Correlation Between Beat‐to‐Beat QT Interval Variability and Impaired Left Ventricular Function in Patients with Previous Myocardial Infarction

Background: Beat‐to‐beat QT interval variability (QTV) is associated with sudden cardiac death and New York Heat Association functional class severity. We sought to evaluate the relationship between QTV and left ventricular (LV) function in patients with previous myocardial infarction (MI).

[1]  J. Sarma,et al.  Circadian rhythmicity of heart rate and QTc interval in diabetic autonomic neuropathy: implications for the mechanism of sudden death. , 1993, American heart journal.

[2]  C. Braun,et al.  Independent Autonomic Modulation of Sinus Node and Ventricular Myocardium in Healthy Young Men During Sleep , 2000, Journal of cardiovascular electrophysiology.

[3]  E. Erdmann,et al.  Alterations of K+ currents in isolated human ventricular myocytes from patients with terminal heart failure. , 1993, Circulation research.

[4]  A. Beckett,et al.  AKUFO AND IBARAPA. , 1965, Lancet.

[5]  S. Vanni,et al.  Cardiac Angiotensin II Formation in the Clinical Course of Heart Failure and Its Relationship With Left Ventricular Function , 2001, Circulation research.

[6]  Bramahn . Singh,et al.  Circadian and power spectral changes of RR and QT intervals during treatment of patients with angina pectoris with nadolol providing evidence for differential autonomic modulation of heart rate and ventricular repolarization. , 1994, The American journal of cardiology.

[7]  Ferdinand J. Venditti,et al.  Reduced Heart Rate Variability and Mortalit Risk in an Elderly Cohort: The Framingham Heart Study , 1994, Circulation.

[8]  J. Loeb,et al.  Beat-by-beat modulation of AV conduction. II. Autonomic neural mechanisms. , 1986, The American journal of physiology.

[9]  H Calkins,et al.  Beat‐to‐Beat Repolarization Lability Identifies Patients at Risk for Sudden Cardiac Death , 1998, Journal of cardiovascular electrophysiology.

[10]  W. C. Randall,et al.  Differential sympathetic regulation of automatic, conductile, and contractile tissue in dog heart. , 1988, The American journal of physiology.

[11]  I. Ninomiya,et al.  Differential regional responses of myocardial interstitial noradrenaline levels to coronary occlusion. , 1993, Cardiovascular research.

[12]  G R Hageman,et al.  Differential sympathetic-parasympathetic interactions in sinus node and AV junction. , 1986, The American journal of physiology.

[13]  H. Calkins,et al.  Beat-to-beat QT interval variability: novel evidence for repolarization lability in ischemic and nonischemic dilated cardiomyopathy. , 1997, Circulation.

[14]  G Baumann,et al.  Autonomic Effects of Dipyridamole Stress Testing on Frequency Distribution of RR and QT Interval Variability , 1998, Pacing and clinical electrophysiology : PACE.

[15]  D. Altman,et al.  STATISTICAL METHODS FOR ASSESSING AGREEMENT BETWEEN TWO METHODS OF CLINICAL MEASUREMENT , 1986, The Lancet.

[16]  D. Zipes,et al.  Changes in atrial and ventricular refractoriness and in atrioventricular nodal conduction produced by combinations of vagal and sympathetic stimulation that result in a constant spontaneous sinus cycle length. , 1987, Circulation research.