Acute gastrointestinal permeability responses to different non-steroidal anti-inflammatory drugs
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E. Smecuol | H. Vázquez | S. Niveloni | E. Mauriño | J. Bai | J. Meddings | S. Pedreira | E. Sugai
[1] J. Bolognese,et al. COX-2 inhibition with rofecoxib does not increase intestinal permeability in healthy subjects: a double blind crossover study comparing rofecoxib with placebo and indomethacin , 2000, Gut.
[2] I. Bjarnason,et al. Comparison of the intestinal toxicity of celecoxib, a selective COX-2 inhibitor, and indomethacin in the experimental rat. , 2000, Scandinavian journal of gastroenterology.
[3] Jacob,et al. Uncoupling of intestinal mitochondrial oxidative phosphorylation and inhibition of cyclooxygenase are required for the development of NSAID‐enteropathy in the rat , 2000, Alimentary pharmacology & therapeutics.
[4] A. Røseth,et al. High prevalence of NSAID enteropathy as shown by a simple faecal test , 1999, Gut.
[5] B. Thjódleifsson,et al. Gastrointestinal toxicity of non-steroidal anti-inflammatory drugs: the effect of nimesulide compared with naproxen on the human gastrointestinal tract. , 1999, Rheumatology.
[6] E. Hond,et al. Effect of glutamine on the intestinal permeability changes induced by indomethacin in humans , 1999, Alimentary pharmacology & therapeutics.
[7] M. Feldman,et al. Cyclooxygenase-1 and cyclooxygenase-2 selectivity of widely used nonsteroidal anti-inflammatory drugs. , 1998, The American journal of medicine.
[8] N. Davies,et al. Review article: non‐steroidal anti‐inflammatory drug‐induced gastrointestinal permeability , 1998, Alimentary pharmacology & therapeutics.
[9] D. Riendeau,et al. Comparison of the cyclooxygenase-1 inhibitory properties of nonsteroidal anti-inflammatory drugs (NSAIDs) and selective COX-2 inhibitors, using sensitive microsomal and platelet assays. , 1997, Canadian journal of physiology and pharmacology.
[10] W. S. Clark,et al. Association of Nonsteroidal Antiinflammatory Drugs with Outcome in Upper and Lower Gastrointestinal Bleeding , 1997, Digestive Diseases and Sciences.
[11] J. Evans,et al. Non-steroidal anti-inflammatory drugs are associated with emergency admission to hospital for colitis due to inflammatory bowel disease. , 1997, Gut.
[12] G. Oberhuber,et al. Gastrointestinal permeability in celiac disease. , 1997, Gastroenterology.
[13] C. Hawkey,et al. Review article: COX‐II inhibitors—a new generation of safer NSAIDs? , 1997, Alimentary pharmacology & therapeutics.
[14] Foutch Pg. Diverticular bleeding: are nonsteroidal anti-inflammatory drugs risk factors for hemorrhage and can colonoscopy predict outcome for patients? , 1995 .
[15] J. Wallace,et al. A simple, non-invasive marker of gastric damage: sucrose permeability , 1994, The Lancet.
[16] A. Florence,et al. Novel Oral Drug Formulations , 1994, Drug safety.
[17] A. Macpherson,et al. Side effects of nonsteroidal anti-inflammatory drugs on the small and large intestine in humans. , 1993, Gastroenterology.
[18] J. Wallace,et al. Sucrose: a novel permeability marker for gastroduodenal disease. , 1993, Gastroenterology.
[19] G. R. Davies,et al. Effects of metronidazole and misoprostol on indomethacin-induced changes in intestinal permeability , 1993, Digestive diseases and sciences.
[20] A. Macpherson,et al. Glucose and citrate reduce the permeability changes caused by indomethacin in humans. , 1992, Gastroenterology.
[21] E. Whitacre,et al. Colitis induced by nonsteroidal anti-inflammatory drugs. Report of four cases and review of the literature. , 1992, Archives of internal medicine.
[22] G. R. Davies,et al. The pro‐drug sulindac may reduce the risk of intestinal damage associated with the use of conventional non‐steroidal anti‐inflammatory drugs , 1991, Alimentary pharmacology & therapeutics.
[23] R. Steele,et al. Non‐steroidal anti‐inflammatory drugs and complicated diverticular disease: A case‐control study , 1991, The British journal of surgery.
[24] R. Madhok,et al. Enteroscopic diagnosis of small bowel ulceration in patients receiving non-steroidal anti-inflammatory drugs , 1991, The Lancet.
