A Nuclear Function of β-Arrestin1 in GPCR Signaling: Regulation of Histone Acetylation and Gene Transcription

[1]  Minoru Yoshida,et al.  [Potent and specific inhibition of mammalian histone deacetylase both in vivo and in vitro by trichostatin A]. , 1990, Tanpakushitsu kakusan koso. Protein, nucleic acid, enzyme.

[2]  J. David Sweatt,et al.  Epigenetic mechanisms in memory formation , 2005, Nature Reviews Neuroscience.

[3]  U. Moens,et al.  What turns CREB on? , 2004, Cellular signalling.

[4]  P. Paez,et al.  Apotransferrin induces cAMP/CREB pathway and cell cycle exit in immature oligodendroglial cells , 2004, Journal of neuroscience research.

[5]  R. Lefkowitz,et al.  β-Arrestin inhibits NF-κB activity by means of its interaction with the NF-κB inhibitor IκBα , 2004 .

[6]  Yue Sun,et al.  Identification of β-Arrestin2 as a G Protein-Coupled Receptor-Stimulated Regulator of NF-κB Pathways , 2004 .

[7]  R. Schulz,et al.  Extracellular Signal-Regulated Kinase/Mitogen-Activated Protein Kinases Block Internalization of δ-Opioid Receptors , 2004, Journal of Pharmacology and Experimental Therapeutics.

[8]  Alexandre Benmerah.  Endocytosis: Signaling from Endocytic Membranes to the Nucleus , 2004, Current Biology.

[9]  Robert J. Lefkowitz,et al.  beta-arrestins: traffic cops of cell signaling. , 2004, Current opinion in cell biology.

[10]  Bianca Habermann,et al.  APPL Proteins Link Rab5 to Nuclear Signal Transduction via an Endosomal Compartment , 2004, Cell.

[11]  R. Lefkowitz,et al.  beta-Arrestin inhibits NF-kappaB activity by means of its interaction with the NF-kappaB inhibitor IkappaBalpha. , 2004, Proceedings of the National Academy of Sciences of the United States of America.

[12]  Yue Sun,et al.  Identification of beta-arrestin2 as a G protein-coupled receptor-stimulated regulator of NF-kappaB pathways. , 2004, Molecular cell.

[13]  Adam J. Smith,et al.  The Database of Interacting Proteins: 2004 update , 2004, Nucleic Acids Res..

[14]  R. Lefkowitz,et al.  Multifaceted roles of β-arrestins in the regulation of seven-membrane-spanning receptor trafficking and signalling , 2003 .

[15]  V. Pospelov,et al.  Transient expression of E1A and Ras oncogenes causes downregulation of c-fos gene transcription in nontransformed REF52 cells , 2003, Oncogene.

[16]  D. Moazed,et al.  Heterochromatin and Epigenetic Control of Gene Expression , 2003, Science.

[17]  Jiuhong Kang,et al.  Nickel-induced histone hypoacetylation: the role of reactive oxygen species. , 2003, Toxicological sciences : an official journal of the Society of Toxicology.

[18]  P. Eriksson,et al.  Opioid-induced proliferation through the MAPK pathway in cultures of adult hippocampal progenitors , 2003, Molecular and Cellular Neuroscience.

[19]  J. Massagué,et al.  Mechanisms of TGF-β Signaling from Cell Membrane to the Nucleus , 2003, Cell.

[20]  S. Henikoff,et al.  Maintenance of chromatin states: an open-and-shut case. , 2003, Current opinion in cell biology.

[21]  Ping Wang,et al.  Subcellular Localization of β-Arrestins Is Determined by Their Intact N Domain and the Nuclear Export Signal at the C Terminus* , 2003, The Journal of Biological Chemistry.

[22]  Max Costa,et al.  Epigenetics and the Environment , 2003, Annals of the New York Academy of Sciences.

[23]  Ping Wang,et al.  β-Arrestin 2 Functions as a G-Protein-coupled Receptor-activated Regulator of Oncoprotein Mdm2* , 2003, The Journal of Biological Chemistry.

[24]  B. Sharp,et al.  Phosphorylation of activating transcription factor in murine splenocytes through delta opioid receptors. , 2003, Cellular immunology.

[25]  Clifford S. Deutschman,et al.  Transcription , 2003, The Quran: Word List (Volume 3).

[26]  J. Massagué,et al.  Mechanisms of TGF-beta signaling from cell membrane to the nucleus. , 2003, Cell.

[27]  Yue Sun,et al.  β-Arrestin2 Is Critically Involved in CXCR4-mediated Chemotaxis, and This Is Mediated by Its Enhancement of p38 MAPK Activation* , 2002, The Journal of Biological Chemistry.

[28]  S. Benichou,et al.  Differential Nucleocytoplasmic Shuttling of β-Arrestins , 2002, The Journal of Biological Chemistry.

