HIV-1-Resistant Strains during 8-Week on 8-Week off Intermittent Therapy and their Effect on CD4+ T-cell Counts and Antiviral Response
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[1] D. Katzenstein,et al. A Pilot Study Evaluating Time to CD4 T-cell Count <350 cells/mm3 After Treatment Interruption Following Antiretroviral Therapy ± Interleukin 2: Results of ACTG A5102 , 2006, Journal of acquired immune deficiency syndromes.
[2] D. Pillay,et al. Evolution of Transmitted HIV-1 with Drug-Resistance Mutations in the Absence of Therapy: Effects on Cd4+ T-Cell Count and HIV-1 Rna Load , 2006, Antiviral therapy.
[3] V. Calvez,et al. Repeated HIV‐1 resistance genotyping external quality assessments improve virology laboratory performance , 2006, Journal of medical virology.
[4] K. Tashima,et al. Pharmacogenetics of plasma efavirenz exposure after treatment discontinuation: an Adult AIDS Clinical Trials Group Study. , 2006, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.
[5] C. Rouzioux,et al. HIV-1 resistant strains acquired at the time of primary infection massively fuel the cellular reservoir and persist for lengthy periods of time , 2006, AIDS.
[6] C. Gil,et al. Risk of selecting de novo drug-resistance mutations during structured treatment interruptions in patients with chronic HIV infection. , 2005, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.
[7] T. Perneger,et al. Biphasic decline of CD4 cell count during scheduled treatment interruptions , 2005, AIDS.
[8] J. Ananworanich,et al. Development of HIV with drug resistance after CD4 cell count-guided structured treatment interruptions in patients treated with highly active antiretroviral therapy after dual-nucleoside analogue treatment. , 2005, Clinical infectious diseases : an official publication of the Infectious Diseases Society of America.
[9] D. Pillay,et al. Long-Term Persistence of Primary Genotypic Resistance After HIV-1 Seroconversion , 2004, Journal of acquired immune deficiency syndromes.
[10] L. Montaner,et al. Randomized, Controlled Trial of Therapy Interruption in Chronic HIV-1 Infection , 2004, PLoS medicine.
[11] M. Wainberg,et al. Persistence of multidrug-resistant HIV-1 in primary infection leading to superinfection , 2004, AIDS.
[12] M. Segal,et al. Persistence of primary drug resistance among recently HIV-1 infected adults , 2004, AIDS.
[13] B. Berkhout,et al. Declining trend in transmission of drug-resistant HIV-1 in Amsterdam , 2004, AIDS.
[14] Annapaola Callegaro,et al. Effect of prolonged discontinuation of successful antiretroviral therapy on CD4 T cells: a controlled, prospective trial , 2004, AIDS.
[15] K. Metzner,et al. Emergence of minor populations of human immunodeficiency virus type 1 carrying the M184V and L90M mutations in subjects undergoing structured treatment interruptions. , 2003, The Journal of infectious diseases.
[16] L. Montaner,et al. Lack of persistent drug-resistant mutations evaluated within and between treatment interruptions in chronically HIV-1-infected patients , 2003, AIDS.
[17] Michelle Le Braz,et al. Failures of 1 week on, 1 week off antiretroviral therapies in a randomized trial , 2003, AIDS.
[18] W. Rozenbaum,et al. Emergence of HIV-1 mutated strains after interruption of highly active antiretroviral therapy in chronically infected patients , 2003, AIDS.
[19] K. Hertogs,et al. Long-cycle structured intermittent versus continuous highly active antiretroviral therapy for the treatment of chronic infection with human immunodeficiency virus: effects on drug toxicity and on immunologic and virologic parameters. , 2003, The Journal of infectious diseases.
[20] A. Telenti,et al. A prospective trial of structured treatment interruptions in human immunodeficiency virus infection. , 2003, Archives of internal medicine.
[21] H. Günthard,et al. Drug Resistance Mutations during Structured Treatment Interruptions , 2002, Antiviral therapy.
[22] G. M. Ortiz,et al. Emergence of drug-resistant HIV-1 variants in patients undergoing structured treatment interruptions. , 2002, AIDS.
[23] P. Massip,et al. Evolution of total and integrated HIV-1 DNA and change in DNA sequences in patients with sustained plasma virus suppression. , 2002, Virology.
[24] J. Kahn,et al. Time trends in primary HIV-1 drug resistance among recently infected persons. , 2002, JAMA.
[25] M. Alvarez,et al. Evolution of human immunodeficiency virus type 1 populations after resumption of therapy following treatment interruption and shift in resistance genotype. , 2002, The Journal of infectious diseases.
[26] S. Deeks. Nonnucleoside Reverse Transcriptase Inhibitor Resistance , 2001 .
[27] K. Crandall,et al. Human immunodeficiency virus type 1 quasi species that rebound after discontinuation of highly active antiretroviral therapy are similar to the viral quasi species present before initiation of therapy. , 2001, The Journal of infectious diseases.
[28] K. Hertogs,et al. Virological and immunological effects of treatment interruptions in HIV-1 infected patients with treatment failure , 2000, AIDS.
[29] K. Lange,et al. Mutation Takes No Vacation: Can Structured Treatment Interruptions Increase the Risk of Drug‐Resistant HIV‐1? , 2000, Journal of acquired immune deficiency syndromes.
[30] G. M. Ortiz,et al. Risks and benefits of structured antiretroviral drug therapy interruptions in HIV-1 infection , 2000, AIDS.
[31] P. Massip,et al. Shift in HIV resistance genotype after treatment interruption and short-term antiviral effect following a new salvage regimen , 2000, AIDS.
[32] Anthony S. Fauci,et al. Relationship between pre-existing viral reservoirs and the re-emergence of plasma viremia after discontinuation of highly active anti-retroviral therapy , 2000, Nature Medicine.
[33] C. Verhofstede,et al. Interruption of reverse transcriptase inhibitors or a switch from reverse transcriptase to protease inhibitors resulted in a fast reappearance of virus strains with a reverse transcriptase inhibitor-sensitive genotype. , 1999, AIDS.
[34] M. Youle,et al. Rapid decline in detectability of HIV-1 drug resistance mutations after stopping therapy. , 1999, AIDS.
[35] R. Lempicki,et al. HIV-1 and T cell dynamics after interruption of highly active antiretroviral therapy (HAART) in patients with a history of sustained viral suppression. , 1999, Proceedings of the National Academy of Sciences of the United States of America.
[36] M. Plana,et al. Dynamics of viral load rebound and immunological changes after stopping effective antiretroviral therapy. , 1999, AIDS.
[37] V. Calvez,et al. HIV-1 rebound during interruption of highly active antiretroviral therapy has no deleterious effect on reinitiated treatment. Comet Study Group. , 1999, AIDS.
[38] E. Clercq. HIV resistance to reverse transcriptase inhibitors , 1994 .