Bradykinin modulates spontaneous nerve growth factor production and stretch-induced ATP release in human urothelium.

[1]  J. Calixto,et al.  Effects of kinin B1 and B2 receptor antagonists on overactive urinary bladder syndrome induced by spinal cord injury in rats , 2012, British journal of pharmacology.

[2]  N. Yoshimura,et al.  Neurotrophins as regulators of urinary bladder function , 2012, Nature Reviews Urology.

[3]  John S. Young,et al.  Inhibition of stretching‐evoked ATP release from bladder mucosa by anticholinergic agents , 2012, BJU international.

[4]  P. Wiklund,et al.  Expression profiling of G‐protein‐coupled receptors in human urothelium and related cell lines , 2012, BJU international.

[5]  L. Gharat,et al.  Rat detrusor overactivity induced by chronic spinalization can be abolished by a transient receptor potential vanilloid 1 (TRPV1) antagonist , 2012, Autonomic Neuroscience.

[6]  P. Spano,et al.  Muscarinic receptors stimulate cell proliferation in the human urothelium-derived cell line UROtsa. , 2011, Pharmacological research.

[7]  C. Cruz,et al.  Nerve growth factor in bladder dysfunction: Contributing factor, biomarker, and therapeutic target , 2011, Neurourology and urodynamics.

[8]  C. Fry,et al.  Intracellular Ca(2+) regulation and electrophysiolgical properties of bladder urothelium subjected to stretch and exogenous agonists. , 2011, Cell calcium.

[9]  B. Uvelius,et al.  Biochemical and functional correlates of an increased membrane density of caveolae in hypertrophic rat urinary bladder. , 2010, European journal of pharmacology.

[10]  P. Dinis,et al.  Trigonal injection of botulinum toxin A in patients with refractory bladder pain syndrome/interstitial cystitis. , 2010, European urology.

[11]  R. Chess-Williams,et al.  In vitro release of adenosine triphosphate from the urothelium of human bladders with detrusor overactivity, both neurogenic and idiopathic. , 2010, European urology.

[12]  Y. Choi,et al.  Increased urinary nerve growth factor as a predictor of persistent detrusor overactivity after bladder outlet obstruction relief in a rat model. , 2010, The Journal of urology.

[13]  D. Cockayne,et al.  Overexpression of NGF in mouse urothelium leads to neuronal hyperinnervation, pelvic sensitivity, and changes in urinary bladder function. , 2010, American journal of physiology. Regulatory, integrative and comparative physiology.

[14]  B. Nilius,et al.  Functional characterization of transient receptor potential channels in mouse urothelial cells. , 2010, American journal of physiology. Renal physiology.

[15]  I. Saiki,et al.  Bradykinin increases the secretion and expression of endothelin-1 through kinin B2 receptors in melanoma cells , 2010, Peptides.

[16]  I. Nagy,et al.  Functional transient receptor potential vanilloid 1 is expressed in human urothelial cells. , 2009, The Journal of urology.

[17]  M. Tominaga,et al.  The TRPV4 Cation Channel Mediates Stretch-evoked Ca2+ Influx and ATP Release in Primary Urothelial Cell Cultures , 2009, The Journal of Biological Chemistry.

[18]  H. Kuo,et al.  Urinary nerve growth factor level could be a potential biomarker for diagnosis of overactive bladder. , 2008, The Journal of urology.

[19]  A. Ford,et al.  Expression and function of rat urothelial P2Y receptors. , 2008, American journal of physiology. Renal physiology.

[20]  W. D. de Groat,et al.  Heterogeneity of muscarinic receptor-mediated Ca2+ responses in cultured urothelial cells from rat. , 2008, American journal of physiology. Renal physiology.

[21]  L. Birder,et al.  The uroepithelial-associated sensory web. , 2007, Kidney international.

[22]  F. Cruz,et al.  Transient receptor potential vanilloid subfamily 1 is essential for the generation of noxious bladder input and bladder overactivity in cystitis. , 2007, The Journal of urology.

[23]  P. Santicioli,et al.  Characterization of kinin receptors in human cultured detrusor smooth muscle cells , 2007, British journal of pharmacology.

[24]  S. Lewis,et al.  Kinetics of urothelial ATP release. , 2006, American journal of physiology. Renal physiology.

[25]  E. Park,et al.  Effects of intravesical instillation of cyclooxygenase‐2 inhibitor on the expression of inducible nitric oxide synthase and nerve growth factor in cyclophosphamide‐induced overactive bladder , 2006, BJU international.

[26]  S. Murakami,et al.  Non-neuronal cholinergic system in human bladder urothelium. , 2006, Urology.

[27]  D. Cockayne,et al.  ATP and purinergic receptor-dependent membrane traffic in bladder umbrella cells. , 2005, The Journal of clinical investigation.

[28]  A. Ford,et al.  Expression and function of bradykinin B1 and B2 receptors in normal and inflamed rat urinary bladder urothelium , 2005, The Journal of physiology.

[29]  M. Vizzard,et al.  Cyclophosphamide induced cystitis alters neurotrophin and receptor tyrosine kinase expression in pelvic ganglia and bladder. , 2004, The Journal of urology.

