Affinity analysis of differentially expressed genes in hepatocytes expressing HCV core genotype 1b or 3a
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Charles Auffray | Manlio Vinciguerra | Tommaso Mazza | Valerio Pazienza | Stefano Castellana | Angelo Andriulli | C. Auffray | M. Vinciguerra | T. Mazza | S. Castellana | A. Andriulli | V. Pazienza
[1] X. Causse,et al. Comparison of 1 or 3 MU of interferon alfa-2b and placebo in patients with chronic non-A, non-B hepatitis. , 1991, Gastroenterology.
[2] O. Weiland,et al. Serum HCV RNA levels in patients with chronic hepatitis C given a second course of interferon alpha-2b treatment after relapse following initial treatment. , 1993, Scandinavian journal of infectious diseases.
[3] C. Rice,et al. Expression and identification of hepatitis C virus polyprotein cleavage products , 1993, Journal of virology.
[4] J. Pawlotsky,et al. Relationship between hepatitis C virus genotypes and sources of infection in patients with chronic hepatitis C. , 1995, The Journal of infectious diseases.
[5] K. Meyer,et al. Suppression of apoptotic cell death by hepatitis C virus core protein. , 1996, Virology.
[6] K Koike,et al. Hepatitis C virus core protein induces hepatic steatosis in transgenic mice. , 1997, The Journal of general virology.
[7] Dumoulin,et al. Hepatitis C virus core protein does not inhibit apoptosis in human hepatoma cells , 1999, European journal of clinical investigation.
[8] M. Honda,et al. Hepatitis C virus core protein induces apoptosis and impairs cell‐cycle regulation in stably transformed chinese hamster ovary cells , 2000, Hepatology.
[9] Huang Yuan-zhu. Preliminary analysis of gene expression profiles in human myocardium of atrial septal defect by cDNA microarray , 2003 .
[10] M. Miyazaki,et al. Stearoyl-CoA Desaturase 1 Gene Expression Is Necessary for Fructose-mediated Induction of Lipogenic Gene Expression by Sterol Regulatory Element-binding Protein-1c-dependent and -independent Mechanisms* , 2004, Journal of Biological Chemistry.
[11] T. Kawaguchi,et al. Hepatitis C virus down-regulates insulin receptor substrates 1 and 2 through up-regulation of suppressor of cytokine signaling 3. , 2004, The American journal of pathology.
[12] V. Pazienza,et al. An in vitro model of hepatitis C virus genotype 3a-associated triglycerides accumulation. , 2005, Journal of hepatology.
[13] Colin W Shepard,et al. Global epidemiology of hepatitis C virus infection. , 2005, The Lancet. Infectious diseases.
[14] D. Lavillette,et al. Characterization of host‐range and cell entry properties of the major genotypes and subtypes of hepatitis C virus , 2005, Hepatology.
[15] Alan Bundy,et al. Constructing Induction Rules for Deductive Synthesis Proofs , 2006, CLASE.
[16] John S. Satterlee,et al. An ARC/Mediator subunit required for SREBP control of cholesterol and lipid homeostasis , 2006, Nature.
[17] J. Dou,et al. Preliminary analysis of gene expression profiles in HepG2 cell line induced by different genotype core proteins of HCV. , 2006, Cellular & molecular immunology.
[18] Sutapa Banerjee,et al. Hepatitis C Virus Core Protein Upregulates Serine Phosphorylation of Insulin Receptor Substrate-1 and Impairs the Downstream Akt/Protein Kinase B Signaling Pathway for Insulin Resistance , 2007, Journal of Virology.
[19] L. Guarente,et al. SIRT1 deacetylates and positively regulates the nuclear receptor LXR. , 2007, Molecular cell.
[20] B. Giraudeau,et al. The genotype 3-specific hepatitis C virus core protein residue phenylalanine 164 increases steatosis in an in vitro cellular model , 2007, Gut.
[21] Min Jung Park,et al. HCV core protein induces hepatic lipid accumulation by activating SREBP1 and PPARgamma. , 2007, Biochemical and biophysical research communications.
