Identifying advanced glycation end products as a major source of oxidants in aging: implications for the management and/or prevention of reduced renal function in elderly persons.
暂无分享,去创建一个
L. Ferrucci | G. Striker | J. Post | J. Uribarri | H. Vlassara | M. Torreggiani | F. Zheng | W. Cai
[1] R. Foley,et al. Long-term consequences of kidney donation. , 2009, The New England journal of medicine.
[2] L. Ferrucci,et al. Protection against loss of innate defenses in adulthood by low advanced glycation end products (AGE) intake: role of the antiinflammatory AGE receptor-1. , 2009, The Journal of clinical endocrinology and metabolism.
[3] W. Weimar,et al. Long-term consequences of kidney donation. , 2009, The New England journal of medicine.
[4] S. Bandinelli,et al. Predictivity of survival according to different equations for estimating renal function in community-dwelling elderly subjects. , 2008, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.
[5] G. Striker,et al. Oral glycotoxins determine the effects of calorie restriction on oxidant stress, age-related diseases, and lifespan. , 2008, The American journal of pathology.
[6] A. Go,et al. The risk of acute renal failure in patients with chronic kidney disease. , 2008, Kidney international.
[7] L. Ferrucci,et al. High basal metabolic rate is a risk factor for mortality: the Baltimore Longitudinal Study of Aging. , 2008, The journals of gerontology. Series A, Biological sciences and medical sciences.
[8] J. Uribarri,et al. Endothelial dysfunction in patients with chronic kidney disease results from advanced glycation end products (AGE)-mediated inhibition of endothelial nitric oxide synthase through RAGE activation. , 2008, Clinical journal of the American Society of Nephrology : CJASN.
[9] G. Striker,et al. Glycotoxins in the diet promote diabetes and diabetic complications , 2007, Current diabetes reports.
[10] G. Striker,et al. Reduced oxidant stress and extended lifespan in mice exposed to a low glycotoxin diet: association with increased AGER1 expression. , 2007, The American journal of pathology.
[11] L. Ferrucci,et al. Circulating glycotoxins and dietary advanced glycation endproducts: two links to inflammatory response, oxidative stress, and aging. , 2007, The journals of gerontology. Series A, Biological sciences and medical sciences.
[12] G. Striker,et al. Combined AGE inhibition and ACEi decreases the progression of established diabetic nephropathy in B6 db/db mice. , 2006, Kidney International.
[13] G. Striker,et al. Differential effects of continuous and intermittent 17beta-estradiol replacement and tamoxifen therapy on the prevention of glomerulosclerosis: modulation of the mesangial cell phenotype in vivo. , 2006, The American journal of pathology.
[14] Z. Varghese,et al. PPARα agonist fenofibrate improves diabetic nephropathy in db/db mice. Commentary , 2006 .
[15] Anne B. Newman,et al. Chronic Kidney Disease and Functional Limitation in Older People: Health, Aging and Body Composition Study , 2006, Journal of the American Geriatrics Society.
[16] G. Striker,et al. PPARalpha agonist fenofibrate improves diabetic nephropathy in db/db mice. , 2006, Kidney international.
[17] S. Genuth,et al. Glycation and carboxymethyllysine levels in skin collagen predict the risk of future 10-year progression of diabetic retinopathy and nephropathy in the diabetes control and complications trial and epidemiology of diabetes interventions and complications participants with type 1 diabetes. , 2005, Diabetes.
[18] David E. Misek,et al. Podocyte hypertrophy, "adaptation," and "decompensation" associated with glomerular enlargement and glomerulosclerosis in the aging rat: prevention by calorie restriction. , 2005, Journal of the American Society of Nephrology : JASN.
[19] G. Striker,et al. Glomerular aging in females is a multi-stage reversible process mediated by phenotypic changes in progenitors. , 2005, The American journal of pathology.
[20] H. Vlassara. Advanced Glycation in Health and Disease: Role of the Modern Environment , 2005, Annals of the New York Academy of Sciences.
[21] B. Hudson,et al. Pyridoxamine as a multifunctional pharmaceutical: targeting pathogenic glycation and oxidative damage , 2005, Cellular and Molecular Life Sciences CMLS.
