Angiotensin Converting Enzyme Inhibition and Angiotensin II AT1-Receptor Blockade Reduce the Levels of Asymmetrical NG, NG-Dimethylarginine in Human Essential Hypertension*

[1]  M. Kimoto,et al.  Renal expression of constitutive NOS and DDAH: separate effects of salt intake and angiotensin. , 2000, Kidney international.

[2]  J. Cooke,et al.  Does ADMA cause endothelial dysfunction? , 2000, Arteriosclerosis, thrombosis, and vascular biology.

[3]  D. Tsikas,et al.  LDL cholesterol upregulates synthesis of asymmetrical dimethylarginine in human endothelial cells: involvement of S-adenosylmethionine-dependent methyltransferases. , 2000, Circulation research.

[4]  A. Quyyumi,et al.  Acute and chronic angiotensin-1 receptor antagonism reverses endothelial dysfunction in atherosclerosis. , 2000, Circulation.

[5]  G. Mancini.  Long-term use of angiotensin-converting enzyme inhibitors to modify endothelial dysfunction: a review of clinical investigations. , 2000, Clinical and investigative medicine. Medecine clinique et experimentale.

[6]  R. Carey,et al.  Nitric oxide: a physiological mediator of the type 2 (AT2) angiotensin receptor. , 2000, Acta physiologica Scandinavica.

[7]  P. Tsao,et al.  Novel mechanism for endothelial dysfunction: dysregulation of dimethylarginine dimethylaminohydrolase. , 1999, Circulation.

[8]  R. Schmieder,et al.  Inadequate suppression of angiotensin II modulates left ventricular structure in humans. , 1997, Clinical nephrology.

[9]  V. Shah,et al.  Nitric oxide synthase inhibitors and hypertension in children and adolescents , 1997, Journal of hypertension.

[10]  S. Silbernagl,et al.  Compartmentation of amino acids in the rat kidney. , 1996, The American journal of physiology.

[11]  S. Moncada,et al.  Effect of local intra-arterial asymmetric dimethylarginine (ADMA) on the forearm arteriolar bed of healthy volunteers. , 1993, Journal of human hypertension.

[12]  S. Moncada,et al.  Accumulation of an endogenous inhibitor of nitric oxide synthesis in chronic renal failure , 1992, The Lancet.