MSA prions exhibit remarkable stability and resistance to inactivation
暂无分享,去创建一个
S. Prusiner | G. Halliday | L. Middleton | Daniel A. Mordes | S. Gentleman | Abby Oehler | Smita Patel | S. Olson | A. Woerman | A. Aoyagi | S. A. Kazmi | Y. Freyman | Daniel A Mordes
[1] S. Prusiner,et al. Bioassays and Inactivation of Prions. , 2017, Cold Spring Harbor perspectives in biology.
[2] M. Pourfar,et al. Deep brain stimulation for Tourette syndrome: a single-center series. , 2017, Journal of neurosurgery.
[3] M. Okun,et al. Six-Nine Year Follow-Up of Deep Brain Stimulation for Obsessive-Compulsive Disorder , 2016, PloS one.
[4] P. McKenna,et al. Clinical Improvement in a Treatment-Resistant Patient With Schizophrenia Treated With Deep Brain Stimulation , 2016, Biological Psychiatry.
[5] H. Wille,et al. Neuroinvasion of α-Synuclein Prionoids after Intraperitoneal and Intraglossal Inoculation , 2016, Journal of Virology.
[6] H. Budka,et al. Dura mater is a potential source of Aβ seeds , 2016, Acta Neuropathologica.
[7] D. Guehl,et al. Outcome of deep brain stimulation in slowly progressive multiple system atrophy: A clinico-pathological series and review of the literature. , 2016, Parkinsonism & related disorders.
[8] A. Aguzzi,et al. Amyloid-β pathology and cerebral amyloid angiopathy are frequent in iatrogenic Creutzfeldt-Jakob disease after dural grafting. , 2016, Swiss medical weekly.
[9] B. Meier,et al. An Efficient Procedure for Removal and Inactivation of Alpha-Synuclein Assemblies from Laboratory Materials , 2015, Journal of Parkinson's disease.
[10] Sebastian Brandner,et al. Evidence for human transmission of amyloid-β pathology and cerebral amyloid angiopathy , 2015, Nature.
[11] D. Geschwind,et al. Evidence for α-synuclein prions causing multiple system atrophy in humans with parkinsonism , 2015, Proceedings of the National Academy of Sciences.
[12] T. Südhof,et al. Propagation of prions causing synucleinopathies in cultured cells , 2015, Proceedings of the National Academy of Sciences.
[13] D. Dickson,et al. When DLB, PD, and PSP masquerade as MSA , 2015, Neurology.
[14] M. Giugliano,et al. α-Synuclein strains cause distinct synucleinopathies after local and systemic administration , 2015, Nature.
[15] G. Wenning,et al. Multiple-system atrophy. , 2015, The New England journal of medicine.
[16] S. Prusiner,et al. Mechanism of Scrapie Prion Precipitation with Phosphotungstate Anions , 2014, ACS chemical biology.
[17] W. Schulz-Schaeffer,et al. Decontamination of medical devices from pathological amyloid-β-, tau- and α-synuclein aggregates , 2014, Acta neuropathologica communications.
[18] K. Nilsson,et al. Aβ seeds resist inactivation by formaldehyde , 2014, Acta Neuropathologica.
[19] Jacob I. Ayers,et al. Intramuscular injection of α-synuclein induces CNS α-synuclein pathology and a rapid-onset motor phenotype in transgenic mice , 2014, Proceedings of the National Academy of Sciences.
[20] M. Goedert,et al. Peripheral administration of tau aggregates triggers intracerebral tauopathy in transgenic mice , 2013, Acta Neuropathologica.
[21] S. Prusiner,et al. Transmission of multiple system atrophy prions to transgenic mice , 2013, Proceedings of the National Academy of Sciences.
[22] Masato Hasegawa,et al. Prion-like spreading of pathological α-synuclein in brain , 2013, Brain : a journal of neurology.
[23] M. Frye,et al. Deep brain stimulation for treatment-resistant depression: Efficacy, safety and mechanisms of action , 2012, Neuroscience & Biobehavioral Reviews.
[24] Frank Baumann,et al. Peripherally Applied Aβ-Containing Inoculates Induce Cerebral β-Amyloidosis , 2010, Science.
[25] E. Callaway,et al. Deep brain stimulation for obsessive-compulsive disorder and treatment-resistant depression: systematic review , 2010, BMC Research Notes.
[26] G. Wenning,et al. Multiple system atrophy: an update , 2009, The Lancet Neurology.
