Requirement for ERK Activation in Cisplatin-induced Apoptosis*
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N. Holbrook | Xiantao Wang | J. Martindale | Nikki J. Holbrook | Jennifer L. Martindale | Xiantao Wang
[1] M. Cobb,et al. Mitogen-activated protein kinase pathways. , 1997, Current opinion in cell biology.
[2] S. Howell,et al. Enhancement of drug sensitivity of human malignancies by epidermal growth factor. , 1995, British Journal of Cancer.
[3] Alan R. Saltiel,et al. Blockade of the MAP kinase pathway suppresses growth of colon tumors in vivo , 1999, Nature Medicine.
[4] Qingbo Xu,et al. Activation of Mitogen-activated Protein Kinase by HO , 1996, The Journal of Biological Chemistry.
[5] Bertram Wiedenmann,et al. Oxidative Stress Activates the Human Histidine Decarboxylase Promoter in AGS Gastric Cancer Cells* , 1998, The Journal of Biological Chemistry.
[6] L. Zon,et al. The stress-activated protein kinase pathway mediates cell death following injury induced by cis-platinum, UV irradiation or heat , 1996, Current Biology.
[7] J. Yodoi,et al. Redox control of resistance to cis-diamminedichloroplatinum (II) (CDDP): protective effect of human thioredoxin against CDDP-induced cytotoxicity. , 1996, The Journal of clinical investigation.
[8] A. Miyajima,et al. Role of reactive oxygen species in cis-dichlorodiammineplatinum-induced cytotoxicity on bladder cancer cells. , 1997, British Journal of Cancer.
[9] M. Gorospe,et al. p27Kip1 overexpression causes apoptotic death of mammalian cells , 1997, Oncogene.
[10] Y. Zou,et al. Oxidative stress activates extracellular signal-regulated kinases through Src and Ras in cultured cardiac myocytes of neonatal rats. , 1997, The Journal of clinical investigation.
[11] D. Mercola,et al. Inhibition of Extracellular Signal-regulated Protein Kinase or c-Jun N-terminal Protein Kinase Cascade, Differentially Activated by Cisplatin, Sensitizes Human Ovarian Cancer Cell Line* , 1999, The Journal of Biological Chemistry.
[12] N. Mulder,et al. Modulation of cis-diamminedichloroplatinum(II) resistance: a review. , 1992, British Journal of Cancer.
[13] M. Nagasawa,et al. 12-O-Tetradecanoylphorbol-13acetate Activates the Ras/ Extracellular Signal-regulated Kinase (ERK) Signaling Pathway Upstream of SOS Involving Serine Phosphorylation of Shc in NIH3T3 Cells* , 1997, The Journal of Biological Chemistry.
[14] R. Davis,et al. The role of c-Jun N-terminal kinase (JNK) in apoptosis induced by ultraviolet C and gamma radiation. Duration of JNK activation may determine cell death and proliferation. , 1996, The Journal of biological chemistry.
[15] C. Arteaga,et al. Abrogation of cisplatin-induced programmed cell death in human breast cancer cells by epidermal growth factor antisense RNA. , 1997, Journal of the National Cancer Institute.
[16] B. Mossman,et al. Asbestos causes stimulation of the extracellular signal-regulated kinase 1 mitogen-activated protein kinase cascade after phosphorylation of the epidermal growth factor receptor. , 1996, Cancer research.
[17] R. Perona,et al. Cisplatin induces a persistent activation of JNK that is related to cell death , 1998, Oncogene.
[18] Brent R. Stockwell,et al. An Induced Proximity Model for Caspase-8 Activation* , 1998, The Journal of Biological Chemistry.
[19] Xiaodong Wang,et al. Induction of Apoptotic Program in Cell-Free Extracts: Requirement for dATP and Cytochrome c , 1996, Cell.
[20] G. Chu,et al. Cellular responses to cisplatin. The roles of DNA-binding proteins and DNA repair. , 1994, The Journal of biological chemistry.
[21] M. Gold,et al. Differential activation of the ERK, JNK, and p38 mitogen-activated protein kinases by CD40 and the B cell antigen receptor. , 1996, Journal of immunology.
[22] N. Ahn,et al. Transformation of mammalian cells by constitutively active MAP kinase kinase. , 1994, Science.
[23] G. Rao. Hydrogen peroxide induces complex formation of SHC-Grb2-SOS with receptor tyrosine kinase and activates Ras and extracellular signal-regulated protein kinases group of mitogen-activated protein kinases. , 1996, Oncogene.
[24] M. Kuroda,et al. Sensitivity of anticancer drugs in NIH3T3' cells transfected with oncogenes accompanied by pSV2neo vector. , 1995, Anticancer Research.
[25] A. Bridges,et al. A synthetic inhibitor of the mitogen-activated protein kinase cascade. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[26] Dean P. Jones,et al. Mitochondrial control of apoptosis: the role of cytochrome c. , 1998, Biochimica et biophysica acta.
[27] Michael E. Greenberg,et al. Opposing Effects of ERK and JNK-p38 MAP Kinases on Apoptosis , 1995, Science.
