Transforming growth factor-beta 1 stimulates angiotensinogen gene expression in kidney proximal tubular cells.
暂无分享,去创建一个
J. Ingelfinger | Shao-Ling Zhang | D. Guo | Marie-Luise Brezniceanu | T. Hsieh | M. Hébert | J. Filep | J. Chan | C.-C. Wei | S. Zhang | C. Wei
[1] L. Cantley,et al. Signals Which Build a Tubule , 2005, Nephron Experimental Nephrology.
[2] S. Uh,et al. Role of Reactive Oxygen Species in TGF-β1-Induced Mitogen-Activated Protein Kinase Activation and Epithelial-Mesenchymal Transition in Renal Tubular Epithelial Cells , 2005 .
[3] K. Sharma,et al. Transforming growth factor-beta: A clinical target for the treatment of diabetic nephropathy , 2004, Current diabetes reports.
[4] H. Endou,et al. Oxidative stress and chronic allograft nephropathy. , 2004, Yonsei medical journal.
[5] G. Wolf,et al. Angiotensin II and Cell Cycle Regulation , 2004, Hypertension.
[6] Stephen W. Michnick,et al. PKB/Akt modulates TGF-β signalling through a direct interaction with Smad3 , 2004, Nature Cell Biology.
[7] K. Luo,et al. Akt interacts directly with Smad3 to regulate the sensitivity to TGF-β-induced apoptosis , 2004, Nature Cell Biology.
[8] Choon-Sik Park,et al. Reactive oxygen species mediate TGF-beta1-induced plasminogen activator inhibitor-1 upregulation in mesangial cells. , 2003, Biochemical and biophysical research communications.
[9] I. G. Fantus,et al. High glucose stimulates angiotensinogen gene expression and cell hypertrophy via activation of the hexosamine biosynthesis pathway in rat kidney proximal tubular cells. , 2003, Endocrinology.
[10] D. Sheppard,et al. Transforming Growth Factor-β-Dependent and -Independent Pathways of Induction of Tubulointerstitial Fibrosis in β6−/− Mice , 2003 .
[11] C. Steer,et al. Ursodeoxycholic Acid Modulates E2F-1 and p53 Expression through a Caspase-independent Mechanism in Transforming Growth Factor β1-induced Apoptosis of Rat Hepatocytes* , 2003, Journal of Biological Chemistry.
[12] H. Ha,et al. Reactive oxygen species-regulated signaling pathways in diabetic nephropathy. , 2003, Journal of the American Society of Nephrology : JASN.
[13] T. Kavanagh,et al. TGFβ1‐induced suppression of glutathione antioxidant defenses in hepatocytes: caspase‐dependent posttranslational and caspase‐independent transcriptional regulatory mechanisms , 2003, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[14] C. Wenner,et al. Biphasic role of TGF‐β1 in signal transduction and crosstalk , 2003 .
[15] C. Craciunescu,et al. Mitochondrial and microsomal derived reactive oxygen species mediate apoptosis induced by transforming growth factor‐β1 in immortalized rat hepatocytes , 2003, Journal of cellular biochemistry.
[16] S. Dupont,et al. Links between Tumor Suppressors p53 Is Required for TGF-β Gene Responses by Cooperating with Smads , 2003, Cell.
[17] Hisayo Fujita,et al. The lack of cyclin kinase inhibitor p27(Kip1) ameliorates progression of diabetic nephropathy. , 2003, Journal of the American Society of Nephrology : JASN.
[18] Sang-Gu Hwang,et al. p38 Kinase Regulates Nitric Oxide-induced Apoptosis of Articular Chondrocytes by Accumulating p53 via NFκB-dependent Transcription and Stabilization by Serine 15 Phosphorylation* , 2002, The Journal of Biological Chemistry.
[19] J. Ingelfinger,et al. High glucose stimulates angiotensinogen gene expression via reactive oxygen species generation in rat kidney proximal tubular cells. , 2002, Endocrinology.
[20] S. Shankland,et al. The hypertrophic effect of transforming growth factor-beta is reduced in the absence of cyclin-dependent kinase-inhibitors p21 and p27. , 2002, Journal of the American Society of Nephrology : JASN.
[21] J. Ingelfinger,et al. Essential role(s) of the intrarenal renin-angiotensin system in transforming growth factor-beta1 gene expression and induction of hypertrophy of rat kidney proximal tubular cells in high glucose. , 2002, Journal of the American Society of Nephrology : JASN.
[22] J. Ingelfinger,et al. Hyperglycemia induces insulin resistance on angiotensinogen gene expression in diabetic rat kidney proximal tubular cells. , 2002, The Journal of endocrinology.
[23] B. Safai,et al. Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death. , 2001, Diabetes.
[24] Robert A. Weinberg,et al. TGF-β-induced apoptosis is mediated by the adapter protein Daxx that facilitates JNK activation , 2001, Nature Cell Biology.
[25] S. Shankland,et al. High Glucose-Induced Hypertrophy of Mesangial Cells Requires p27Kip1, an Inhibitor of Cyclin-Dependent Kinases , 2001 .