[27] A. Aguzzi,et al. Induction of cerebral β-amyloidosis: Intracerebral versus systemic Aβ inoculation , 2009, Proceedings of the National Academy of Sciences.
[28] P Sandroni,et al. Second consensus statement on the diagnosis of multiple system atrophy , 2008, Neurology.
[29] M. T. Pellecchia,et al. Progression of multiple system atrophy (MSA): A prospective natural history study by the European MSA Study Group (EMSA SG) , 2006, Movement disorders : official journal of the Movement Disorder Society.
[30] S. Prusiner,et al. Inactivation of Prions by Acidic Sodium Dodecyl Sulfate , 2006, Journal of Virology.
[31] K. Roth,et al. Infectivity of Prion Protein Bound to Stainless Steel Wires: A Model for Testing Decontamination Procedures for Transmissible Spongiform Encephalopathies , 2004, Infection Control & Hospital Epidemiology.
[32] M. Beekes,et al. Widespread PrPSc accumulation in muscles of hamsters orally infected with scrapie , 2003, EMBO reports.
[33] A. Kincaid,et al. Rapid Prion Neuroinvasion following Tongue Infection , 2003, Journal of Virology.
[34] C. Mathias,et al. Do published criteria improve clinical diagnostic accuracy in multiple system atrophy? , 2002, Neurology.
[35] J. Trojanowski,et al. Neuronal α-Synucleinopathy with Severe Movement Disorder in Mice Expressing A53T Human α-Synuclein , 2002, Neuron.
[36] Yoav Ben-Shlomo,et al. The accuracy of diagnosis of parkinsonian syndromes in a specialist movement disorder service. , 2002, Brain : a journal of neurology.
[37] J. Collinge,et al. Transmission of Scrapie by Steel-surface-bound Prions , 2001, Molecular medicine.
[38] E. Kuusisto,et al. Ubiquitin-binding protein p62 is present in neuronal and glial inclusions in human tauopathies and synucleinopathies , 2001, Neuroreport.
[39] D. Taylor. Inactivation of prions by physical and chemical means. , 1999, The Journal of hospital infection.
[40] F. Cohen,et al. Eight prion strains have PrPSc molecules with different conformations , 1998, Nature Medicine.
[41] J Q Trojanowski,et al. Glial cytoplasmic inclusions in white matter oligodendrocytes of multiple system atrophy brains contain insoluble α‐synuclein , 1998, Annals of neurology.
[42] Nigel J. Cairns,et al. Filamentous α-synuclein inclusions link multiple system atrophy with Parkinson's disease and dementia with Lewy bodies , 1998, Neuroscience Letters.
[43] Hitoshi Takahashi,et al. α-Synuclein immunoreactivity in glial cytoplasmic inclusions in multiple system atrophy , 1998, Neuroscience Letters.
[44] N. Quinn,et al. Multiple system atrophy: A review of 203 pathologically proven cases , 1997, Movement disorders : official journal of the Movement Disorder Society.
[45] D. Gajdusek,et al. Transmission of Creutzfeldt-Jakob disease to a chimpanzee by electrodes contaminated during neurosurgery. , 1994, Journal of neurology, neurosurgery, and psychiatry.
[46] Peter L. Lantos,et al. Glial cytoplasmic inclusions in the CNS of patients with multiple system atrophy (striatonigral degeneration, olivopontocerebellar atrophy and Shy-Drager syndrome) , 1989, Journal of the Neurological Sciences.
[47] S. Prusiner,et al. Linkage of prion protein and scrapie incubation time genes , 1986, Cell.
[48] R. Kimberlin,et al. Pathogenesis of scrapie (strain 263K) in hamsters infected intracerebrally, intraperitoneally or intraocularly. , 1986, The Journal of general virology.
[49] D. Gajdusek,et al. DANGER OF ACCIDENTAL PERSON-TO-PERSON TRANSMISSION OF CREUTZFELDT-JAKOB DISEASE BY SURGERY , 1977, The Lancet.
[50] Gordon Ws. Advances in veterinary research. , 1946 .
[51] Yvonne S. Eisele,et al. Formaldehyde-fixed brain tissue from spontaneously ill α-synuclein transgenic mice induces fatal α-synucleinopathy in transgenic hosts , 2014, Acta Neuropathologica.
[52] Molecular Medicine © 1999 The Picower Institute Press Infectivity of Scrapie Prions Bound to a Stainless , 1999 .