Increased frequencies of HPRT mutant T lymphocytes in patients with Guillain-Barré syndrome and chronic inflammatory demyelinating polyneuropathy: further evidence for a role of T cells in the etiopathogenesis of peripheral demyelinating diseases
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[1] S. Sriram. Longitudinal study of frequency of HPRT mutant T cells in patients with multiple sclerosis , 1994, Neurology.
[2] P. Sfikakis,et al. Increased frequency of in vivo hprt gene-mutated T cells in the peripheral blood of patients with systemic sclerosis. , 1994, Annals of the rheumatic diseases.
[3] A. Steinberg,et al. Assessment of clinical parameters associated with increased frequency of mutant T cells in patients with systemic lupus erythematosus. , 1994, Arthritis and rheumatism.
[4] A. Steinberg,et al. Assessment of in vivo frequency of mutated T cells in patients with systemic lupus erythematosus , 1992, The Journal of experimental medicine.
[5] H. Hartung,et al. Serum interleukin‐2 concentrations in Guillain‐Barré syndrome and chronic idiopathic demyelinating polyradiculoneuropathy: Comparison with other neurological diseases of presumed immunopathogenesis , 1991, Annals of neurology.
[6] S. Brostoff,et al. P2 specific lymphocyte transformation in Guillain-Barré syndrome and chronic idiopathic demyelinating polyradiculoneuropathy , 1991, Journal of the Neurological Sciences.
[7] M. Honavar,et al. A clinicopathological study of the Guillain-Barré syndrome. Nine cases and literature review. , 1991, Brain : a journal of neurology.
[8] R J Albertini,et al. T cells responsive to myelin basic protein in patients with multiple sclerosis. , 1990, Science.
[9] H. Hartung,et al. T cell activation in Guillain‐Barré syndrome and in MS , 1990, Neurology.
[10] R. Hughes,et al. T lymphocyte activation antigens in Guillain-Barré syndrome and chronic idiopathic demyelinating polyradiculoneuropathy , 1989, Journal of Neuroimmunology.
[11] R. Albertini,et al. Refinement of a T-lymphocyte cloning assay to quantify the in vivo thioguanine-resistant mutant frequency in humans. , 1987, Mutagenesis.
[12] D. Pleasure,et al. Isolation of P2 protein–reactive T‐cell lines from human blood , 1986, Annals of neurology.
[13] R. Raper,et al. Macrophage procoagulant activity as a measure of cell-mediated immunity to P2 protein of peripheral nerves in the Guillain-Barré syndrome , 1985, Journal of Neuroimmunology.
[14] E. Alvord,et al. Sequence homology between certain viral proteins and proteins related to encephalomyelitis and neuritis. , 1985, Science.
[15] R. Fujinami,et al. Molecular mimicry in virus infection: crossreaction of measles virus phosphoprotein or of herpes simplex virus protein with human intermediate filaments. , 1983, Proceedings of the National Academy of Sciences of the United States of America.
[16] Kevin J. Trainor,et al. Measurement of in vivo mutations in human lymphocytes , 1983, Nature.
[17] D. Pleasure,et al. Immune reactions to P2 protein in human inflammatory demyelinative neuropathies , 1983, Neurology.
[18] R. Albertini,et al. T-cell cloning to detect the mutant 6-thioguanine-resistant lymphocytes present in human peripheral blood. , 1982, Proceedings of the National Academy of Sciences of the United States of America.
[19] J. Mcleod,et al. Chronic relapsing polyneuritis , 1976, Journal of the Neurological Sciences.
[20] P. Dyck,et al. Chronic inflammatory polyradiculoneuropathy. , 1975, Mayo Clinic proceedings.
[21] R. Adams,et al. The inflammatory lesion in idiopathic polyneuritis. Its role in pathogenesis. , 1969, Medicine.