry.
[8] R. Weinberg,et al. Tackling the cancer stem cells — what challenges do they pose? , 2014, Nature Reviews Drug Discovery.
[9] Xiang Lu,et al. Bmi-1 plays a critical role in protection from renal tubulointerstitial injury by maintaining redox balance , 2014, Aging cell.
[10] Xin Chen,et al. miRNA-128 suppresses prostate cancer by inhibiting BMI-1 to inhibit tumor-initiating cells. , 2014, Cancer research.
[11] Pei Zhang,et al. Down Regulation of miR200c Promotes Radiation‐Induced Thymic Lymphoma by Targeting BMI1 , 2014, Journal of cellular biochemistry.
[12] Hugo Naya,et al. Activation of the PI3K/AKT pathway by microRNA-22 results in CLL B-cell proliferation , 2014, Leukemia.
[13] W. Di,et al. MicroRNA-7 Inhibits Tumor Metastasis and Reverses Epithelial-Mesenchymal Transition through AKT/ERK1/2 Inactivation by Targeting EGFR in Epithelial Ovarian Cancer , 2014, PloS one.
[14] Kang Yang,et al. MiR-21 inhibits c-Ski signaling to promote the proliferation of rat vascular smooth muscle cells. , 2014, Cellular signalling.
[15] H. El-Hadaad,et al. Immunohistochemical expression and prognostic relevance of Bmi-1, a stem cell factor, in epithelial ovarian cancer. , 2014, Annals of diagnostic pathology.
[16] Li-ping Guo,et al. miR-203 inhibits the proliferation and self-renewal of esophageal cancer stem-like cells by suppressing stem renewal factor Bmi-1. , 2014, Stem cells and development.
[17] Libing Song,et al. Overexpression of AKIP1 promotes angiogenesis and lymphangiogenesis in human esophageal squamous cell carcinoma , 2014, Oncogene.
[18] A. Margulis,et al. Changes of PI3K/AKT/BCL2 signaling proteins in congenital Giant Nevi: melanocytes contribute to their increased survival and integrity , 2013, Journal of receptor and signal transduction research.
[19] Shanling Liu,et al. miR-126 Suppresses the proliferation of cervical cancer cells and alters cell sensitivity to the chemotherapeutic drug bleomycin. , 2013, Asian Pacific journal of cancer prevention : APJCP.
[20] H. Qin,et al. MiR-34a targets GAS1 to promote cell proliferation and inhibit apoptosis in papillary thyroid carcinoma via PI3K/Akt/Bad pathway. , 2013, Biochemical and biophysical research communications.
[21] Ana Kozomara,et al. miRBase: annotating high confidence microRNAs using deep sequencing data , 2013, Nucleic Acids Res..
[22] Liang Zhou,et al. Overexpression of Bmi-1 contributes to the invasion and metastasis of hepatocellular carcinoma by increasing the expression of matrix metalloproteinase (MMP)‑2, MMP-9 and vascular endothelial growth factor via the PTEN/PI3K/Akt pathway. , 2013, International journal of oncology.
[23] H. Shigeishi,et al. AKT primes snail‐induced EMT concomitantly with the collective migration of squamous cell carcinoma cells , 2013, Journal of cellular biochemistry.
[24] Zengtong Zhou,et al. Bmi1 expression in oral lichen planus and the risk of progression to oral squamous cell carcinoma. , 2013, Annals of diagnostic pathology.
[25] A. Lam,et al. BMI-1 activation is crucial in hTERT-induced epithelial-mesenchymal transition of oral epithelial cells. , 2013, Experimental and molecular pathology.
[26] M. S. Parvathi,et al. Regulation of BMI1 Polycomb gene expression in histological grades of invasive ductal breast carcinomas and its correlation with hormone receptor status , 2013, Tumor Biology.
[27] R. Blelloch,et al. miR-294/miR-302 promotes proliferation, suppresses G1-S restriction point, and inhibits ESC differentiation through separable mechanisms. , 2013, Cell reports.
[28] M. Zeng,et al. Sp1 and c‐Myc regulate transcription of BMI1 in nasopharyngeal carcinoma , 2013, The FEBS journal.
[29] Shu-Jen Chen,et al. MicroRNA-495 inhibits proliferation of glioblastoma multiforme cells by downregulating cyclin-dependent kinase 6 , 2013, World Journal of Surgical Oncology.
[30] R. Balleine,et al. Ki67 and proliferation in breast cancer , 2013, Journal of Clinical Pathology.
[31] Xun Zhu,et al. Bmi-1 Promotes Glioma Angiogenesis by Activating NF-κB Signaling , 2013, PloS one.
[32] T. Godfrey,et al. Clinicopathologic characteristics of high expression of Bmi-1 in esophageal adenocarcinoma and squamous cell carcinoma , 2012, BMC Gastroenterology.
[33] Dong-Woo Lee,et al. miR-495 and miR-551a inhibit the migration and invasion of human gastric cancer cells by directly interacting with PRL-3. , 2012, Cancer letters.
[34] P. Nelson,et al. VEGF/neuropilin-2 regulation of Bmi-1 and consequent repression of IGF-IR define a novel mechanism of aggressive prostate cancer. , 2012, Cancer discovery.
