Influence of plasma aldosterone on left ventricular geometry and diastolic function in treated essential hypertension.
暂无分享,去创建一个
[1] B. Pitt. “Escape” of aldosterone production in patients with left ventricular dysfunction treated with an angiotensin converting enzyme inhibitor: Implications for therapy , 1995, Cardiovascular Drugs and Therapy.
[2] T. Saruta,et al. Aldosterone Escape during Angiotensinconverting Enzyme Inhibitor Therapy in Essential Hypertensive Patients with Left Ventricular Hypertrophy , 2001, The Journal of international medical research.
[3] K. Arakawa,et al. The regression of left ventricular hypertrophy by imidapril and the reduction of serum procollagen type III amino-terminal peptide in hypertensive patients. , 2000, Hypertension research : official journal of the Japanese Society of Hypertension.
[4] B. Pitt,et al. The Effect of Spironolactone on Morbidity and Mortality in Patients with Severe Heart Failure , 2000 .
[5] C. Brilla. Aldosterone and Myocardial Fibrosis in Heart Failure , 2000, Herz.
[6] T. Saruta,et al. Plasma aldosterone concentrations are not related to the degree of angiotensin-converting enzyme inhibition in essential hypertensive patients. , 2000, Hypertension research : official journal of the Japanese Society of Hypertension.
[7] B. Pitt,et al. The effect of spironolactone on morbidity and mortality in patients with severe heart failure. Randomized Aldactone Evaluation Study Investigators. , 1999, The New England journal of medicine.
[8] T. Saruta,et al. Effects of spironolactone and angiotensin-converting enzyme inhibitor on left ventricular hypertrophy in patients with essential hypertension. , 1999, Hypertension research : official journal of the Japanese Society of Hypertension.
[9] R. Fagard,et al. Opposite associations of circulating aldosterone and atrial natriuretic peptide with left ventricular diastolic function in essential hypertension , 1998, Journal of Human Hypertension.
[10] H. Schunkert,et al. Neurohormonal activity and left ventricular geometry in patients with essential arterial hypertension. , 1998, American heart journal.
[11] P. Palatini,et al. Remodeling of the left ventricle in primary aldosteronism due to Conn's adenoma. , 1997, Circulation.
[12] T. Nishikimi,et al. Left ventricular systolic and diastolic function and mass before and after antihypertensive treatment in patients with essential hypertension. , 1997, Hypertension research : official journal of the Japanese Society of Hypertension.
[13] L. Lind,et al. Left ventricular hypertrophy and geometry in a population sample of elderly males. , 1996, European heart journal.
[14] R. Schmieder,et al. Angiotensin II related to sodium excretion modulates left ventricular structure in human essential hypertension. , 1996, Circulation.
[15] Randolph P. Martin,et al. Canadian consensus recommendations for the measurement and reporting of diastolic dysfunction by echocardiography: from the Investigators of Consensus on Diastolic Dysfunction by Echocardiography. , 1996, Journal of the American Society of Echocardiography : official publication of the American Society of Echocardiography.
[16] T. Nishikimi,et al. Comparison of changes in cardiac structure after treatment in secondary hypertension. , 1996, Hypertension.
[17] K. Thygesen,et al. Influence of humoral and neurohormonal factors on cardiovascular hypertrophy in untreated essential hypertensives. , 1996, American journal of hypertension.
[18] C. Delcayre,et al. Biological determinants of aldosterone-induced cardiac fibrosis in rats. , 1995, Hypertension.
[19] D. Levy,et al. Prognosis of left ventricular geometric patterns in the Framingham Heart Study. , 1995, Journal of the American College of Cardiology.
[20] H. Matsuoka,et al. Clinical evidence for an association between left ventricular geometric adaptation and extracardiac target organ damage in essential hypertension , 1995, Journal of hypertension.
[21] T. Ogihara,et al. Association between a deletion polymorphism of the angiotensin-converting-enzyme gene and left ventricular hypertrophy. , 1994, The New England journal of medicine.
[22] C. Delcayre,et al. Increased cardiac types I and III collagen mRNAs in aldosterone-salt hypertension. , 1994, Hypertension.