[22] M. Laakso,et al. High Serum Levels of Advanced Glycation End Products Predict Increased Coronary Heart Disease Mortality in Nondiabetic Women but not in Nondiabetic Men: A Population-Based 18-Year Follow-Up Study , 2005, Arteriosclerosis, thrombosis, and vascular biology.
[23] A. Murray,et al. Chronic kidney disease and the risk for cardiovascular disease, renal replacement, and death in the United States Medicare population, 1998 to 1999. , 2005, Journal of the American Society of Nephrology : JASN.
[24] G. Striker,et al. The glomerulosclerosis of aging in females: contribution of the proinflammatory mesangial cell phenotype to macrophage infiltration. , 2004, The American journal of pathology.
[25] J. Li,et al. Development of albuminuria and glomerular lesions in normoglycemic B6 recipients of db/db mice bone marrow: the role of mesangial cell progenitors. , 2004, Diabetes.
[26] J. He,et al. Advanced glycation endproduct (AGE) receptor 1 is a negative regulator of the inflammatory response to AGE in mesangial cells. , 2004, Proceedings of the National Academy of Sciences of the United States of America.
[27] E. Stadtman. Role of oxidant species in aging. , 2004, Current medicinal chemistry.
[28] J. Uribarri,et al. Glycoxidation and inflammation in renal failure patients. , 2004, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[29] S. Bandinelli,et al. Diverse Effect of Inflammatory Markers on Insulin Resistance and Insulin‐Resistance Syndrome in the Elderly , 2004, Journal of the American Geriatrics Society.
[30] J. Morrow,et al. Increased prevalence of oxidant stress and inflammation in patients with moderate to severe chronic kidney disease. , 2004, Kidney international.
[31] G. Striker,et al. Response to sex hormones differs in atherosclerosis-susceptible and -resistant mice. , 2003, American journal of physiology. Endocrinology and metabolism.
[32] G. Striker,et al. Glucose induces clonal selection and reversible dinucleotide repeat expansion in mesangial cells isolated from glomerulosclerosis-prone mice. , 2003, Diabetes.
[33] J. Gore,et al. Creatinine clearance and adverse hospital outcomes in patients with acute coronary syndromes: findings from the global registry of acute coronary events (GRACE) , 2003, Heart.
[34] E. Fisher,et al. Dietary glycotoxins promote diabetic atherosclerosis in apolipoprotein E-deficient mice. , 2003, Atherosclerosis.
[35] G. Striker,et al. Estrogen deficiency accelerates progression of glomerulosclerosis in susceptible mice. , 2003, The American journal of pathology.
[36] L. Ferrucci,et al. Insulin-like growth factor I and interleukin-6 contribute synergistically to disability and mortality in older women. , 2003, The Journal of clinical endocrinology and metabolism.
[37] G. Striker,et al. Resistance to glomerulosclerosis in B6 mice disappears after menopause. , 2003, The American journal of pathology.
[38] J. Uribarri,et al. Restriction of dietary glycotoxins reduces excessive advanced glycation end products in renal failure patients. , 2003, Journal of the American Society of Nephrology : JASN.
[39] H. Bosshart. Interleukin-3 receptors in Hodgkin's disease. , 2003, The American journal of pathology.
[40] G. Eknoyan,et al. Prevalence of chronic kidney disease and decreased kidney function in the adult US population: Third National Health and Nutrition Examination Survey. , 2003, American journal of kidney diseases : the official journal of the National Kidney Foundation.
[41] M. Peppa,et al. Inflammatory mediators are induced by dietary glycotoxins, a major risk factor for diabetic angiopathy , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[42] G. Striker,et al. Estrogen-related abnormalities in glomerulosclerosis-prone mice: reduced mesangial cell estrogen receptor expression and prosclerotic response to estrogens. , 2002, The American journal of pathology.
[43] G. Striker,et al. Upregulation of type I collagen by TGF-β in mesangial cells is blocked by PPARγ activation , 2002 .
[44] L. Ferrucci,et al. Peripheral Blood Markers of Inflammation Predict Mortality and Functional Decline in High‐Functioning Community‐Dwelling Older Persons , 2002, Journal of the American Geriatrics Society.
