Bach1 inhibits oxidative stress–induced cellular senescence by impeding p53 function on chromatin
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T. Noda | Y. Katoh | Minoru Yoshida | Y. Hoshikawa | A. Ito | T. Ohta | K. Igarashi | T. Ikura | S. Omura | A. Muto | A. Nakanome | K. Ota | Yoshihiro Dohi | Kazushige Ota
[1] Kevin Struhl,et al. Relationships between p63 binding, DNA sequence, transcription activity, and biological function in human cells. , 2006, Molecular cell.
[2] R. Bernards,et al. Plasminogen activator inhibitor-1 is a critical downstream target of p53 in the induction of replicative senescence , 2006, Nature Cell Biology.
[3] K. Igarashi,et al. The heme-Bach1 pathway in the regulation of oxidative stress response and erythroid differentiation. , 2006, Antioxidants & redox signaling.
[4] Z. Weng,et al. A Global Map of p53 Transcription-Factor Binding Sites in the Human Genome , 2006, Cell.
[5] K. Roemer,et al. NIR is a novel INHAT repressor that modulates the transcriptional activity of p53. , 2005, Genes & development.
[6] P. Chumakov,et al. The antioxidant function of the p53 tumor suppressor , 2005, Nature Medicine.
[7] H. Stein,et al. Oncogene-induced senescence as an initial barrier in lymphoma development , 2005, Nature.
[8] J. Shay,et al. BRAFE600-associated senescence-like cell cycle arrest of human naevi , 2005, Nature.
[9] M. Barbacid,et al. Tumour biology: Senescence in premalignant tumours , 2005, Nature.
[10] Jason A. Koutcher,et al. Crucial role of p53-dependent cellular senescence in suppression of Pten-deficient tumorigenesis , 2005, Nature.
[11] S. Numazawa,et al. Redox regulation of the transcriptional repressor Bach1. , 2005, Free radical biology & medicine.
[12] R. Weinberg,et al. The signals and pathways activating cellular senescence. , 2005, The international journal of biochemistry & cell biology.
[13] Y. Nishito,et al. Dynamic cytoplasmic anchoring of the transcription factor Bach1 by intracellular hyaluronic acid binding protein IHABP. , 2005, Journal of biochemistry.
[14] M. Toyofuku,et al. Effects of genetic ablation of bach1 upon smooth muscle cell proliferation and atherosclerosis after cuff injury , 2005, Genes to cells : devoted to molecular & cellular mechanisms.
[15] J. Campisi. Senescent Cells, Tumor Suppression, and Organismal Aging: Good Citizens, Bad Neighbors , 2005, Cell.
[16] S. Lowe,et al. Intrinsic tumour suppression , 2004, Nature.
[17] P. Pandolfi,et al. PML is a direct p53 target that modulates p53 effector functions. , 2004, Molecular cell.
[18] M. Brand,et al. Heme regulates the dynamic exchange of Bach1 and NF-E2-related factors in the Maf transcription factor network. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[19] T. Jacks,et al. Multiple response elements and differential p53 binding control Perp expression during apoptosis. , 2003, Molecular cancer research : MCR.
[20] S. Melov,et al. Oxygen sensitivity severely limits the replicative lifespan of murine fibroblasts , 2003, Nature Cell Biology.
[21] T. Finkel. Oxidant signals and oxidative stress. , 2003, Current opinion in cell biology.
[22] Hiroshi Suzuki,et al. Hemoprotein Bach1 regulates enhancer availability of heme oxygenase‐1 gene , 2002, The EMBO journal.
[23] R. Iggo,et al. Chromatin immunoprecipitation analysis fails to support the latency model for regulation of p53 DNA binding activity in vivo , 2001, Proceedings of the National Academy of Sciences of the United States of America.
[24] P. Pelicci,et al. PML interaction with p53 and its role in apoptosis and replicative senescence , 2001, Oncogene.
