Cuprizone-induced demyelination in mice: age-related vulnerability and exploratory behavior deficit

[1]  Lan Xiao,et al.  Improving myelin/oligodendrocyte-related dysfunction: a new mechanism of antipsychotics in the treatment of schizophrenia? , 2013, The international journal of neuropsychopharmacology.

[2]  H. Kazui,et al.  The KCNH2 gene is associated with neurocognition and the risk of schizophrenia , 2013, The world journal of biological psychiatry : the official journal of the World Federation of Societies of Biological Psychiatry.

[3]  Jianrong Li,et al.  Demyelination and remyelination in anatomically distinct regions of the corpus callosum following cuprizone intoxication , 2012, Neuroscience Research.

[4]  H. Kinney,et al.  Aberrant Upregulation of Astroglial Ceramide Potentiates Oligodendrocyte Injury , 2012, Brain pathology.

[5]  L. Fañanás,et al.  Neurodevelopmental liability to schizophrenia: The complex mediating role of age at onset and premorbid adjustment , 2011, Schizophrenia Research.

[6]  S. Ludwin,et al.  Neurobiological effects of sphingosine 1‐phosphate receptor modulation in the cuprizone model , 2011, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[7]  Christos Pantelis,et al.  Age of onset of schizophrenia: perspectives from structural neuroimaging studies. , 2011, Schizophrenia bulletin.

[8]  K. Davis,et al.  Linking oligodendrocyte and myelin dysfunction to neurocircuitry abnormalities in schizophrenia , 2011, Progress in Neurobiology.

[9]  C. Konradi,et al.  Myelin, copper, and the cuprizone model of schizophrenia. , 2011, Frontiers in bioscience.

[10]  J. Newcombe,et al.  Contribution of Astrocyte-Derived IL-15 to CD8 T Cell Effector Functions in Multiple Sclerosis , 2010, The Journal of Immunology.

[11]  H. Naqvi,et al.  Gender difference in age at onset of schizophrenia: a cross sectional study from Pakistan. , 2010, JPMA. The Journal of the Pakistan Medical Association.

[12]  V. Haroutunian,et al.  Gene expression abnormalities and oligodendrocyte deficits in the internal capsule in schizophrenia , 2010, Schizophrenia Research.

[13]  K. Fujihara,et al.  Inflammation induced by innate immunity in the central nervous system leads to primary astrocyte dysfunction followed by demyelination , 2010, Acta Neuropathologica.

[14]  Haiyun Xu,et al.  Haloperidol activates quiescent oligodendroglia precursor cells in the adult mouse brain , 2010, Schizophrenia Research.

[15]  C. Beyer,et al.  Cuprizone effect on myelination, astrogliosis and microglia attraction in the mouse basal ganglia , 2009, Brain Research.

[16]  Takahira Yamauchi,et al.  Demyelination in the juvenile period, but not in adulthood, leads to long-lasting cognitive impairment and deficient social interaction in mice , 2009, Progress in Neuro-Psychopharmacology and Biological Psychiatry.

[17]  Martin Stangel,et al.  Regional differences between grey and white matter in cuprizone induced demyelination , 2009, Brain Research.

[18]  S. Floresco,et al.  Disruptions in spatial working memory, but not short-term memory, induced by repeated ketamine exposure , 2009, Progress in Neuro-Psychopharmacology and Biological Psychiatry.

[19]  R. Browning,et al.  Region-specific susceptibilities to cuprizone-induced lesions in the mouse forebrain: Implications for the pathophysiology of schizophrenia , 2009, Brain Research.

[20]  Richard Clough,et al.  Behavioral and neurobiological changes in C57BL/6 mice exposed to cuprizone. , 2009, Behavioral neuroscience.

[21]  Haiyun Xu,et al.  Quetiapine alleviates the cuprizone-induced white matter pathology in the brain of C57BL/6 mouse , 2008, Schizophrenia Research.

[22]  R. Franklin,et al.  Age-dependent epigenetic control of differentiation inhibitors is critical for remyelination efficiency , 2008, Nature Neuroscience.

[23]  J. He,et al.  Quetiapine facilitates oligodendrocyte development and prevents mice from myelin breakdown and behavioral changes , 2008, Molecular Psychiatry.

[24]  E. Vilella,et al.  Behavioral deficits in the cuprizone-induced murine model of demyelination/remyelination. , 2007, Toxicology letters.

[25]  J. Decety,et al.  Social brain dysfunctions in schizophrenia: A review of neuroimaging studies , 2006, Psychiatry Research: Neuroimaging.

[26]  Cheuk Y. Tang,et al.  Diffusion tensor imaging of frontal lobe white matter tracts in schizophrenia , 2006, Annals of general psychiatry.

[27]  W. F. Blakemore,et al.  Age increases axon loss associated with primary demyelination in cuprizone-induced demyelination in C57BL/6 mice , 2006, Journal of Neuroimmunology.

[28]  N. Jareborg,et al.  Human QKI, a potential regulator of mRNA expression of human oligodendrocyte-related genes involved in schizophrenia. , 2006, Proceedings of the National Academy of Sciences of the United States of America.

[29]  J. Neumaier,et al.  Differential effects of ovarian steroids on anxiety versus fear as measured by open field test and fear-potentiated startle , 2006, Behavioural Brain Research.

[30]  J. Garbern.  Pelizaeus-Merzbacher disease: Genetic and cellular pathogenesis , 2006, Cellular and Molecular Life Sciences.

[31]  Carl-Fredrik Westin,et al.  DTI and MTR abnormalities in schizophrenia: Analysis of white matter integrity , 2005, NeuroImage.

[32]  Dinesh Bhugra,et al.  The Global Prevalence of Schizophrenia , 2005, PLoS medicine.

[33]  K. Davis,et al.  Possible contributions of myelin and oligodendrocyte dysfunction to schizophrenia. , 2004, International review of neurobiology.

[34]  Ueli Suter,et al.  Quantifying the Early Stages of Remyelination Following Cuprizone‐induced Demyelination , 2003, Brain pathology.

[35]  T. Kinoshita,et al.  Diffusion Tensor Magnetic Resonance Imaging of Disruption of Regional White Matter in Schizophrenia , 2003, Neuropsychobiology.

[36]  W. Honer,et al.  Abnormalities of myelination in schizophrenia detected in vivo with MRI, and post-mortem with analysis of oligodendrocyte proteins , 2003, Molecular Psychiatry.

[37]  Chao Zhao,et al.  The Age-Related Decrease in CNS Remyelination Efficiency Is Attributable to an Impairment of Both Oligodendrocyte Progenitor Recruitment and Differentiation , 2002, The Journal of Neuroscience.

[38]  P. Morell,et al.  The Neurotoxicant, Cuprizone, as a Model to Study Demyelination and Remyelination in the Central Nervous System , 2001, Brain pathology.

[39]  D. Weinberger,et al.  From neuropathology to neurodevelopment , 1995, The Lancet.