Graying: gerontobiology of the hair follicle pigmentary unit
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[1] B. Bernard,et al. Melanocyte subpopulation turnover during the human hair cycle: an immunohistochemical study. , 2000, Pigment cell research.
[2] N. Jablonski,et al. The evolution of human skin coloration. , 2000, Journal of human evolution.
[3] J. Rees,et al. The melanocortin 1 receptor (MC1R): more than just red hair. , 2000, Pigment cell research.
[4] E. Glazenburg. Is the reversal of lichenification in patients with atopic dermatitis always a long-term process? , 2000 .
[5] D. Tobin,et al. The fate of hair follicle melanocytes during the hair growth cycle. , 1999, The journal of investigative dermatology. Symposium proceedings.
[6] Pier Paolo Pandolfi,et al. The p66shc adaptor protein controls oxidative stress response and life span in mammals , 1999, Nature.
[7] S. Nishikawa,et al. Removal of stem cell factor or addition of monoclonal anti-c-KIT antibody induces apoptosis in murine melanocyte precursors. , 1999, The Journal of investigative dermatology.
[8] R. Boissy,et al. What's the use of generating melanin? , 1999, Experimental dermatology.
[9] D. Tobin,et al. Do hair bulb melanocytes undergo apoptosis during hair follicle regression (catagen)? , 1998, The Journal of investigative dermatology.
[10] D. Champeval,et al. Endothelin 3 selectively promotes survival and proliferation of neural crest-derived glial and melanocytic precursors in vitro. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[11] M. Picardo,et al. Increased sensitivity to peroxidizing agents is correlated with an imbalance of antioxidants in normal melanocytes from melanoma patients , 1998, Experimental dermatology.
[12] J. Campisi. The role of cellular senescence in skin aging. , 1998, The journal of investigative dermatology. Symposium proceedings.
[13] B. A. Gilchrest,et al. What controls melanogenesis? , 1998, Experimental dermatology.
[14] Carina,et al. Analysis of apoptosis during hair follicle regression (catagen) , 1997, The American journal of pathology.
[15] I. Reid,et al. Premature hair graying and bone mineral density. , 1997, The Journal of clinical endocrinology and metabolism.
[16] B. Gilchrest,et al. Human melanocytes as a model system for studies of Alzheimer disease. , 1997, Archives of dermatology.
[17] B. Gilchrest,et al. Nerve growth factor rescues pigment cells from ultraviolet-induced apoptosis by upregulating BCL-2 levels. , 1996, Experimental cell research.
[18] Ian Jackson,et al. Variants of the melanocyte–stimulating hormone receptor gene are associated with red hair and fair skin in humans , 1995, Nature Genetics.
[19] K. Saga,et al. Proliferating Activity of the Hair Follicular Melanocytes at the Early and Anagen III Stages in the Hair Growth Cycle: Detection by Immunocytochemistry for Bromodeoxyuridine Combined with DOPA Reaction Cytochemistry , 1995, The Journal of dermatology.
[20] W. Nagl. Different growth rates of pigmented and white hair in the beard: differentiation vs. proliferation? , 1995, The British journal of dermatology.
[21] M. Holick,et al. Premature graying of hair is a risk marker for osteopenia. , 1994, The Journal of clinical endocrinology and metabolism.
[22] A. Chakraborty,et al. Melanogenesis During the Anagen-Catagen-Telogen Transformation of the Murine Hair Cycle , 1994 .
[23] D. Loh,et al. Targeted disruption of Bcl-2 alpha beta in mice: occurrence of gray hair, polycystic kidney disease, and lymphocytopenia. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[24] Z. Oltvai,et al. Bcl-2 functions in an antioxidant pathway to prevent apoptosis , 1993, Cell.
[25] D. Schadendorf,et al. Melanocytes as "sensory" and regulatory cells in the epidermis. , 1993, Journal of theoretical biology.
[26] R. Paus,et al. Melanogenesis is coupled to murine anagen: toward new concepts for the role of melanocytes and the regulation of melanogenesis in hair growth. , 1993, The Journal of investigative dermatology.
[27] D. Tobin,et al. Partial reversal of canities in a 22-year-old normal Chinese male. , 1993, Archives of dermatology.
[28] K. Takada,et al. Presence of amelanotic melanocytes within the outer root sheath in senile white hair. , 1992, The Journal of investigative dermatology.
[29] I. Jackson,et al. Light is a dominant mouse mutation resulting in premature cell death , 1992, Nature Genetics.
[30] D. Paslier,et al. Isolation of chromosome 21–specific yeast artificial chromosomes from a total human genome library , 1992, Nature Genetics.
[31] G. Westgate,et al. Immunology of the Hair Follicle , 1991, Annals of the New York Academy of Sciences.
[32] D. McGee,et al. Induction of skin tanning by subcutaneous administration of a potent synthetic melanotropin. , 1991, JAMA.
[33] R. Paus,et al. Differential expression and activity of melanogenesis-related proteins during induced hair growth in mice. , 1991, The Journal of investigative dermatology.
[34] D. Tobin,et al. Ultrastructural observations on the hair bulb melanocytes and melanosomes in acute alopecia areata. , 1990, The Journal of investigative dermatology.
[35] B. Gilchrest,et al. Effect of chronologic aging and ultraviolet irradiation on Langerhans cells in human epidermis. , 1982, The Journal of investigative dermatology.
[36] A. Jarrett. Biology and Disease of the Hair , 1977 .
[37] M. Allende. The enigmas of pigmentation. , 1972, JAMA.
[38] H. B. Chase,et al. DNA synthesis in the adult hair germ during dormancy (telogen) and activation (early anagen). , 1970, Developmental biology.
[39] W. C. Quevedo,et al. Influence of age and UV on the populations of dopa-positive melanocytes in human skin. , 1969, The Journal of investigative dermatology.
[40] E. Keogh,et al. Rate of Greying of Human Hair , 1965, Nature.
[41] L. Hayflick. THE LIMITED IN VITRO LIFETIME OF HUMAN DIPLOID CELL STRAINS. , 1965, Experimental cell research.
[42] J. Nordlund. The pigmentary system : physiology and pathophysiology , 1998 .
[43] T. Horikawa,et al. DOPA-negative melanocytes in the outer root sheath of human hair follicles express premelanosomal antigens but not a melanosomal antigen or the melanosome-associated glycoproteins tyrosinase, TRP-1, and TRP-2. , 1996, The Journal of investigative dermatology.
[44] D. Tobin,et al. Isolation and long-term culture of human hair-follicle melanocytes. , 1995, The Journal of investigative dermatology.
[45] V. Riley. Pigmentation; its genesis and biologic control , 1972 .