Burn size determines the inflammatory and hypermetabolic response

[1]  D. Herndon,et al.  CYTOKINE EXPRESSION PROFILE OVER TIME IN SEVERELY BURNED PEDIATRIC PATIENTS , 2006, Shock.

[2]  D. Chinkes,et al.  Body composition changes with time in pediatric burn patients. , 2006, The Journal of trauma.

[3]  R. Barrow,et al.  Age-dependent differences in survival after severe burns: a unicentric review of 1,674 patients and 179 autopsies over 15 years. , 2006, Journal of the American College of Surgeons.

[4]  G. Van den Berghe,et al.  Intensive insulin therapy in the medical ICU. , 2006, The New England journal of medicine.

[5]  D. Herndon,et al.  Post burn muscle wasting and the effects of treatments. , 2005, The international journal of biochemistry & cell biology.

[6]  B. Johansson,et al.  Hormonal regulation of glucose and system A amino acid transport in first trimester placental villous fragments. , 2005, American journal of physiology. Regulatory, integrative and comparative physiology.

[7]  J. DiMaio,et al.  Macrophage migration inhibitory factor mediates late cardiac dysfunction after burn injury. , 2005, American journal of physiology. Heart and circulatory physiology.

[8]  R. Tompkins,et al.  Support of the metabolic response to burn injury , 2004, The Lancet.

[9]  Steven E Wolf,et al.  Hyperglycemia exacerbates muscle protein catabolism in burn-injured patients , 2002, Critical care medicine.

[10]  E. Deitch,et al.  Burn-Induced Impairment of Cardiac Contractile Function Is Due to Gut-Derived Factors Transported in Mesenteric Lymph , 2002, Shock.

[11]  G. Greeley,et al.  Ghrelin—not just another stomach hormone , 2002, Regulatory Peptides.

[12]  R. Barrow,et al.  IGF-I/IGFBP-3 Equilibrates Ratios of Pro- to Anti-inflammatory Cytokines, Which Are Predictors for Organ Function in Severely Burned Pediatric Patients , 2002, Molecular medicine.

[13]  D. Chinkes,et al.  Energy Expenditure and Caloric Balance After Burn: Increased Feeding Leads to Fat Rather Than Lean Mass Accretion , 2002, Annals of surgery.

[14]  G. V. Berghe,et al.  Intensive insulin therapy in critically ill patients. , 2001, The New England journal of medicine.

[15]  R. Barrow,et al.  IGF-I/BP-3 administration preserves hepatic homeostasis after thermal injury which is associated with increases in no and hepatic NF-kappa B. , 2001, Shock.

[16]  D. Chinkes,et al.  Association of hyperglycemia with increased mortality after severe burn injury. , 2001, The Journal of trauma.

[17]  D. Chinkes,et al.  Efficacy of a high-carbohydrate diet in catabolic illness , 2001, Critical care medicine.

[18]  R. Wolfe,et al.  Influence of glucose kinetics on plasma lactate concentration and energy expenditure in severely burned patients. , 2000, The Journal of trauma.

[19]  D. Chinkes,et al.  Determinants of Skeletal Muscle Catabolism After Severe Burn , 2000, Annals of surgery.

[20]  R. Demling,et al.  Metabolic Management of Patients with Severe Burns , 2000, World Journal of Surgery.

[21]  R. Barrow,et al.  Insulin-like growth factor I in combination with insulin-like growth factor binding protein 3 affects the hepatic acute phase response and hepatic morphology in thermally injured rats. , 2000, Annals of surgery.

[22]  R. Demling,et al.  Effect of a Hypocaloric Diet, Increased Protein Intake and Resistance Training on Lean Mass Gains and Fat Mass Loss in Overweight Police Officers , 2000, Annals of Nutrition and Metabolism.

[23]  D. Chinkes,et al.  Anabolic effects of insulin-like growth factor in combination with insulin-like growth factor binding protein-3 in severely burned adults. , 1999, The Journal of trauma.

[24]  D. Chinkes,et al.  Muscle protein catabolism after severe burn: effects of IGF-1/IGFBP-3 treatment. , 1999, Annals of surgery.

[25]  F. Chen,et al.  [Myocardial contractile and calcium transport function after severe burn injury]. , 1998, Zhonghua zheng xing shao shang wai ke za zhi = Zhonghua zheng xing shao shang waikf [i.e. waike] zazhi = Chinese journal of plastic surgery and burns.

[26]  P. Delhanty.  Interleukin-1 beta suppresses growth hormone-induced acid-labile subunit mRNA levels and secretion in primary hepatocytes. , 1998, Biochemical and biophysical research communications.

[27]  G. Biolo,et al.  Metabolic response to injury and sepsis: changes in protein metabolism. , 1997, Nutrition.

[28]  G. Biolo,et al.  An abundant supply of amino acids enhances the metabolic effect of exercise on muscle protein. , 1997, The American journal of physiology.

[29]  M. Binoux.  The IGF system in metabolism regulation. , 1995, Diabete & metabolisme.

[30]  G. Biolo,et al.  Increased rates of muscle protein turnover and amino acid transport after resistance exercise in humans. , 1995, The American journal of physiology.

[31]  W. Souba,et al.  Growth Hormone Enhances Amino Acid Uptake by the Human Small Intestine , 1994, Annals of surgery.

[32]  G. Biolo,et al.  Relationship Between Plasma Amino Acid Kinetics and Tissue Protein Synthesis and Breakdown , 1994 .

[33]  W. Souba,et al.  Growth hormone attenuates Na(+)-dependent hepatic amino acid transport in endotoxemic rats. , 1993, The Journal of trauma.

[34]  I. Macdonald,et al.  Increased whole body protein breakdown predominates over increased whole body protein synthesis in multiple organ failure. , 1993, Clinical science.

[35]  W. Souba,et al.  Growth hormone regulates amino acid transport in human and rat liver. , 1992, Annals of surgery.

[36]  L A Stephenson,et al.  Role of changes in insulin and glucagon in glucose homeostasis in exercise. , 1986, The Journal of clinical investigation.

[37]  J. H. Shaw,et al.  Response to glucose infusion in humans: role of changes in insulin concentration. , 1986, The American journal of physiology.

[38]  D. Wilmore,et al.  Hormonal responses and their effect on metabolism. , 1976, The Surgical clinics of North America.

[39]  A. Mason,et al.  Catecholamines: Znediator of the Hypermetabolic Response to Thermal Injury , 1974, Annals of surgery.

[40]  Goodall Mg.  Sympathetic nerve and adrenal medullary response to thermal burn. Clinical analysis of adrenaline and noradrenaline depletion. , 1966 .

[41]  B. Haynes,et al.  Urinary output of adrenaline and noradrenaline in severe thermal burns. , 1957, Annals of surgery.

[42]  J. B. Weir.  New methods for calculating metabolic rate with special reference to protein metabolism , 1949, The Journal of physiology.

[43]  B. Bistrian,et al.  Intensive insulin therapy in critically ill patients. , 2002, The New England journal of medicine.

[44]  G. Biolo,et al.  Transmembrane transport and intracellular kinetics of amino acids in human skeletal muscle. , 1995, The American journal of physiology.

[45]  M. Rennie,et al.  Muscle protein turnover and the wasting due to injury and disease. , 1985, British medical bulletin.

[46]  M. Goodall.  Sympathetic nerve and adrenal medullary response to thermal burn. Clinical analysis of adrenaline and noradrenaline depletion. , 1966, The American surgeon.