GM1 ganglioside attenuates convulsions and thiobarbituric acid reactive substances production induced by the intrastriatal injection of methylmalonic acid.
暂无分享,去创建一个
J. Bonini | J. Rocha | C. Dutra-filho | M. R. Fighera | C. F. Mello | M. Rubin | R. Frussa‐Filho | T. G. de Oliveira | M. Fighera
[1] J. Mocco,et al. Dehydroascorbic acid, a blood–brain barrier transportable form of vitamin C, mediates potent cerebroprotection in experimental stroke , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[2] J. Rocha,et al. Diphenyl diselenide and ascorbic acid changes deposition of selenium and ascorbic acid in liver and brain of mice. , 2001, Pharmacology & toxicology.
[3] C. Scriver,et al. The Metabolic and Molecular Bases of Inherited Disease, 8th Edition 2001 , 2001, Journal of Inherited Metabolic Disease.
[4] M. Wajner,et al. Methylmalonate administration decreases Na+,K+‐ATPase activity in cerebral cortex of rats , 2000, Neuroreport.
[5] M. Beal. Energetics in the pathogenesis of neurodegenerative diseases , 2000, Trends in Neurosciences.
[6] M. Rice. Ascorbate regulation and its neuroprotective role in the brain , 2000, Trends in Neurosciences.
[7] M. Wajner,et al. Propionic and L‐methylmalonic acids induce oxidative stress in brain of young rats , 2000, Neuroreport.
[8] G. Zeevalk,et al. Oxidative stress during energy impairment in mesencephalic cultures is not a downstream consequence of a secondary excitotoxicity , 2000, Neuroscience.
[9] M. Wajner,et al. Ascorbic acid and alpha-tocopherol attenuate methylmalonic acid-induced convulsions. , 1999, Neuroreport.
[10] Taro Tokui,et al. A family of mammalian Na+-dependent L-ascorbic acid transporters , 1999, Nature.
[11] J. Palermo-neto,et al. Kinetic analysis of GM1 effects on haloperidol-induced dopaminergic supersensitivity. , 1999, General pharmacology.
[12] D. Cassarino,et al. An evaluation of the role of mitochondria in neurodegenerative diseases: mitochondrial mutations and oxidative pathology, protective nuclear responses, and cell death in neurodegeneration , 1999, Brain Research Reviews.
[13] V. Maupoil,et al. Studies by electron paramagnetic resonance of the importance of iron in the hydroxyl scavenging properties of ascorbic acid in plasma: Effects of iron chelators , 1998, Fundamental & clinical pharmacology.
[14] M. Rice,et al. Estrogen-dependent modulation of rat brain ascorbate levels and ischemia-induced ascorbate loss , 1998, Brain Research.
[15] I. Silver,et al. Methylmalonate toxicity in primary neuronal cultures , 1998, Neuroscience.
[16] S. Tufik,et al. Effects of haloperidol and GM1 ganglioside treatment on striatal D2 receptor binding and dopamine turnover. , 1998, Life sciences.
[17] J. Baselga,et al. Vitamin C crosses the blood-brain barrier in the oxidized form through the glucose transporters. , 1997, The Journal of clinical investigation.
[18] B. Sabel,et al. The Role of Striatal Glutamatergic System in Haloperidol-Induced Dopamine Receptor Supersensitivity and Effects of Monosialoganglioside GM1 , 1997, Pharmacology Biochemistry and Behavior.
[19] A. Cuello,et al. Responses of cortical noradrenergic and somatostinergic fibres and terminals to adjacent strokes and subsequent treatment with NGF and/or the ganglioside GM1 , 1997, Journal of neuroscience research.
[20] J. Palermo-neto,et al. Effects of monosialoganglioside on a new model of tardive dyskinesia , 1997, Progress in Neuro-Psychopharmacology and Biological Psychiatry.
[21] R. H. Silva,et al. Effects of long-term ganglioside GM1 administration on a new discriminative avoidance test in normal adult mice. , 1997, Psychopharmacology.
[22] J. Palermo-neto,et al. Repeated monosialoganglioside administration attenuates behavioral sensitization to amphetamine , 1997, Brain Research.
[23] M. Rice,et al. Enhanced oxidative stress in female rat brain after gonadectomy , 1996, Brain Research.
[24] R. Camarini,et al. Monosialoganglioside attenuates the excitatory and behavioural sensitization effects of ethanol. , 1996, European journal of pharmacology.
[25] A. G. Nasello,et al. Effect of ganglioside (GM1) on memory in senescent rats , 1996, Neurobiology of Aging.
[26] J. Schulz,et al. Striatal Malonate Lesions Are Attenuated in Neuronal Nitric Oxide Synthase Knockout Mice , 1996, Journal of neurochemistry.