[23] R. Dilley,et al. Mineralocorticoids, hypertension, and cardiac fibrosis. , 1994, The Journal of clinical investigation.
[24] J. Parameshwar,et al. The role of calcium antagonists in the treatment of chronic heart failure. , 1993, European heart journal.
[25] A. de la Sierra,et al. Left ventricular hypertrophy in asymptomatic essential hypertension: its relationship with aldosterone and the increase in sodium-proton exchanger activity. , 1993, European heart journal.
[26] J. Laragh,et al. Patterns of left ventricular hypertrophy and geometric remodeling in essential hypertension. , 1992, Journal of the American College of Cardiology.
[27] J. Kaufman,et al. Influence of the arterial blood pressure and nonhemodynamic factors on left ventricular hypertrophy in moderate essential hypertension. , 1991, The American journal of cardiology.
[28] J S Janicki,et al. Impaired diastolic function and coronary reserve in genetic hypertension. Role of interstitial fibrosis and medial thickening of intramyocardial coronary arteries. , 1991, Circulation research.
[29] K. Weber,et al. Pathological Hypertrophy and Cardiac Interstitium: Fibrosis and Renin‐Angiotensin‐Aldosterone System , 1991, Circulation.
[30] K Lindpaintner,et al. The cardiac renin-angiotensin system. An appraisal of present experimental and clinical evidence. , 1991, Circulation research.
[31] J. Laragh,et al. Relation of left ventricular mass and geometry to morbidity and mortality in uncomplicated essential hypertension. , 1991, Annals of internal medicine.
[32] A. Labovitz,et al. Echocardiographic evaluation of cardiac structure and function in elderly subjects with isolated systolic hypertension. , 1991, Journal of the American College of Cardiology.
[33] K. Baker,et al. Cardiac Hypertrophy: Mechanical, Neural, and Endocrine Dependence , 1991 .
[34] D. Levy,et al. Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study. , 1990, The New England journal of medicine.
[35] P. Granier,et al. Correlations between M-mode markers of left ventricular hypertrophy and radionuclide angiographic indices of left ventricular diastolic function in mild to moderate hypertension. , 1989, Journal of hypertension. Supplement : official journal of the International Society of Hypertension.
[36] D. Bonaduce,et al. Myocardial hypertrophy and left ventricular diastolic function in hypertensive patients: an echo Doppler evaluation. , 1989, European heart journal.
[37] M. Kuramochi,et al. Left ventricular structural characteristics in unilateral renovascular hypertension and primary aldosteronism. , 1988, The American journal of cardiology.
[38] R. Schmieder,et al. Does the renin-angiotensin-aldosterone system modify cardiac structure and function in essential hypertension? , 1988, The American journal of medicine.
[39] W. White,et al. Echocardiographic Assessment of Left Ventricular Diastolic Performance in Hypertensive Subjects Correlation with Changes in Left Ventricular Mass , 1987, Hypertension.
[40] R. Devereux. Detection of Left Ventricular Hypertrophy by M‐Mode Echocardiography: Anatomic Validation, Standardization, and Comparison to Other Methods , 1987, Hypertension.
[41] R. Tarazi,et al. Left ventricular diastolic function in hypertension: relation to left ventricular mass and systolic function. , 1984, Journal of the American College of Cardiology.
[42] B. Massie,et al. Abnormal left ventricular filling: an early finding in mild to moderate systemic hypertension. , 1984, The American journal of cardiology.
[43] J. Staessen,et al. Increase in plasma aldosterone during prolonged captopril treatment. , 1982, The American journal of cardiology.
[44] R. Tarazi,et al. The multifactorial role of catecholamines in hypertensive cardiac hypertrophy. , 1982, European heart journal.
[45] J. Staessen,et al. Rise in plasma concentration of aldosterone during long-term angiotensin II suppression. , 1981, The Journal of endocrinology.
[46] N Reichek,et al. Echocardiographic Determination of Left Ventricular Mass in Man: Anatomic Validation of the Method , 1977, Circulation.