[22] V. Pazienza,et al. The hepatitis C virus core protein of genotypes 3a and 1b downregulates insulin receptor substrate 1 through genotype‐specific mechanisms , 2007, Hepatology.
[23] Y. Karino,et al. Treatment of chronic non-A, non-B hepatitis with interferon , 1991, Gastroenterologia Japonica.
[24] David Warde-Farley,et al. GeneMANIA: a real-time multiple association network integration algorithm for predicting gene function , 2008, Genome Biology.
[25] Brad T. Sherman,et al. Systematic and integrative analysis of large gene lists using DAVID bioinformatics resources , 2008, Nature Protocols.
[26] P. Pfluger,et al. Sirt1 protects against high-fat diet-induced metabolic damage , 2008, Proceedings of the National Academy of Sciences.
[27] V. Pazienza,et al. Gene expression profile of Huh‐7 cells expressing hepatitis C virus genotype 1b or 3a core proteins , 2009, Liver international : official journal of the International Association for the Study of the Liver.
[28] Yves Moreau,et al. Integrating Computational Biology and Forward Genetics in Drosophila , 2009, PLoS genetics.
[29] L. Hood,et al. Systems medicine: the future of medical genomics and healthcare , 2009, Genome Medicine.
[30] Paolo Ballarini,et al. Studying Irreversible Transitions in a Model of Cell Cycle Regulation , 2009, PASM@EPEW.
[31] Qing Xu,et al. Hepatocyte-specific deletion of SIRT1 alters fatty acid metabolism and results in hepatic steatosis and inflammation. , 2009, Cell metabolism.
[32] Jing Chen,et al. ToppGene Suite for gene list enrichment analysis and candidate gene prioritization , 2009, Nucleic Acids Res..
[33] Xiaoling Xu,et al. Hepatic-specific disruption of SIRT6 in mice results in fatty liver formation due to enhanced glycolysis and triglyceride synthesis. , 2010, Cell metabolism.
[34] T. Shioda,et al. MicroRNA-33 and the SREBP Host Genes Cooperate to Control Cholesterol Homeostasis , 2010, Science.
[35] I. Gérin,et al. Expression of miR-33 from an SREBP2 Intron Inhibits Cholesterol Export and Fatty Acid Oxidation* , 2010, The Journal of Biological Chemistry.
[36] Ferenc Jordán,et al. Estimating the divisibility of complex biological networks by sparseness indices , 2010, Briefings Bioinform..
[37] T. Veenstra,et al. SIRT1 Deacetylates and Inhibits SREBP-1C Activity in Regulation of Hepatic Lipid Metabolism* , 2010, The Journal of Biological Chemistry.
[38] K. Moore,et al. miR-33a/b contribute to the regulation of fatty acid metabolism and insulin signaling , 2011, Proceedings of the National Academy of Sciences.
[39] V. Pazienza,et al. Impact of HCV genetic differences on pathobiology of disease , 2011, Expert review of anti-infective therapy.
[40] Teresa Balsamo,et al. Hypermethylated levels of E-cadherin promoter in Huh-7 cells expressing the HCV core protein. , 2011, Virus research.
[41] Tommaso Mazza,et al. Mirna Expression Profiles Identify Drivers in Colorectal and Pancreatic Cancers , 2012, PloS one.
[42] Rosita Guido,et al. The Relevance of Topology in Parallel Simulation of Biological Networks , 2012, IEEE/ACM Transactions on Computational Biology and Bioinformatics.
[43] M. Vinciguerra,et al. DNA Methyltransferases 1 and 3b Expression in Huh-7 Cells Expressing HCV Core Protein of Different Genotypes , 2012, Digestive Diseases and Sciences.
[44] Angela Re,et al. AURA: Atlas of UTR Regulatory Activity , 2012, Bioinform..
[45] M. Vinciguerra,et al. Redox homeostasis and epigenetics in non-alcoholic fatty liver disease (NAFLD). , 2013, Current pharmaceutical design.