[28] M. Gorospe,et al. Phorbol ester-induced mononuclear cell differentiation is blocked by the mitogen-activated protein kinase kinase (MEK) inhibitor PD98059. , 1999, Cell growth & differentiation : the molecular biology journal of the American Association for Cancer Research.
[29] J. Avruch,et al. Mitogen-activated protein kinase/extracellular signal-regulated protein kinase activation by oncogenes, serum, and 12-O-tetradecanoylphorbol-13-acetate requires Raf and is necessary for transformation. , 1994, The Journal of biological chemistry.
[30] D. Kufe,et al. Functional Role for Protein Kinase Cβ as a Regulator of Stress-Activated Protein Kinase Activation and Monocytic Differentiation of Myeloid Leukemia Cells , 1999, Molecular and Cellular Biology.
[31] X. Liu,et al. An APAF-1·Cytochrome c Multimeric Complex Is a Functional Apoptosome That Activates Procaspase-9* , 1999, The Journal of Biological Chemistry.
[32] N. Holbrook,et al. Tumor Promoter Arsenite Activates Extracellular Signal-Regulated Kinase through a Signaling Pathway Mediated by Epidermal Growth Factor Receptor and Shc , 1998, Molecular and Cellular Biology.
[33] Philip R. Cohen,et al. PD 098059 Is a Specific Inhibitor of the Activation of Mitogen-activated Protein Kinase Kinase in Vitro and in Vivo(*) , 1995, The Journal of Biological Chemistry.
[34] N. Park,et al. 12-O-Tetradecanoylphorbol-13-acetate (TPA)-induced c-Jun N-terminal Kinase (JNK) Phosphatase Renders Immortalized or Transformed Epithelial Cells Refractory to TPA-inducible JNK Activity* , 2000, The Journal of Biological Chemistry.
[35] G. Johnson,et al. Sequential protein kinase reactions controlling cell growth and differentiation. , 1994, Current opinion in cell biology.
[36] O. Potapova,et al. The Jun Kinase/Stress-activated Protein Kinase Pathway Functions to Regulate DNA Repair and Inhibition of the Pathway Sensitizes Tumor Cells to Cisplatin* , 1997, The Journal of Biological Chemistry.
[37] M. Moskowitz,et al. MEK1 protein kinase inhibition protects against damage resulting from focal cerebral ischemia. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[38] R. Perona,et al. Lack of c‐Jun activity increases survival to cisplatin , 1999, FEBS letters.
[39] N. Holbrook,et al. The cellular response to oxidative stress: influences of mitogen-activated protein kinase signalling pathways on cell survival. , 1998, The Biochemical journal.
[40] Xiaodong Wang,et al. Apaf-1, a Human Protein Homologous to C. elegans CED-4, Participates in Cytochrome c–Dependent Activation of Caspase-3 , 1997, Cell.
[41] J C Reed,et al. Mitochondria and apoptosis. , 1998, Science.
[42] D. Israeli,et al. p53 Activates the CD95 (APO-1/Fas) Gene in Response to DNA Damage by Anticancer Drugs , 1998, The Journal of experimental medicine.
[43] V. Cryns,et al. Proteases to die for. , 1998, Genes & development.
[44] E. Krebs,et al. Involvement of stress-activated protein kinase and p38 mitogen-activated protein kinase in mIgM-induced apoptosis of human B lymphocytes. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[45] O. Linderkamp,et al. Fas- or Ceramide-induced Apoptosis Is Mediated by a Rac1-regulated Activation of Jun N-terminal Kinase/p38 Kinases and GADD153* , 1997, The Journal of Biological Chemistry.
[46] J. Hansson,et al. The Ras farnesylation inhibitor BZA-5B increases the resistance to cisplatin in a human melanoma cell line. , 1997, Anticancer research.
[47] F. Hobbs,et al. Identification of a Novel Inhibitor of Mitogen-activated Protein Kinase Kinase* , 1998, The Journal of Biological Chemistry.
[48] E. Yazlovitskaya,et al. Cisplatin-induced activation of mitogen-activated protein kinases in ovarian carcinoma cells: inhibition of extracellular signal-regulated kinase activity increases sensitivity to cisplatin. , 1999, Clinical cancer research : an official journal of the American Association for Cancer Research.
[49] G. Chu,et al. Cisplatin-resistant cells express increased levels of a factor that recognizes damaged DNA. , 1990, Proceedings of the National Academy of Sciences of the United States of America.
[50] Sheila M. Thomas,et al. Ras is essential for nerve growth factor- and phorbol ester-induced tyrosine phosphorylation of MAP kinases , 1992, Cell.
[51] I. Herr,et al. The CD95 (APO-1/Fas) system mediates drug-induced apoptosis in neuroblastoma cells. , 1997, Cancer research.
[52] R. Perez,et al. Cellular and molecular determinants of cisplatin resistance. , 1998, European journal of cancer.
[53] S. Srinivasula,et al. Cytochrome c and dATP-Dependent Formation of Apaf-1/Caspase-9 Complex Initiates an Apoptotic Protease Cascade , 1997, Cell.