CTRP3 plays an important role in the development of collagen-induced arthritis in mice.

[1]  M. Ikawa,et al.  Production of mouse pups from germline transmission-failed knockout chimeras , 2013, Transgenic Research.

[2]  C. Hofmann,et al.  C1q/TNF‐related protein‐3 (CTRP‐3) is secreted by visceral adipose tissue and exerts antiinflammatory and antifibrotic effects in primary human colonic fibroblasts , 2011, Inflammatory bowel diseases.

[3]  Tetsuya S. Tanaka,et al.  Functional analysis of CTRP3/cartducin in Meckel’s cartilage and developing condylar cartilage in the fetal mouse mandible , 2011, Journal of anatomy.

[4]  Chih-Hsin Tang,et al.  Adiponectin increases MMP‐3 expression in human chondrocytes through adipor1 signaling pathway , 2011, Journal of cellular biochemistry.

[5]  C. López-Otín,et al.  Matrix metalloproteinase-8 deficiency increases joint inflammation and bone erosion in the K/BxN serum-transfer arthritis model , 2010, Arthritis research & therapy.

[6]  J. Schölmerich,et al.  C1q/TNF-related protein-3 represents a novel and endogenous lipopolysaccharide antagonist of the adipose tissue. , 2010, Endocrinology.

[7]  P. Peake,et al.  Factor H binds to the N-terminus of adiponectin and modulates complement activation. , 2010, Biochemical and biophysical research communications.

[8]  T. Kishimoto IL-6: from its discovery to clinical applications. , 2010, International immunology.

[9]  M. Feldmann,et al.  Role of cytokines in rheumatoid arthritis: an education in pathophysiology and therapeutics , 2008, Immunological reviews.

[10]  P. Peake,et al.  Adiponectin binds C1q and activates the classical pathway of complement. , 2008, Biochemical and biophysical research communications.

[11]  T. Yonezawa,et al.  Dcir deficiency causes development of autoimmune diseases in mice due to excess expansion of dendritic cells , 2008, Nature Medicine.

[12]  T. Miwa,et al.  Activator-specific requirement of properdin in the initiation and amplification of the alternative pathway complement. , 2008, Blood.

[13]  Richard O. Williams,et al.  Protocol for the induction of arthritis in C57BL/6 mice , 2008, Nature Protocols.

[14]  M. Gadjeva,et al.  C1q and its growing family. , 2007, Immunobiology.

[15]  Y. Iwakura,et al.  Identification of arthritis-related gene clusters by microarray analysis of two independent mouse models for rheumatoid arthritis , 2006, Arthritis research & therapy.

[16]  M. Takigawa,et al.  Cartducin, a paralog of Acrp30/adiponectin, is induced during chondrogenic differentiation and promotes proliferation of chondrogenic precursors and chondrocytes , 2006, Journal of cellular physiology.

[17]  J. Schölmerich,et al.  The adiponectin paralog CORS‐26 has anti‐inflammatory properties and is produced by human monocytic cells , 2005, FEBS letters.

[18]  A. Sjöholm,et al.  Functional analysis of the classical, alternative, and MBL pathways of the complement system: standardization and validation of a simple ELISA. , 2005, Journal of immunological methods.

[19]  J. Edwards,et al.  Sustained improvement in rheumatoid arthritis following a protocol designed to deplete B lymphocytes. , 2001, Rheumatology.

[20]  S. Nakae,et al.  Development of Chronic Inflammatory Arthropathy Resembling Rheumatoid Arthritis in Interleukin 1 Receptor Antagonist–Deficient Mice , 2000, The Journal of experimental medicine.

[21]  Masatoshi Suzuki,et al.  Production of Mice Deficient in Genes for Interleukin (IL)-1α, IL-1β, IL-1α/β, and IL-1 Receptor Antagonist Shows that IL-1β Is Crucial in Turpentine-induced Fever Development and Glucocorticoid Secretion , 1998, The Journal of experimental medicine.

[22]  al. et,et al.  Induction of inflammatory arthropathy resembling rheumatoid arthritis in mice transgenic for HTLV-I , 1991, Science.