[45] G. Striker,et al. Reversibility of glucose-induced changes in mesangial cell extracellular matrix depends on the genetic background. , 2002, Diabetes.
[46] G. Striker,et al. Autocrine activation of the IGF-I signaling pathway in mesangial cells isolated from diabetic NOD mice. , 2002, Diabetes.
[47] G. Striker,et al. Glomerulosclerosis is transmitted by bone marrow-derived mesangial cell progenitors. , 2001, The Journal of clinical investigation.
[48] H. Vlassara. The AGE‐receptor in the pathogenesis of diabetic complications , 2001, Diabetes/metabolism research and reviews.
[49] A. Schmidt,et al. The multiligand receptor RAGE as a progression factor amplifying immune and inflammatory responses. , 2001, The Journal of clinical investigation.
[50] G. Striker,et al. Pentosan polysulfate prevents glomerular hypertension and structural injury despite persisting hypertension in 5/6 nephrectomy rats. , 2001, Journal of the American Society of Nephrology : JASN.
[51] T. Koschinsky,et al. Presence of Diabetic Complications in Type 1 Diabetic Patients Correlates with Low Expression of Mononuclear Cell AGE-Receptor-1 and Elevated Serum AGE , 2001, Molecular medicine.
[52] J. Himmelfarb,et al. Manifestations of Oxidant Stress in Uremia , 2000, Blood Purification.
[53] A. Schmidt,et al. The biology of the receptor for advanced glycation end products and its ligands. , 2000, Biochimica et biophysica acta.
[54] N. Holbrook,et al. Oxidants, oxidative stress and the biology of ageing , 2000, Nature.
[55] J. Baynes,et al. Glycoxidation and lipoxidation in atherogenesis. , 2000, Free radical biology & medicine.
[56] H. Cohen,et al. Associations of elevated interleukin-6 and C-reactive protein levels with mortality in the elderly. , 1999, The American journal of medicine.
[57] J. Baynes,et al. Role of oxidative stress in diabetic complications: a new perspective on an old paradigm. , 1999, Diabetes.
[58] M. Huijberts,et al. Breakers of advanced glycation end products restore large artery properties in experimental diabetes. , 1998, Proceedings of the National Academy of Sciences of the United States of America.
[59] C. Baylis,et al. The aging kidney: insights from experimental studies. , 1998, Journal of the American Society of Nephrology : JASN.
[60] R. Bucala,et al. Orally absorbed reactive glycation products (glycotoxins): an environmental risk factor in diabetic nephropathy. , 1997, Proceedings of the National Academy of Sciences of the United States of America.
[61] N. Hotta,et al. Rapid formation of advanced glycation end products by intermediate metabolites of glycolytic pathway and polyol pathway. , 1996, Biochemical and biophysical research communications.
[62] Paul J Thornalley. Pharmacology of methylglyoxal: formation, modification of proteins and nucleic acids, and enzymatic detoxification--a role in pathogenesis and antiproliferative chemotherapy. , 1996, General pharmacology.
[63] H. Vlassara,et al. Protein glycation in the kidney: role in diabetes and aging. , 1996, Kidney international.
[64] R. Bucala,et al. Prevention of cardiovascular and renal pathology of aging by the advanced glycation inhibitor aminoguanidine. , 1996, Proceedings of the National Academy of Sciences of the United States of America.
[65] A. Schmidt,et al. Cellular receptors for advanced glycation end products. Implications for induction of oxidant stress and cellular dysfunction in the pathogenesis of vascular lesions. , 1994, Arteriosclerosis and thrombosis : a journal of vascular biology.
[66] K. Kuwabara,et al. Receptor for advanced glycation end products (AGEs) has a central role in vessel wall interactions and gene activation in response to circulating AGE proteins. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[67] R. Bucala,et al. Lipid advanced glycosylation: pathway for lipid oxidation in vivo. , 1993, Proceedings of the National Academy of Sciences of the United States of America.
[68] N W Shock,et al. Longitudinal Studies on the Rate of Decline in Renal Function with Age , 1985, Journal of the American Geriatrics Society.