[29]  J. Darnell,et al.  Signalling: STATs: transcriptional control and biological impact , 2002, Nature Reviews Molecular Cell Biology.

[30]  Prahlad T. Ram,et al.  G Protein Pathways , 2002, Science.

[31]  C. Peterson,et al.  Unlocking the Gates to Gene Expression , 2002, Science.

[32]  M. Caron,et al.  Endocytosis of G protein-coupled receptors: roles of G protein-coupled receptor kinases and ß-arrestin proteins , 2002, Progress in Neurobiology.

[33]  S. Benichou,et al.  Differential nucleocytoplasmic shuttling of beta-arrestins. Characterization of a leucine-rich nuclear export signal in beta-arrestin2. , 2002, The Journal of biological chemistry.

[34]  S. Schreiber,et al.  Identification of a Class of Small Molecule Inhibitors of the Sirtuin Family of NAD-dependent Deacetylases by Phenotypic Screening* , 2001, The Journal of Biological Chemistry.

[35]  Marc Montminy,et al.  Transcriptional regulation by the phosphorylation-dependent factor CREB , 2001, Nature Reviews Molecular Cell Biology.

[36]  A. Wolffe,et al.  Chromatin remodeling and transcriptional activation: the cast (in order of appearance) , 2001, Oncogene.

[37]  T. Kohout,et al.  beta-Arrestin 1 and 2 differentially regulate heptahelical receptor signaling and trafficking. , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[38]  Ioannis Xenarios,et al.  DIP: The Database of Interacting Proteins: 2001 update , 2001, Nucleic Acids Res..

[39]  K. Imai,et al.  Histone deacetylase inhibitors suppress IL-2-mediated gene expression prior to induction of apoptosis. , 2000 .

[40]  M. Caron,et al.  Differential Affinities of Visual Arrestin, βArrestin1, and βArrestin2 for G Protein-coupled Receptors Delineate Two Major Classes of Receptors* , 2000, The Journal of Biological Chemistry.

[41]  M. Kuo,et al.  Nickel compounds are novel inhibitors of histone H4 acetylation. , 2000, Cancer research.

[42]  G. Pei,et al.  Endogenous δ‐Opioid and ORL1 Receptors Couple to Phosphorylation and Activation of p38 MAPK in NG108‐15 Cells and This Is Regulated by Protein Kinase A and Protein Kinase C , 1999 .

[43]  L. Ma,et al.  Endogenous delta-opioid and ORL1 receptors couple to phosphorylation and activation of p38 MAPK in NG108-15 cells and this is regulated by protein kinase A and protein kinase C. , 1999, Journal of neurochemistry.

[44]  A. Shaywitz,et al.  CREB: a stimulus-induced transcription factor activated by a diverse array of extracellular signals. , 1999, Annual review of biochemistry.

[45]  B. Stillman,et al.  Persistent initiation of DNA replication and chromatin-bound MCM proteins during the cell cycle in cdc6 mutants. , 1997, Genes & development.

[46]  R. Lefkowitz,et al.  Clathrin-mediated Endocytosis of the β-Adrenergic Receptor Is Regulated by Phosphorylation/Dephosphorylation of β-Arrestin1* , 1997, The Journal of Biological Chemistry.

[47]  M. Grunstein.  Histone acetylation in chromatin structure and transcription , 1997, Nature.

[48]  F. Zhong,et al.  Carboxyl terminus of delta opioid receptor is required for agonist-dependent receptor phosphorylation. , 1997, Biochemical and biophysical research communications.

[49]  L. Drury,et al.  Cdc6p-dependent loading of Mcm proteins onto pre-replicative chromatin in budding yeast. , 1997, Proceedings of the National Academy of Sciences of the United States of America.

[50]  Nobuyuki Shishido,et al.  Mice Lacking p27 Kip1 Display Increased Body Size, Multiple Organ Hyperplasia, Retinal Dysplasia, and Pituitary Tumors , 1996, Cell.

[51]  K. Manova-Todorova,et al.  Enhanced Growth of Mice Lacking the Cyclin-Dependent Kinase Inhibitor Function of p27 Kip1 , 1996, Cell.

[52]  M. Sallese,et al.  Molecular analysis of human beta-arrestin-1: cloning, tissue distribution, and regulation of expression. Identification of two isoforms generated by alternative splicing. , 1993, The Journal of biological chemistry.

[53]  W. Sadee,et al.  A human neuroblastoma cell line expresses mu and delta opioid receptor sites. , 1986, The Journal of biological chemistry.

[54]  T. Curran,et al.  Transient induction of c-fos and c-myc in an immediate consequence of growth factor stimulation. , 1985, Cancer surveys.

[55]  R. Roeder,et al.  Accurate transcription initiation by RNA polymerase II in a soluble extract from isolated mammalian nuclei. , 1983, Nucleic acids research.

[56]  B. Alberts,et al.  Different accessibilities in chromatin to histone acetylase. , 1979, The Journal of biological chemistry.