Th1 cells promote neurite outgrowth from cortical neurons via a mechanism dependent on semaphorins.

[1]  E. Ward,et al.  Autoantigen Immunization at Different Sites Reveals a Role for Anti-Inflammatory Effects of IFN-γ in Regulating Susceptibility to Experimental Autoimmune Encephalomyelitis1 , 2009, The Journal of Immunology.

[2]  D. Wraith,et al.  Cutting Edge: Th1 Cells Facilitate the Entry of Th17 Cells to the Central Nervous System during Experimental Autoimmune Encephalomyelitis1 , 2008, The Journal of Immunology.

[3]  H. Cohen,et al.  IFN‐γ enhances neurogenesis in wild‐type mice and in a mouse model of Alzheimer's disease , 2008, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

[4]  Nathalie Arbour,et al.  Human TH17 lymphocytes promote blood-brain barrier disruption and central nervous system inflammation , 2007, Nature Medicine.

[5]  R. Nitsch,et al.  The role of T helper cells in neuroprotection and regeneration , 2007, Journal of Neuroimmunology.

[6]  P. Waite,et al.  Augmented locomotor recovery after spinal cord injury in the athymic nude rat. , 2006, Journal of neurotrauma.

[7]  M. Maeda,et al.  Semaphorin 4A induces growth cone collapse of hippocampal neurons in a Rho/Rho-kinase-dependent manner. , 2005, International journal of molecular medicine.

[8]  丸川 聡子.  Class IV semaphorin Sema4A enhances T-cell activation and interacts with Tim-2 , 2005 .

[9]  S. Akira,et al.  Nonredundant roles of Sema4A in the immune system: defective T cell priming and Th1/Th2 regulation in Sema4A-deficient mice. , 2005, Immunity.

[10]  D. Basso,et al.  Passive or Active Immunization with Myelin Basic Protein Impairs Neurological Function and Exacerbates Neuropathology after Spinal Cord Injury in Rats , 2004, The Journal of Neuroscience.

[11]  M. Azari,et al.  Degenerative and regenerative mechanisms governing spinal cord injury , 2004, Neurobiology of Disease.

[12]  H. Kikutani,et al.  Immune semaphorins: a new area of semaphorin research , 2003, Journal of Cell Science.

[13]  Jacques J. Peschon,et al.  Semaphorin 7A promotes axon outgrowth through integrins and MAPKs , 2003, Nature.

[14]  M. Hart,et al.  Activated/effector CD4+ T cells exacerbate acute damage in the central nervous system following traumatic injury , 2003, Journal of Neuroimmunology.

[15]  P. Lehmann,et al.  Autoreactive T cells promote post-traumatic healing in the central nervous system , 2003, Journal of Neuroimmunology.

[16]  Michal Schwartz,et al.  Neuroprotective autoimmunity: Naturally occurring CD4+CD25+ regulatory T cells suppress the ability to withstand injury to the central nervous system , 2002, Proceedings of the National Academy of Sciences of the United States of America.

[17]  H. Weiner,et al.  Protective Autoimmunity Is a Physiological Response to CNS Trauma , 2001, The Journal of Neuroscience.

[18]  E. Agranov,et al.  Passive or Active Immunization with Myelin Basic Protein Promotes Recovery from Spinal Cord Contusion , 2000, The Journal of Neuroscience.

[19]  S. Cullheim,et al.  Neuroprotection by Encephalomyelitis: Rescue of Mechanically Injured Neurons and Neurotrophin Production by CNS-Infiltrating T and Natural Killer Cells , 2000, The Journal of Neuroscience.

[20]  I. Bechmann,et al.  Axonal Damage Induced by Invading T Cells in Organotypic Central Nervous System Tissue in vitro: Involvement of Microglial Cells , 2000, Brain pathology.

[21]  K. Shirouzu,et al.  CDw108 expression during T-cell development. , 2000, Tissue antigens.

[22]  Irun R. Cohen,et al.  Autoimmune T cells protect neurons from secondary degeneration after central nervous system axotomy , 1999, Nature Medicine.

[23]  S. Kushimoto,et al.  Role of neutrophils in spinal cord injury in the rat , 1997, Neuroscience.