[30]  A. Ford,et al.  Pharmacological and functional characterization of bradykinin B2 receptor in human prostate. , 2004, European journal of pharmacology.

[31]  Y. Nakata,et al.  Sensitization of vanilloid receptor 1 induced by bradykinin via the activation of second messenger signaling cascades in rat primary afferent neurons. , 2004, European journal of pharmacology.

[32]  Shinichi Watanabe,et al.  Histamine enhances the production of nerve growth factor in human keratinocytes. , 2003, The Journal of investigative dermatology.

[33]  J. Roppolo,et al.  Feline interstitial cystitis results in mechanical hypersensitivity and altered ATP release from bladder urothelium. , 2003, American journal of physiology. Renal physiology.

[34]  M. Caterina,et al.  Altered urinary bladder function in mice lacking the vanilloid receptor TRPV1 , 2002, Nature Neuroscience.

[35]  P. D. De Deyne,et al.  Augmented stretch activated adenosine triphosphate release from bladder uroepithelial cells in patients with interstitial cystitis. , 2001, The Journal of urology.

[36]  Simon C Watkins,et al.  Vanilloid receptor expression suggests a sensory role for urinary bladder epithelial cells , 2001, Proceedings of the National Academy of Sciences of the United States of America.

[37]  W. Steers,et al.  Histological and neurotrophic changes triggered by varying models of bladder inflammation. , 2001, The Journal of urology.

[38]  D. Cockayne,et al.  P2X3 Knock-Out Mice Reveal a Major Sensory Role for Urothelially Released ATP , 2001, The Journal of Neuroscience.

[39]  A. Basbaum,et al.  Bradykinin and nerve growth factor release the capsaicin receptor from PtdIns(4,5)P2-mediated inhibition , 2001, Nature.

[40]  A. Arner,et al.  Up-regulation of bradykinin response in rat and human bladder smooth muscle. , 2000, The Journal of urology.

[41]  W. Steers,et al.  Stretch‐activated signaling of nerve growth factor secretion in bladder and vascular smooth muscle cells from hypertensive and hyperactive rats , 2000, Journal of cellular physiology.

[42]  C. Pinna,et al.  Prostaglandin-release impairment in the bladder epithelium of streptozotocin-induced diabetic rats. , 2000, European journal of pharmacology.

[43]  M. Vizzard.  Changes in Urinary Bladder Neurotrophic Factor mRNA and NGF Protein Following Urinary Bladder Dysfunction , 2000, Experimental Neurology.

[44]  D. Pruneau,et al.  Pharmacological and molecular evidence for kinin B1 receptor expression in urinary bladder of cyclophosphamide‐treated rats , 1999, British journal of pharmacology.

[45]  C. Maggi,et al.  Kinin B1 receptor-mediated motor responses in normal or inflamed rat urinary bladder in vivo , 1999, Regulatory Peptides.

[46]  C. Maggi,et al.  Inflammation modifies the role of cyclooxygenases in the contractile responses of the rat detrusor smooth muscle to kinin agonists. , 1998, The Journal of pharmacology and experimental therapeutics.

[47]  A. Kanai,et al.  Adrenergic- and capsaicin-evoked nitric oxide release from urothelium and afferent nerves in urinary bladder. , 1998, American journal of physiology. Renal physiology.

[48]  I. Kennedy,et al.  ATP is released from rabbit urinary bladder epithelial cells by hydrostatic pressure changes–possible sensory mechanism? , 1997, The Journal of physiology.

[49]  D. Bjorling,et al.  Spontaneously released substance P and bradykinin from isolated guinea-pig bladder. , 1997, British journal of urology.

[50]  W. Steers,et al.  Protein kinase C in cyclic stretch-induced nerve growth factor production by urinary tract smooth muscle cells. , 1995, The American journal of physiology.

[51]  P. Brachet,et al.  Interactions between second messenger pathways influence NGF synthesis in mouse primary astrocytes , 1995, Brain Research.

[52]  C. Maggi,et al.  Pharmacological analysis of the local and reflex responses to bradykinin on rat urinary bladder motility in vivo , 1995, British journal of pharmacology.

[53]  C. Maggi,et al.  Effect of bradykinin and tachykinin receptor antagonist on xylene-induced cystitis in rats. , 1993 .

[54]  C. Maggi,et al.  Evidence for the involvement of bradykinin in chemically-evoked cystitis in anaesthetized rats , 1993, Naunyn-Schmiedeberg's Archives of Pharmacology.

[55]  G. Heinrich,et al.  Nerve growth factor gene expression: Involvement of a downstream AP-1 element in basal and modulated transcription , 1991, Molecular and Cellular Neuroscience.

[56]  G. Apodaca,et al.  Expression and distribution of transient receptor potential (TRP) channels in bladder epithelium. , 2011, American journal of physiology. Renal physiology.

[57]  W. C. Groat,et al.  Mechanisms of Disease: involvement of the urothelium in bladder dysfunction , 2007, Nature Clinical Practice Urology.

[58]  J. Masters,et al.  Immortalisation of human urothelial cells , 2004, Urological Research.