The relationship between acetylcholine-evoked Ca2+-dependent current and the Ca2+ concentrations in the cytosol and the lumen of the endoplasmic reticulum in pancreatic acinar cells
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[1] R. Schreiber,et al. Carbachol activates a K+ channel of very small conductance in the basolateral membrane of rat pancreatic acinar cells , 1999, Pflügers Archiv.
[2] D. Burdakov,et al. Regulation of store-operated calcium entry: lessons from a polarized cell. , 1999, European journal of cell biology.
[3] Grant C. Churchill,et al. Coordination of agonist-induced Ca2+-signalling patterns by NAADP in pancreatic acinar cells , 1999, Nature.
[4] T. Pozzan,et al. Free [Ca2+] dynamics measured in agonist‐sensitive stores of single living intact cells: a new look at the refilling process , 1998, The EMBO journal.
[5] O. Petersen,et al. The cyclic ADP ribose antagonist 8-NH2-cADP-ribose blocks cholecystokinin-evoked cytosolic Ca2+ spiking in pancreatic acinar cells , 1998, Pflügers Archiv.
[6] A. Tepikin,et al. Termination of cytosolic Ca2+ signals: Ca2+ reuptake into intracellular stores is regulated by the free Ca2+ concentration in the store lumen , 1998, The EMBO journal.
[7] R. Greger,et al. Chloride channels in the luminal membrane of rat pancreatic acini , 1997, Pflügers Archiv.
[8] A. Tepikin,et al. Ca2+ Flow via Tunnels in Polarized Cells: Recharging of Apical Ca2+ Stores by Focal Ca2+ Entry through Basal Membrane Patch , 1997, Cell.
[9] E. Krause,et al. Depletion of Intracellular Calcium Stores Activates a Calcium Conducting Nonselective Cation Current in Mouse Pancreatic Acinar Cells* , 1996, The Journal of Biological Chemistry.
[10] I. Schulz,et al. Quantification of intraluminal free [Ca] in the agonist-sensitive internal calcium store using compartmentalized fluorescent indicators: some considerations. , 1996, Cell calcium.
[11] M. Berridge,et al. Multiple, coordinated Ca2+ ‐release events underlie the inositol trisphosphate‐induced local Ca2+ spikes in mouse pancreatic acinar cells. , 1996, The EMBO journal.
[12] M. Berridge,et al. Capacitative calcium entry. , 1995, The Biochemical journal.
[13] O. Petersen,et al. Local and global cytosolic Ca2+ oscillations in exocrine cells evoked by agonists and inositol trisphosphate , 1993, Cell.
[14] Y. Miyashita,et al. Subcellular distribution of Ca2+ release channels underlying Ca2+ waves and oscillations in exocrine pancreas , 1993, Cell.
[15] O H Petersen,et al. Stimulus‐secretion coupling: cytoplasmic calcium signals and the control of ion channels in exocrine acinar cells. , 1992, The Journal of physiology.
[16] A. Tepikin,et al. Acetylcholine-evoked increase in the cytoplasmic Ca2+ concentration and Ca2+ extrusion measured simultaneously in single mouse pancreatic acinar cells. , 1992, The Journal of biological chemistry.
[17] Haruo Kasai,et al. Cytosolic Ca2+ gradients triggering unidirectional fluid secretion from exocrine pancreas , 1990, Nature.
[18] M. Wakui,et al. Cytoplasmic Ca2+ oscillations evoked by receptor stimulation, G‐protein activation, internal application of inositol trisphosphate or Ca2+: simultaneous microfluorimetry and Ca2+ dependent Cl‐ current recording in single pancreatic acinar cells. , 1990, The EMBO journal.
[19] Ole H. Petersen,et al. Pulsatile intracellular calcium release does not depend on fluctuations in inositol trisphosphate concentration , 1989, Nature.
[20] M. J. Berridge,et al. Release of Ca2+ from a nonmitochondrial intracellular store in pancreatic acinar cells by inositol-1,4,5-trisphosphate , 1983, Nature.
[21] C. Petersen,et al. Calcium and hormone action. , 1994, Annual review of physiology.
[22] A. Marty,et al. Diffusion into the patch-clamp recording pipette of a factor necessary for muscarinic current response. , 1989, Cellular signalling.