Heat Shock Proteins and Whole Body Physiology

[1]  C. Apovian,et al.  Obesity prevalence from a European perspective: a systematic review , 2008, BMC public health.

[2]  Jason Chung,et al.  HSP72 protects against obesity-induced insulin resistance , 2008, Proceedings of the National Academy of Sciences.

[3]  Y. B. Lee,et al.  Hsp70 suppresses apoptosis in sympathetic neurones by preventing the activation of c‐Jun , 2007, Journal of neurochemistry.

[4]  G. Hotamisligil,et al.  Inflammation and metabolic disorders , 2006, Nature.

[5]  E. Lander,et al.  Reactive oxygen species have a causal role in multiple forms of insulin resistance , 2006, Nature.

[6]  Jean-Daniel Zucker,et al.  Reduction of macrophage infiltration and chemoattractant gene expression changes in white adipose tissue of morbidly obese subjects after surgery-induced weight loss. , 2005, Diabetes.

[7]  Sang‐Gun Ahn,et al.  Polo-like Kinase 1 Phosphorylates Heat Shock Transcription Factor 1 and Mediates Its Nuclear Translocation during Heat Stress* , 2005, Journal of Biological Chemistry.

[8]  Michael Karin,et al.  Reactive Oxygen Species Promote TNFα-Induced Death and Sustained JNK Activation by Inhibiting MAP Kinase Phosphatases , 2005, Cell.

[9]  J. Hawley Exercise as a therapeutic intervention for the prevention and treatment of insulin resistance , 2004, Diabetes/metabolism research and reviews.

[10]  D. Laaksonen,et al.  Exercise training modulates heat shock protein response in diabetic rats. , 2004, Journal of applied physiology.

[11]  A. Saltiel,et al.  Insulin Signaling and the Regulation of Glucose Transport , 2004, Molecular medicine.

[12]  M. Febbraio,et al.  Intramuscular heat shock protein 72 and heme oxygenase-1 mRNA are reduced in patients with type 2 diabetes: evidence that insulin resistance is associated with a disturbed antioxidant defense mechanism. , 2003, Diabetes.

[13]  András Fiser,et al.  Bimoclomol, a heat shock protein co-inducer, acts by the prolonged activation of heat shock factor-1. , 2003, Biochemical and biophysical research communications.

[14]  I. Cohen,et al.  T cells and autoantibodies to human HSP70 in type 1 diabetes in children. , 2003, Journal of autoimmunity.

[15]  A. Burnett,et al.  Liposomal Delivery of Heat Shock Protein 72 Into Renal Tubular Cells Blocks Nuclear Factor-&kgr;B Activation, Tumor Necrosis Factor-&agr; Production, and Subsequent Ischemia-Induced Apoptosis , 2003, Circulation research.

[16]  Michael Karin,et al.  A central role for JNK in obesity and insulin resistance , 2002, Nature.

[17]  K. Flegal,et al.  Prevalence and trends in obesity among US adults, 1999-2000. , 2002, JAMA.

[18]  K. Eriksson,et al.  Decreased expression of heat shock protein 72 in skeletal muscle of patients with type 2 diabetes correlates with insulin resistance. , 2002, Diabetes.

[19]  M. White,et al.  Phosphorylation of Ser307 in Insulin Receptor Substrate-1 Blocks Interactions with the Insulin Receptor and Inhibits Insulin Action* , 2002, The Journal of Biological Chemistry.

[20]  Bernstein Rk Hot-tub therapy for type 2 diabetes mellitus. , 2000 .

[21]  S. C. Chen,et al.  Previous hyperthermic treatment increases mitochondria oxidative enzyme activity and exercise capacity in rats. , 1999, The Kaohsiung journal of medical sciences.

[22]  H Atlan,et al.  T cell proliferative responses of type 1 diabetes patients and healthy individuals to human hsp60 and its peptides. , 1999, Journal of autoimmunity.

[23]  N. Mivechi,et al.  Glycogen Synthase Kinase 3β and Extracellular Signal-Regulated Kinase Inactivate Heat Shock Transcription Factor 1 by Facilitating the Disappearance of Transcriptionally Active Granules after Heat Shock , 1998, Molecular and Cellular Biology.

[24]  Dick D. Mosser,et al.  Hsp70 Prevents Activation of Stress Kinases , 1997, The Journal of Biological Chemistry.

[25]  H. Kolb,et al.  Low Stress Response Enhances Vulnerability of Islet Cells in Diabetes-Prone BB Rats , 1997, Diabetes.

[26]  Brendan D. Price,et al.  Sequential Phosphorylation by Mitogen-activated Protein Kinase and Glycogen Synthase Kinase 3 Represses Transcriptional Activation by Heat Shock Factor-1* , 1996, The Journal of Biological Chemistry.

[27]  I. Cohen,et al.  NOD mouse diabetes: the ubiquitous mouse hsp60 is a beta-cell target antigen of autoimmune T cells. , 1996, Journal of autoimmunity.

[28]  B. Spiegelman,et al.  IRS-1-Mediated Inhibition of Insulin Receptor Tyrosine Kinase Activity in TNF-α- and Obesity-Induced Insulin Resistance , 1996, Science.

[29]  J. Bach,et al.  Insulin-dependent diabetes mellitus as an autoimmune disease. , 1994, Endocrine reviews.

[30]  I. Cohen,et al.  Autoimmune Diabetes Induced by the β-cell Toxin STZ: Immunity to the 60-kDa Heat Shock Protein and to Insulin , 1994, Diabetes.

[31]  R. Morimoto,et al.  Effect of sodium salicylate on the human heat shock response. , 1992, Science.

[32]  I. Cohen,et al.  Vaccination against autoimmune mouse diabetes with a T-cell epitope of the human 65-kDa heat shock protein. , 1991, Proceedings of the National Academy of Sciences of the United States of America.

[33]  I. Cohen,et al.  Induction and therapy of autoimmune diabetes in the non-obese diabetic (NOD/Lt) mouse by a 65-kDa heat shock protein. , 1990, Proceedings of the National Academy of Sciences of the United States of America.

[34]  J. Amin,et al.  Key features of heat shock regulatory elements , 1988, Molecular and cellular biology.

[35]  N. Marceau,et al.  Synthesis and degradation of heat shock proteins during development and decay of thermotolerance. , 1982, Cancer research.

[36]  Bin Chen,et al.  Guidelines for the nomenclature of the human heat shock proteins , 2008, Cell Stress and Chaperones.

[37]  S. Shoelson,et al.  Local and systemic insulin resistance resulting from hepatic activation of IKK-beta and NF-kappaB. , 2005, Nature medicine.

[38]  Ping H Wang,et al.  Insulin deficiency downregulated heat shock protein 60 and IGF-1 receptor signaling in diabetic myocardium. , 2005, Diabetes.

[39]  I. Chang,et al.  Death effectors of beta-cell apoptosis in type 1 diabetes. , 2004, Molecular genetics and metabolism.

[40]  B. Spiegelman,et al.  Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance. , 1993, Science.