Impaired CCR9/CCL25 signalling induced by inefficient dendritic cells contributes to intestinal immune imbalance in nonalcoholic steatohepatitis.

[1]  I. Sandrock,et al.  A Subset of CCL25-Induced Gut-Homing T Cells Affects Intestinal Immunity to Infection and Cancer , 2019, Front. Immunol..

[2]  P. Gérard,et al.  The links between the gut microbiome and non-alcoholic fatty liver disease (NAFLD) , 2019, Cellular and Molecular Life Sciences.

[3]  A. Stagg Intestinal Dendritic Cells in Health and Gut Inflammation , 2018, Front. Immunol..

[4]  Si Ming Man Inflammasomes in the gastrointestinal tract: infection, cancer and gut microbiota homeostasis , 2018, Nature Reviews Gastroenterology & Hepatology.

[5]  D. Soler,et al.  MLN3126, an antagonist of the chemokine receptor CCR9, ameliorates inflammation in a T cell mediated mouse colitis model , 2018, International immunopharmacology.

[6]  Zhenghuan Liu,et al.  The role of chemokine receptor 9/chemokine ligand 25 signaling: From immune cells to cancer cells , 2018, Oncology letters.

[7]  M. Thangaraju,et al.  Canonical Wnt Signaling in CD11c+ APCs Regulates Microbiota-Induced Inflammation and Immune Cell Homeostasis in the Colon , 2018, The Journal of Immunology.

[8]  Xiong Ma,et al.  The microbiome and autoimmunity: a paradigm from the gut–liver axis , 2018, Cellular & Molecular Immunology.

[9]  Ying Ding,et al.  Bone marrow lympho-myeloid malfunction in obesity requires precursor cell-autonomous TLR4 , 2018, Nature Communications.

[10]  Y. Saeys,et al.  TGFβR signalling controls CD103+CD11b+ dendritic cell development in the intestine , 2017, Nature Communications.

[11]  U. Yrlid,et al.  A reduced population of CD103(+)CD11b(+) dendritic cells has a limited impact on oral Salmonella infection. , 2016, Immunology letters.

[12]  Gordon F Heidkamp,et al.  Direct Delivery of Antigens to Dendritic Cells via Antibodies Specific for Endocytic Receptors as a Promising Strategy for Future Therapies , 2016, Vaccines.

[13]  Lawrence A. David,et al.  The severity of nonalcoholic fatty liver disease is associated with gut dysbiosis and shift in the metabolic function of the gut microbiota , 2016, Hepatology.

[14]  E. Butcher,et al.  Generation and transcriptional programming of intestinal dendritic cells: Essential role of retinoic acid , 2015, Mucosal Immunology.

[15]  Ting Zhao,et al.  Intestinal mucosal barrier dysfunction participates in the progress of nonalcoholic fatty liver disease. , 2015, International journal of clinical and experimental pathology.

[16]  Jonathan R. Brestoff,et al.  Immune Regulation of Metabolic Homeostasis in Health and Disease , 2015, Cell.

[17]  Jing Li,et al.  Dysbiosis gut microbiota associated with inflammation and impaired mucosal immune function in intestine of humans with non-alcoholic fatty liver disease , 2015, Scientific Reports.

[18]  Beiwen Zheng,et al.  Alterations of the human gut microbiome in liver cirrhosis , 2014, Nature.

[19]  R. Geha,et al.  CCL25/CCR9 Interactions Are Not Essential for Colitis Development but Are Required for Innate Immune Cell Protection from Chronic Experimental Murine Colitis , 2014, Inflammatory bowel diseases.

[20]  G. Núñez,et al.  Regulation of the immune system by the resident intestinal bacteria. , 2014, Gastroenterology.

[21]  Si-young Song,et al.  Retinoic acid prevents mesenteric lymph node dendritic cells from inducing IL-13-producing inflammatory Th2 cells , 2013, Mucosal Immunology.

[22]  F. Powrie,et al.  CD4+ T-cell subsets in intestinal inflammation , 2013, Immunological reviews.

[23]  B. Malissen,et al.  Resident and pro-inflammatory macrophages in the colon represent alternative context-dependent fates of the same Ly6Chi monocyte precursors , 2012, Mucosal Immunology.

[24]  A. Hart,et al.  Human Gut‐Specific Homeostatic Dendritic Cells Are Generated from Blood Precursors by the Gut Microenvironment , 2012, Inflammatory bowel diseases.

[25]  F. Powrie,et al.  Small intestinal CD103+ dendritic cells display unique functional properties that are conserved between mice and humans , 2008, The Journal of experimental medicine.