Inflammatory cytokines in major surgery: a functional perspective

[1]  G. Wanner,et al.  Mixed agonistic-antagonistic cytokine response in whole blood from patients undergoing abdominal aortic aneurysm repair , 1999, Intensive Care Medicine.

[2]  S. Garg,et al.  Exogenous interleukin-10 fails to decrease the mortality or morbidity of sepsis. , 1998, Critical care medicine.

[3]  J. Nick,et al.  p38 Mitogen-activated Protein Kinase-dependent and -independent Intracellular Signal Transduction Pathways Leading to Apoptosis in Human Neutrophils* , 1998, The Journal of Biological Chemistry.

[4]  M. Jordana,et al.  IL-6 is an antiinflammatory cytokine required for controlling local or systemic acute inflammatory responses. , 1998, The Journal of clinical investigation.

[5]  J. Marshall,et al.  Dysregulated expression of neutrophil apoptosis in the systemic inflammatory response syndrome. , 1997, Archives of surgery.

[6]  J. Lederer,et al.  Major injury induces increased production of interleukin-10 by cells of the immune system with a negative impact on resistance to infection. , 1997, Annals of surgery.

[7]  D. Männel,et al.  Role of NFkappaB in the mortality of sepsis. , 1997, The Journal of clinical investigation.

[8]  S. Ashley,et al.  Interleukin-10 prevents early cytokine release in severe intraabdominal infection and sepsis. , 1997, The Journal of surgical research.

[9]  J. Reynolds,et al.  Interleukin-10. A role in the development of postoperative immunosuppression. , 1997 .

[10]  C. Miki,et al.  Influence of intraoperative blood loss on plasma levels of cytokines and endotoxin and subsequent graft liver function. , 1997, Archives of surgery.

[11]  E. Moore,et al.  Interleukin-6 in the injured patient. Marker of injury or mediator of inflammation? , 1996, Annals of surgery.

[12]  J. Bland,et al.  Plasma proinflammatory cytokine concentrations, Acute Physiology and Chronic Health Evaluation (APACHE) III scores and survival in patients in an intensive care unit. , 1996, Critical care medicine.

[13]  R. Bone,et al.  Sir Isaac Newton, sepsis, SIRS, and CARS. , 1996, Critical care medicine.

[14]  S. Lowry,et al.  Biologic Control of Injury and Inflammation: Much More than Too Little or Too Late , 1996, World Journal of Surgery.

[15]  P. Barie,et al.  Monocyte tumor necrosis factor receptor levels as a predictor of risk in human sepsis. , 1996, Archives of surgery.

[16]  T. van der Poll,et al.  Endogenous IL-10 protects mice from death during septic peritonitis. , 1995, Journal of immunology.

[17]  C. Sprung,et al.  Multiple organ dysfunction score: a reliable descriptor of a complex clinical outcome. , 1995, Critical care medicine.

[18]  D. Kwiatkowski,et al.  Systemic cytokine response after major surgery , 1992, The British journal of surgery.

[19]  W. Knaus,et al.  The APACHE III prognostic system. Risk prediction of hospital mortality for critically ill hospitalized adults. , 1991, Chest.

[20]  W. Fraser,et al.  Response of serum interleukin-6 in patients undergoing elective surgery of varying severity. , 1990, Clinical science.

[21]  L. Moldawer,et al.  The biologic characteristics of cytokines and their implication in surgical injury. , 1990, Surgery, gynecology & obstetrics.

[22]  T. Ikejima,et al.  Interleukin 1 induces a shock-like state in rabbits. Synergism with tumor necrosis factor and the effect of cyclooxygenase inhibition. , 1988, The Journal of clinical investigation.

[23]  J. D. Albert,et al.  Shock and tissue injury induced by recombinant human cachectin. , 1986, Science.

[24]  W. Knaus,et al.  APACHE II: a severity of disease classification system. , 1985 .

[25]  OLAND,et al.  TREATMENT OF SEPTIC SHOCK WITH THE TUMOR NECROSIS FACTOR RECEPTOR : Fc FUSION PROTEIN C , 2000 .

[26]  E. Moore,et al.  Interleukin-6 delays neutrophil apoptosis. , 1996, Archives of surgery.

[27]  H. Tilg,et al.  Interleukin-6 (IL-6) as an anti-inflammatory cytokine: induction of circulating IL-1 receptor antagonist and soluble tumor necrosis factor receptor p55. , 1994, Blood.

[28]  C. Dinarello,et al.  Correlations and interactions in the production of interleukin-6 (IL-6), IL-1, and tumor necrosis factor (TNF) in human blood mononuclear cells: IL-6 suppresses IL-1 and TNF. , 1990, Blood.