[26] J. Sen. Signal transduction in thymus development. , 2001, Cellular and molecular biology.
[27] J. Ingelfinger,et al. Effect of Renin-Angiotensin System Blockade on the Expression of the Angiotensinogen Gene and Induction of Hypertrophy in Rat Kidney Proximal Tubular Cells , 2001, Nephron Experimental Nephrology.
[28] D. Poppas,et al. Antibody to transforming growth factor-beta ameliorates tubular apoptosis in unilateral ureteral obstruction. , 2000, Kidney international.
[29] K. Sharma,et al. Long-term prevention of renal insufficiency, excess matrix gene expression, and glomerular mesangial matrix expansion by treatment with monoclonal antitransforming growth factor-beta antibody in db/db diabetic mice. , 2000, Proceedings of the National Academy of Sciences of the United States of America.
[30] E. Lewis,et al. Captopril-induced reduction of serum levels of transforming growth factor-beta1 correlates with long-term renoprotection in insulin-dependent diabetic patients. , 1999, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[31] R. Atkins,et al. Transforming growth factor-beta regulates tubular epithelial-myofibroblast transdifferentiation in vitro. , 1999, Kidney international.
[32] J. Ingelfinger,et al. Molecular mechanisms of glucose action on angiotensinogen gene expression in rat proximal tubular cells. , 1999, Kidney international.
[33] Q. Li,et al. Stretch-mediated release of angiotensin II induces myocyte apoptosis by activating p53 that enhances the local renin-angiotensin system and decreases the Bcl-2-to-Bax protein ratio in the cell. , 1998, The Journal of clinical investigation.
[34] J. Ingelfinger,et al. Synergistic effect of dexamethasone and isoproterenol on the expression of angiotensinogen in immortalized rat proximal tubular cells. , 1998, Kidney international.
[35] B. Tomlinson,et al. Antioxidant power of angiotensin-converting enzyme inhibitors in vitro. , 1998, British journal of clinical pharmacology.
[36] M. Capogrossi,et al. p53 Induces myocyte apoptosis via the activation of the renin-angiotensin system. , 1997, Experimental cell research.
[37] U. Hopfer,et al. Effect of TGF-beta 1 and TNF-alpha on the plasminogen system of rat proximal tubular epithelial cells. , 1997, Journal of the American Society of Nephrology : JASN.
[38] A. Geiser,et al. The Liver in Transforming Growth Factor-Beta-1 (TGF-β1) Null Mutant Mice , 1996 .
[39] S. Thorgeirsson,et al. Transgenic mice with increased plasma levels of TGF-beta 1 develop progressive renal disease. , 1996, Laboratory investigation; a journal of technical methods and pathology.
[40] B. Fanburg,et al. Activation of an H2O2-generating NADH Oxidase in Human Lung Fibroblasts by Transforming Growth Factor β1 (*) , 1995, The Journal of Biological Chemistry.
[41] E Ruoslahti,et al. Expression of transforming growth factor beta is elevated in human and experimental diabetic nephropathy. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[42] R. Schneider,et al. Structure of the rat p53 tumor suppressor gene. , 1993, Nucleic acids research.
[43] T. Nakamura,et al. Production of extracellular matrix by glomerular epithelial cells is regulated by transforming growth factor-beta 1. , 1992, Kidney international.
[44] S. Carrière,et al. Hormonal regulation of expression of the angiotensinogen gene in cultured opossum kidney proximal tubular cells. , 1992, Journal of the American Society of Nephrology : JASN.
[45] M. Sporn,et al. Suppression of experimental glomerulonephritis by antiserum against transforming growth factor β1 , 1990, Nature.
[46] A. Roberts,et al. cDNA cloning by PCR of rat transforming growth factor β-1 , 1990 .
[47] E. Ruoslahti,et al. Transforming growth factor-β regulates production of proteoglycans by mesangial cells , 1990 .
[48] T. Gelehrter,et al. Cloning and sequencing of cDNA for the rat plasminogen activator inhibitor-1. , 1988, Gene.
[49] R. Richards,et al. Molecular cloning of the mouse angiotensinogen gene. , 1988, Genomics.
[50] R. Kageyama,et al. Cloning and sequence analysis of cDNA for rat angiotensinogen. , 1983, Proceedings of the National Academy of Sciences of the United States of America.
[51] M. Shani,et al. The nucleotide sequence of the rat cytoplasmic β–actin gene , 1983 .
[52] P. Vinay,et al. Isolation of a pure suspension of rat proximal tubules. , 1981, The American journal of physiology.
[53] T. Motyl,et al. Molecular mechanism of TGF-beta1-induced apoptosis in HC11 mouse mammary epithelial cells (MEC). , 2001, Cellular and molecular biology.
[54] N. Sarvetnick,et al. Under control of the Ren-1c promoter, locally produced transforming growth factor-beta1 induces accumulation of glomerular extracellular matrix in transgenic mice. , 1999, Diabetes.
[55] G. Wolf,et al. Angiotensin II-mediated expression of p27Kip1 and induction of cellular hypertrophy in renal tubular cells depend on the generation of oxygen radicals. , 1998, Kidney international.