[25] Ettore Appella,et al. p300/CBP‐mediated p53 acetylation is commonly induced by p53‐activating agents and inhibited by MDM2 , 2001, The EMBO journal.
[26] Jun Qin,et al. Involvement of the TIP60 Histone Acetylase Complex in DNA Repair and Apoptosis , 2000, Cell.
[27] S. Lowe,et al. PERP, an apoptosis-associated target of p53, is a novel member of the PMP-22/gas3 family. , 2000, Genes & development.
[28] Yusuke Nakamura,et al. A ribonucleotide reductase gene involved in a p53-dependent cell-cycle checkpoint for DNA damage , 2000, Nature.
[29] A. Yang,et al. p63, a p53 homolog at 3q27-29, encodes multiple products with transactivating, death-inducing, and dominant-negative activities. , 1998, Molecular cell.
[30] S. Nishikawa,et al. Multivalent DNA Binding Complex Generated by Small Maf and Bach1 as a Possible Biochemical Basis for β-Globin Locus Control Region Complex* , 1998, The Journal of Biological Chemistry.
[31] E. Rogakou,et al. DNA Double-stranded Breaks Induce Histone H2AX Phosphorylation on Serine 139* , 1998, The Journal of Biological Chemistry.
[32] D. Wynford‐Thomas,et al. Reinitiation of DNA Synthesis and Cell Division in Senescent Human Fibroblasts by Microinjection of Anti-p53 Antibodies , 1998, Molecular and Cellular Biology.
[33] Hirofumi Tanaka,et al. Oncoprotein MDM2 is a ubiquitin ligase E3 for tumor suppressor p53 , 1997, FEBS letters.
[34] Yoichi Taya,et al. DNA Damage-Induced Phosphorylation of p53 Alleviates Inhibition by MDM2 , 1997, Cell.
[35] Wei Gu,et al. Activation of p53 Sequence-Specific DNA Binding by Acetylation of the p53 C-Terminal Domain , 1997, Cell.
[36] Wenyi Wei,et al. Bypass of senescence after disruption of p21CIP1/WAF1 gene in normal diploid human fibroblasts. , 1997, Science.
[37] Stephen N. Jones,et al. Regulation of p53 stability by Mdm2 , 1997, Nature.
[38] L. Chin,et al. Role for N-CoR and histone deacetylase in Sin3-mediated transcriptional repression , 1997, nature.
[39] C. Glass,et al. A complex containing N-CoR, mSln3 and histone deacetylase mediates transcriptional repression , 1997, nature.
[40] S. Lowe,et al. Oncogenic ras Provokes Premature Cell Senescence Associated with Accumulation of p53 and p16INK4a , 1997, Cell.
[41] H. Müller,et al. Influence of elevated expression of rat wild-type PMP22 and its mutant PMP22Trembler on cell growth of NIH3T3 fibroblasts , 1997, Cell and Tissue Research.
[42] K. Itoh,et al. Bach proteins belong to a novel family of BTB-basic leucine zipper transcription factors that interact with MafK and regulate transcription through the NF-E2 site , 1996, Molecular and cellular biology.
[43] C Roskelley,et al. A biomarker that identifies senescent human cells in culture and in aging skin in vivo. , 1995, Proceedings of the National Academy of Sciences of the United States of America.
[44] Ken Itoh,et al. Regulation of transcription by dimerization of erythroid factor NF-E2 p45 with small Maf proteins , 1994, Nature.
[45] K. Kataoka,et al. Two new members of the maf oncogene family, mafK and mafF, encode nuclear b-Zip proteins lacking putative trans-activator domain. , 1993, Oncogene.
[46] L. Donehower,et al. In vitro growth characteristics of embryo fibroblasts isolated from p53-deficient mice. , 1993, Oncogene.
[47] C. Purdie,et al. Thymocyte apoptosis induced by p53-dependent and independent pathways , 1993, Nature.