Meta‐analysis: the safety and efficacy of vaptans (tolvaptan, satavaptan and lixivaptan) in cirrhosis with ascites or hyponatraemia
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[1] F. Czerwiec,et al. Tolvaptan, an oral vasopressin antagonist, in the treatment of hyponatremia in cirrhosis. , 2012, Journal of hepatology.
[2] A. Gerbes,et al. Satavaptan for the management of ascites in cirrhosis: efficacy and safety across the spectrum of ascites severity , 2011, Gut.
[3] V. Arroyo,et al. Risk factors for hepatic encephalopathy in patients with cirrhosis and refractory ascites: relevance of serum sodium concentration , 2010, Liver international : official journal of the International Association for the Study of the Liver.
[4] P. Gow,et al. Effects of a selective vasopressin V2 receptor antagonist, satavaptan, on ascites recurrence after paracentesis in patients with cirrhosis. , 2010, Journal of hepatology.
[5] T. Berl,et al. Oral tolvaptan is safe and effective in chronic hyponatremia. , 2010, Journal of the American Society of Nephrology : JASN.
[6] R. Brůha,et al. Clinical trial: short‐term effects of combination of satavaptan, a selective vasopressin V2 receptor antagonist, and diuretics on ascites in patients with cirrhosis without hyponatraemia – a randomized, double‐blind, placebo‐controlled study , 2010, Alimentary pharmacology & therapeutics.
[7] A. Gerbes,et al. 857 SATAVAPTAN EFFICACY AND SAFETY IN RECURRENT ASCITES WITHOUT CONCOMITANT DIURETICS: THE SPARE-2 STUDY , 2010 .
[8] F. Nevens,et al. Double-blind, placebo-controlled study of satavaptan in the management of recurrent ascites: the spare-1 study , 2009 .
[9] B. Gómez-Anson,et al. Hyponatremia Is a Risk Factor of Hepatic Encephalopathy in Patients With Cirrhosis: A Prospective Study With Time-Dependent Analysis , 2009, The American Journal of Gastroenterology.
[10] F. Wong,et al. 102 EFFECTS OF SATAVAPTAN, AN ORAL VASOPRESSIN V2 RECEPTOR ANTAGONIST, ON MANAGEMENT OF ASCITES AND MORBIDITY IN LIVER CIRRHOSIS IN A LONG-TERM, PLACEBO-CONTROLLED STUDY , 2009 .
[11] F. Wong,et al. Effects of satavaptan, a selective vasopressin V2 receptor antagonist, on ascites and serum sodium in cirrhosis with hyponatremia: A randomized trial , 2008, Hepatology.
[12] Douglas G Altman,et al. Empirical evidence of bias in treatment effect estimates in controlled trials with different interventions and outcomes: meta-epidemiological study , 2008, BMJ : British Medical Journal.
[13] F. Wong,et al. Transjugular intrahepatic portosystemic shunt for refractory ascites: a meta-analysis of individual patient data. , 2007, Gastroenterology.
[14] F. Wong,et al. [90] LONG-TERM IMPROVEMENT OF SERUM SODIUM BY THE V-RECEPTOR ANTAGONIST SATAVAPTAN IN PATIENTS WITH CIRRHOSIS AND HYPONATRAEMIA , 2007 .
[15] T. Berl,et al. Tolvaptan, a selective oral vasopressin V2-receptor antagonist, for hyponatremia. , 2006, The New England journal of medicine.
[16] F. Wong,et al. 732 Effects of satavaptan (SR121463B), a selective vasopressin V2 receptor antagonist, on serum sodium concentration and ascites in patients with cirrhosis and hyponatraemia , 2006 .
[17] A. Gerbes,et al. Therapy of hyponatremia in cirrhosis with a vasopressin receptor antagonist: a randomized double-blind multicenter trial. , 2003, Gastroenterology.
[18] P. Thuluvath,et al. A vasopressin receptor antagonist (VPA‐985) improves serum sodium concentration in patients with hyponatremia: A multicenter, randomized, placebo‐controlled trial , 2003, Hepatology.
[19] B D Ross,et al. Human cerebral osmolytes during chronic hyponatremia. A proton magnetic resonance spectroscopy study. , 1995, The Journal of clinical investigation.
[20] Vicente Arroyo,et al. Peripheral arterial vasodilation hypothesis: A proposal for the initiation of renal sodium and water retention in cirrhosis , 1988, Hepatology.
[21] G. D’Amico,et al. Survival and prognostic indicators in compensated and decompensated cirrhosis , 1986, Digestive Diseases and Sciences.
[22] V. Arroyo,et al. Compensated cirrhosis: Natural history and prognostic factors , 1987, Hepatology.