WIP Regulates the Stability and Localization of WASP to Podosomes in Migrating Dendritic Cells

[1]  S. Simon,et al.  Impaired integrin-dependent function in Wiskott-Aldrich syndrome protein-deficient murine and human neutrophils. , 2006, Immunity.

[2]  J. Cooper,et al.  Cortactin has an essential and specific role in osteoclast actin assembly. , 2006, Molecular Biology of the Cell.

[3]  R. Liddington,et al.  Role of vinculin in regulating focal adhesion turnover. , 2006, European journal of cell biology.

[4]  N. Carragher,et al.  Inhibition of calpain stabilises podosomes and impairs dendritic cell motility , 2006, Journal of Cell Science.

[5]  S. Tsuboi,et al.  A Complex of Wiskott-Aldrich Syndrome Protein with Mammalian Verprolins Plays an Important Role in Monocyte Chemotaxis1 , 2006, The Journal of Immunology.

[6]  Gareth E. Jones,et al.  The leukocyte podosome. , 2006, European Journal of Cell Biology.

[7]  Stephen D. Smith,et al.  CSF-1 and PI 3-kinase regulate podosome distribution and assembly in macrophages. , 2006, Cell motility and the cytoskeleton.

[8]  R. Geha,et al.  WIP and WASP play complementary roles in T cell homing and chemotaxis to SDF-1alpha. , 2006, International immunology.

[9]  K. J. Amann,et al.  Proteolysis of cortactin by calpain regulates membrane protrusion during cell migration. , 2005, Molecular biology of the cell.

[10]  S. Linder,et al.  Podosomes at a glance , 2005, Journal of Cell Science.

[11]  J. Segall,et al.  A Neural Wiskott-Aldrich Syndrome Protein-mediated Pathway for Localized Activation of Actin Polymerization That Is Regulated by Cortactin* , 2005, Journal of Biological Chemistry.

[12]  W. Vainchenker,et al.  Mechanisms of WASp-mediated hematologic and immunologic disease. , 2004, Blood.

[13]  H. Chou,et al.  Wiskott–Aldrich syndrome protein and the cytoskeletal dynamics of dendritic cells , 2004, The Journal of pathology.

[14]  C. Yoon,et al.  Two novel mutations of Wiskott-Aldrich syndrome: the molecular prediction of interaction between the mutated WASP L101P with WASP-interacting protein by molecular modeling. , 2004, Biochimica et biophysica acta.

[15]  Gareth E. Jones,et al.  Cell motility under the microscope: Vorsprung durch Technik , 2004, Nature Reviews Molecular Cell Biology.

[16]  S. Gygi,et al.  Toca-1 Mediates Cdc42-Dependent Actin Nucleation by Activating the N-WASP-WIP Complex , 2004, Cell.

[17]  J. Hartwig,et al.  WIP Regulates Signaling via the High Affinity Receptor for Immunoglobulin E in Mast Cells , 2004, The Journal of experimental medicine.

[18]  Samantha J. Hardy,et al.  Maturation of DC is associated with changes in motile characteristics and adherence. , 2004, Cell motility and the cytoskeleton.

[19]  D. Webb,et al.  Illuminating adhesion complexes in migrating cells: moving toward a bright future. , 2003, Current opinion in cell biology.

[20]  E. Génot,et al.  Actin Can Reorganize into Podosomes in Aortic Endothelial Cells, a Process Controlled by Cdc42 and RhoA , 2003, Molecular and Cellular Biology.

[21]  L. Blanchoin,et al.  Phosphorylation of the WASP-VCA domain increases its affinity for the Arp2/3 complex and enhances actin polymerization by WASP. , 2003, Molecular cell.

[22]  Alissa M. Weaver,et al.  Cortactin Interacts with WIP in Regulating Arp2/3 Activation and Membrane Protrusion , 2003, Current Biology.

[23]  Alissa M. Weaver,et al.  Integration of signals to the Arp2/3 complex. , 2003, Current opinion in cell biology.

[24]  M. Gandhi,et al.  X-linked thrombocytopenia caused by a mutation in the Wiskott-Aldrich syndrome (WAS) gene that disrupts interaction with the WAS protein (WASP)-interacting protein (WIP). , 2003, Experimental hematology.

[25]  A. Ridley,et al.  Phosphorylation of Tyrosine 291 Enhances the Ability of WASp to Stimulate Actin Polymerization and Filopodium Formation* , 2002, The Journal of Biological Chemistry.

[26]  W. Lim,et al.  Structure of the N-WASP EVH1 Domain-WIP Complex Insight into the Molecular Basis of Wiskott-Aldrich Syndrome , 2002, Cell.

[27]  U. V. von Andrian,et al.  Travellers in many guises: The origins and destinations of dendritic cells , 2002, Immunology and cell biology.

[28]  Adrian J. Thrasher,et al.  Wasp in immune-system organization and function , 2002, Nature Reviews Immunology.

[29]  A. Bershadsky Faculty Opinions recommendation of Phosphatidylinositol 4,5-biphosphate (PIP2)-induced vesicle movement depends on N-WASP and involves Nck, WIP, and Grb2. , 2002 .

[30]  Alissa M. Weaver,et al.  Interaction of Cortactin and N-WASp with Arp2/3 Complex , 2002, Current Biology.

[31]  R. Annan,et al.  Identification of Novel SH3 Domain Ligands for the Src Family Kinase Hck , 2002, The Journal of Biological Chemistry.

[32]  J. Hartwig,et al.  WIP deficiency reveals a differential role for WIP and the actin cytoskeleton in T and B cell activation. , 2002, Immunity.

[33]  T. Takenawa,et al.  WASP and N-WASP in human platelets differ in sensitivity to protease calpain. , 2001, Blood.

[34]  S. Weed,et al.  Cortactin: coupling membrane dynamics to cortical actin assembly , 2001, Oncogene.

[35]  G. E. Jones,et al.  Configuration of human dendritic cell cytoskeleton by Rho GTPases, the WAS protein, and differentiation. , 2001, Blood.

[36]  V. Noireaux,et al.  ActA and human zyxin harbour Arp2/3-independent actin-polymerization activity , 2001, Nature Cell Biology.

[37]  J. Hartwig,et al.  WIP regulates N-WASP-mediated actin polymerization and filopodium formation , 2001, Nature Cell Biology.

[38]  H. Ochs,et al.  CrkL is an adapter for Wiskott-Aldrich syndrome protein and Syk. , 2001, Blood.

[39]  F. Frischknecht,et al.  A complex of N-WASP and WIP integrates signalling cascades that lead to actin polymerization , 2000, Nature Cell Biology.

[40]  D. Nelson,et al.  Mutations that cause the Wiskott-Aldrich syndrome impair the interaction of Wiskott-Aldrich syndrome protein (WASP) with WASP interacting protein. , 1999, Journal of immunology.

[41]  Philip R. Cohen,et al.  Wiskott-Aldrich syndrome protein-deficient mice reveal a role for WASP in T but not B cell activation. , 1998, Immunity.

[42]  Luigi Naldini,et al.  Multiply attenuated lentiviral vector achieves efficient gene delivery in vivo , 1997, Nature Biotechnology.

[43]  S. Lin,et al.  Differentiation dependent expression of tensin and cortactin in chicken osteoclasts. , 1995, Cell motility and the cytoskeleton.