[25] A. Levi,et al. Importance of local versus systemic effects of non-steroidal anti-inflammatory drugs in increasing small intestinal permeability in man. , 1991, Gut.
[26] K. Seibert,et al. The induction and suppression of prostaglandin H2 synthase (cyclooxygenase) in human monocytes. , 1990, The Journal of biological chemistry.
[27] J. Marshall,et al. Acute eosinophilic colitis and hypersensitivity reaction associated with naproxen therapy. , 1990, The American journal of medicine.
[28] I. Bjarnason,et al. Experience with a gastrointestinal marker (51CrCl3) in a combined study of ileal function using 75SeHCAT and 58CoB12 measured by whole body counting. , 1990, Gut.
[29] L. Aabakken,et al. 51Cr-ethylenediaminetetraacetic acid absorption test. Effects of naproxen, a non-steroidal, antiinflammatory drug. , 1990, Scandinavian journal of gastroenterology.
[30] J. Paulley,et al. Reply , 1990, Gut.
[31] A. Swaak,et al. Anaemia in rheumatoid arthritis: the role of iron, vitamin B12, and folic acid deficiency, and erythropoietin responsiveness. , 1990, Annals of the rheumatic diseases.
[32] Hirschowitz Bi,et al. Controversies in the detection and management of nonsteroidal antiinflammatory drug-induced side effects of the upper gastrointestinal tract. , 1989 .
[33] A. Price,et al. Clinicopathological features of nonsteroidal antiinflammatory drug-induced small intestinal strictures. , 1988, Gastroenterology.
[34] A. Levi,et al. BLOOD AND PROTEIN LOSS VIA SMALL-INTESTINAL INFLAMMATION INDUCED BY NON-STEROIDAL ANTI-INFLAMMATORY DRUGS , 1987, The Lancet.
[35] S. Pai,et al. Intestinal perforaction following eneteral administration of indomethacin , 1985 .
[36] M. Langman,et al. Use of anti-inflammatory drugs by patients admitted with small or large bowel perforations and haemorrhage. , 1985, British medical journal.
[37] G. Amir,et al. Localized intestinal perforations after enteral administration of indomethacin in premature infants. , 1985, The Journal of pediatrics.
[38] J. Hoftiezer,et al. COMPARISON OF THE EFFECTS OF REGULAR AND ENTERIC-COATED ASPIRIN ON GASTRODUODENAL MUCOSA OF MAN , 1980, The Lancet.
[39] A. Hofmann,et al. Measurements of intestinal permeability using low molecular weight polyethylene glycols (PEG 400). I. Chemical analysis and biological properties of PEG 400. , 1977, Gastroenterology.
[40] J. Vane,et al. Inhibition of prostaglandin synthesis as a mechanism of action for aspirin-like drugs. , 1971, Nature: New biology.
[41] N. Davies,et al. Sustained release and enteric coated NSAIDs: are they really GI safe? , 1999, Journal of pharmacy & pharmaceutical sciences : a publication of the Canadian Society for Pharmaceutical Sciences, Societe canadienne des sciences pharmaceutiques.
[42] H. Ond,et al. Effect of glutamine on the intestinal permeability changes induced by indomethacin in humans , 1999 .
[43] J. Meddings,et al. Discrimination of site-specific alterations in gastrointestinal permeability in the rat. , 1998, Gastroenterology.
[44] R. Simpson,et al. Comparison of indomethacin and nimesulide, a selective cyclooxygenase-2 inhibitor, on key pathophysiologic steps in the pathogenesis of nonsteroidal anti-inflammatory drug enteropathy in the rat. , 1998, Scandinavian journal of gastroenterology.
[45] J. Vane,et al. Mechanism of action of antiinflammatory drugs. , 1998, International journal of tissue reactions.
[46] E. Smecuol,et al. Gastrointestinal permeability in celiac disease. , 1997, Gastroenterology.
[47] N. Davies,et al. Nonsteroidal anti-inflammatory drug enteropathy in rats: role of permeability, bacteria, and enterohepatic circulation. , 1997, Gastroenterology.
[48] J. Vane,et al. Mechanism of action of anti-inflammatory drugs. , 1996, Scandinavian journal of rheumatology. Supplement.
[49] P. Foutch. Diverticular bleeding: are nonsteroidal anti-inflammatory drugs risk factors for hemorrhage and can colonoscopy predict outcome for patients? , 1995, The American journal of gastroenterology.
[50] S. Pai,et al. Intestinal perforation following enteral administration of indomethacin. , 1985, The Journal of pediatrics.