[35] Byeong-Moo Kim,et al. Non-canonical microRNAs miR-320 and miR-702 promote proliferation in Dgcr8-deficient embryonic stem cells. , 2012, Biochemical and biophysical research communications.
[36] L. Cai,et al. Expression patterns of USP22 and potential targets BMI-1, PTEN, p-AKT in non-small-cell lung cancer. , 2012, Lung cancer.
[37] Ali Montazeri,et al. Literacy and breast cancer prevention: a population-based study from Iran. , 2012, Asian Pacific journal of cancer prevention : APJCP.
[38] H. G. van der Poel,et al. Akt-mediated phosphorylation of Bmi1 modulates its oncogenic potential, E3 ligase activity, and DNA damage repair activity in mouse prostate cancer. , 2012, The Journal of clinical investigation.
[39] L. Min,et al. Clinicopathological and prognostic significance of Bmi‐1 expression in human cervical cancer , 2011, Acta obstetricia et gynecologica Scandinavica.
[40] King-Jen Chang,et al. miR-495 is upregulated by E12/E47 in breast cancer stem cells, and promotes oncogenesis and hypoxia resistance via downregulation of E-cadherin and REDD1 , 2011, Oncogene.
[41] Libing Song,et al. Bmi-1 promotes invasion and metastasis, and its elevated expression is correlated with an advanced stage of breast cancer , 2011, Molecular Cancer.
[42] P. Fisher,et al. Expression patterns of astrocyte elevated gene-1 (AEG-1) during development of the mouse embryo. , 2010, Gene expression patterns : GEP.
[43] Libing Song,et al. Unregulated miR-96 Induces Cell Proliferation in Human Breast Cancer by Downregulating Transcriptional Factor FOXO3a , 2010, PloS one.
[44] Libing Song,et al. Sam68 up‐regulation correlates with, and its down‐regulation inhibits, proliferation and tumourigenicity of breast cancer cells , 2010, The Journal of pathology.
[45] M. Cooperberg,et al. Electronic patient self-assessment and management (SAM): a novel framework for cancer survivorship , 2010, BMC Medical Informatics Decis. Mak..
[46] Goberdhan P Dimri,et al. BMI1 and Mel-18 oppositely regulate carcinogenesis and progression of gastric cancer , 2010, Molecular Cancer.
[47] P. Fisher,et al. Astrocyte elevated gene-1 (AEG-1) functions as an oncogene and regulates angiogenesis , 2009, Proceedings of the National Academy of Sciences.
[48] Wenlin Huang,et al. The polycomb group protein Bmi-1 represses the tumor suppressor PTEN and induces epithelial-mesenchymal transition in human nasopharyngeal epithelial cells. , 2009, The Journal of clinical investigation.
[49] M. Huang,et al. The Growth Factor Independence-1 (Gfi1) Is Overexpressed in Chronic Myelogenous Leukemia , 2009, Acta Haematologica.
[50] U. Rapp,et al. Polycomb Group Protein Bmi1 Is Required for Growth of RAF Driven Non-Small-Cell Lung Cancer , 2009, PloS one.
[51] A. Iwama,et al. The polycomb gene product BMI1 contributes to the maintenance of tumor-initiating side population cells in hepatocellular carcinoma. , 2008, Cancer research.
[52] A. Paetau,et al. Stem cell protein BMI‐1 is an independent marker for poor prognosis in oligodendroglial tumours , 2008, Neuropathology and applied neurobiology.
[53] N. Raab-Traub,et al. EBV Latent Membrane Protein 1 Activates Akt, NFκB, and Stat3 in B Cell Lymphomas , 2007, PLoS pathogens.
[54] W. Tilley,et al. Role of oncoprotein Growth Factor Independent-1 (GFI1) in repression of 25-hydroxyvitamin D 1alpha-hydroxylase (CYP27B1): A comparative analysis in human prostate cancer and kidney cells , 2007, The Journal of Steroid Biochemistry and Molecular Biology.
[55] Yun-Fei Xia,et al. Bmi-1 is a novel molecular marker of nasopharyngeal carcinoma progression and immortalizes primary human nasopharyngeal epithelial cells. , 2006, Cancer research.
[56] T. Sauka-Spengler,et al. Expression of the polycomb group gene bmi-1 in the early chick embryo. , 2004, Gene expression patterns : GEP.
[57] C. Gilks,et al. Growth Factor Independence-1 Is Expressed in Primary Human Neuroendocrine Lung Carcinomas and Mediates the Differentiation of Murine Pulmonary Neuroendocrine Cells , 2004, Cancer Research.
[58] K Kornfeld,et al. Multiple docking sites on substrate proteins form a modular system that mediates recognition by ERK MAP kinase. , 1999, Genes & development.
[59] R. DePinho,et al. The oncogene and Polycomb-group gene bmi-1 regulates cell proliferation and senescence through the ink4a locus , 1999, Nature.
[60] Chao Qin,et al. miR-154 inhibits prostate cancer cell proliferation by targeting CCND2. , 2014, Urologic oncology.
[61] A. Berns,et al. Bmi-1 collaborates with c-Myc in tumorigenesis by inhibiting c-Myc-induced apoptosis via INK4a/ARF. , 1999, Genes & development.