[27] M. Wajner,et al. Intrastriatal methylmalonic acid administration induces rotational behavior and convulsions through glutamatergic mechanisms , 1996, Brain Research.
[28] S. Bondy,et al. Effect of an NMDA receptor antagonist and a ganglioside GM1 derivative upon ethanol-induced modification of parameters of oxidative stress in several brain regions , 1996, Brain Research.
[29] J. Vera,et al. Resolution of the Facilitated Transport of Dehydroascorbic Acid from Its Intracellular Accumulation as Ascorbic Acid (*) , 1995, The Journal of Biological Chemistry.
[30] C. Epstein,et al. 3‐Nitropropionic Acid Neurotoxicity Is Attenuated in Copper/Zinc Superoxide Dismutase Transgenic Mice , 1995, Journal of neurochemistry.
[31] M. Rice,et al. Gender differences in cerebral ascorbate levels and ascorbate loss in ischemia , 1995, Neuroreport.
[32] J. Palermo-neto,et al. Effects of monosialoganglioside on dopaminergic supersensitivity. , 1995, Life sciences.
[33] B. Rosen,et al. Involvement of Free Radicals in Excitotoxicity In Vivo , 1995, Journal of neurochemistry.
[34] R. Reiter,et al. Oxidative processes and antioxidative defense mechanisms in the aging brain 1 , 1995, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[35] B. Rosen,et al. Coenzyme Q10 and nicotinamide block striatal lesions produced by the mitochondrial toxin malonate , 1994, Annals of neurology.
[36] Y. Tyurina,et al. Gangliosides in Postischemic Cellular Dysfunctions , 1994, Annals of the New York Academy of Sciences.
[37] J. Greene,et al. The endogenous cofactors, thioctic acid and dihydrolipoic acid, are neuroprotective against NMDA and malonic acid lesions of striatum , 1994, Neuroscience Letters.
[38] N. Maulik,et al. Reduction of Myocardial Ischemic Reperfusion Injury by Sialylated Glycosphingolipids, Gangliosides , 1993, Journal of cardiovascular pharmacology.
[39] R. Grünewald. Ascorbic acid in the brain , 1993, Brain Research Reviews.
[40] M. Wajner,et al. Inhibition of succinate dehydrogenase and β-hydroxybutyrate dehydrogenase activities by methylmalonate in brain and liver of developing rats , 1993, Journal of Inherited Metabolic Disease.
[41] M. Levine,et al. Ascorbic acid and dehydroascorbic acid measurements in human plasma and serum. , 1991, The American journal of clinical nutrition.
[42] H. Padh. Cellular functions of ascorbic acid. , 1990, Biochemistry and cell biology = Biochimie et biologie cellulaire.
[43] G. Rodriguez,et al. Sex Differences in Regional Cerebral Blood Flow , 1988, Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism.
[44] K. Yagi,et al. Assay for lipid peroxides in animal tissues by thiobarbituric acid reaction. , 1979, Analytical biochemistry.
[45] R. Brady,et al. Functional incorporation of ganglioside into intact cells: induction of choleragen responsiveness. , 1976, Proceedings of the National Academy of Sciences of the United States of America.
[46] S. Kaufman. Coenzymes and hydroxylases: ascorbate and dopamine-beta-hydroxylase; tetrahydropteridines and phenylalanine and tyrosine hydroxylases. , 1966, Pharmacological reviews.
[47] G. Ellman,et al. Tissue sulfhydryl groups. , 1959, Archives of biochemistry and biophysics.
[48] R. Yu,et al. Role of sphingolipid-mediated cell death in neurodegenerative diseases. , 1998, Journal of lipid research.
[49] K. Bagchi,et al. Evaluation of tissue ascorbic acid status in different hormonal states of female rat. , 1993, Life sciences.
[50] S. Trojan,et al. Ascorbic acid in the brain. , 1991, Physiological research.
[51] A. Carolei,et al. Monosialoganglioside GM1 in cerebral ischemia. , 1991, Cerebrovascular and brain metabolism reviews.
[52] R. Ghidoni,et al. Fundamentals of Brain Ganglioside Biosynthesis , 1987 .
[53] S. Seifter,et al. The biochemical functions of ascorbic acid. , 1986, Annual review of nutrition.
[54] G. Paxinos,et al. The Rat Brain in Stereotaxic Coordinates , 1983 .
[55] R. Chalmers,et al. Disorders of propionate and methylmalonate metabolism , 1982 .
[56] S. Hakomori. Glycosphingolipids in cellular interaction, differentiation, and oncogenesis. , 1981, Annual review of biochemistry.
[57] M. Nath,et al. Effect of male sex hormones on ascorbic acid metabolism in rats. , 1974, Journal of nutritional science and vitaminology.