Specific incorporation of cyclophilin A into HIV-1 virions

[1]  B. Rosenwirth,et al.  Inhibition of human immunodeficiency virus type 1 replication by SDZ NIM 811, a nonimmunosuppressive cyclosporine analog , 1994, Antimicrobial Agents and Chemotherapy.

[2]  J. Luban,et al.  Specificity and sequence requirements for interactions between various retroviral Gag proteins , 1994, Journal of virology.

[3]  S. Höglund,et al.  Role of the major homology region of human immunodeficiency virus type 1 in virion morphogenesis , 1994, Journal of virology.

[4]  J. Luban,et al.  Mutational analysis of cis-acting packaging signals in human immunodeficiency virus type 1 RNA , 1994, Journal of virology.

[5]  P. Sharp,et al.  Mosaic genome structure of simian immunodeficiency virus from west African green monkeys. , 1994, The EMBO journal.

[6]  W. Paxton,et al.  Incorporation of Vpr into human immunodeficiency virus type 1 virions: requirement for the p6 region of gag and mutational analysis , 1993, Journal of virology.

[7]  S. Schreiber,et al.  Identification of the immunophilins capable of mediating inhibition of signal transduction by cyclosporin A and FK506: roles of calcineurin binding and cellular location , 1993, Molecular and cellular biology.

[8]  Jeremy Luban,et al.  Human immunodeficiency virus type 1 Gag protein binds to cyclophilins A and B , 1993, Cell.

[9]  J. Sambrook,et al.  Proline isomerases function during heat shock. , 1993, Proceedings of the National Academy of Sciences of the United States of America.

[10]  G. Pavlakis,et al.  Mutational inactivation of an inhibitory sequence in human immunodeficiency virus type 1 results in Rev-independent gag expression , 1992, Journal of virology.

[11]  M. Lowdell,et al.  Inhibition of human immunodeficiency virus and growth of infected T cells by the immunosuppressive drugs cyclosporin A and FK 506. , 1992, Proceedings of the National Academy of Sciences of the United States of America.

[12]  J. Friedman,et al.  Two cytoplasmic candidates for immunophilin action are revealed by affinity for a new cyclophilin: One in the presence and one in the absence of CsA , 1991, Cell.

[13]  Stuart L. Schreiber,et al.  Calcineurin is a common target of cyclophilin-cyclosporin A and FKBP-FK506 complexes , 1991, Cell.

[14]  M. Godbout,et al.  An endoplasmic reticulum-specific cyclophilin , 1991, Molecular and cellular biology.

[15]  J. Luban,et al.  Binding of human immunodeficiency virus type 1 (HIV-1) RNA to recombinant HIV-1 gag polyprotein , 1991, Journal of virology.

[16]  J. Mullins,et al.  Sequence analysis and acute pathogenicity of molecularly cloned SIVSMM-PBj14 , 1990, Nature.

[17]  N. Pedersen,et al.  Induction of AIDS in rhesus monkeys by molecularly cloned simian immunodeficiency virus. , 1990, Science.

[18]  F. Schmid,et al.  The mechanism of protein folding. Implications of in vitro refolding models for de novo protein folding and translocation in the cell. , 1990, Biochemistry.

[19]  M. Wigler,et al.  Purification of a RAS-responsive adenylyl cyclase complex from Saccharomyces cerevisiae by use of an epitope addition method , 1988, Molecular and cellular biology.

[20]  Gilcher Ro Human retroviruses and AIDS. , 1988 .

[21]  M. Emerman,et al.  Genome organization and transactivation of the human immunodeficiency virus type 2 , 1987, Nature.

[22]  J. Mullins,et al.  Cloning of HTLV-4 and its relation to simian and human immunodeficiency viruses , 1987, Nature.

[23]  B. Cullen Use of eukaryotic expression technology in the functional analysis of cloned genes. , 1987, Methods in enzymology.

[24]  M. Harding,et al.  Cyclophilin: distribution and variant properties in normal and neoplastic tissues. , 1986, Journal of immunology.

[25]  H. Gendelman,et al.  Production of acquired immunodeficiency syndrome-associated retrovirus in human and nonhuman cells transfected with an infectious molecular clone , 1986, Journal of virology.

[26]  D. Baltimore,et al.  Quantitation of avian RNA tumor virus reverse transcriptase by radioimmunoassay , 1975, Journal of virology.