Mitochondrial nutrients improve immune dysfunction in the type 2 diabetic Goto-Kakizaki rats
暂无分享,去创建一个
Weili Shen | J. Hao | Zhongbo Liu | Cheng Luo | J. Long | Jiankang Liu | Jiejie Hao | Chuan Tian | Zhongbo Liu | Jinmin Ren | Cheng Luo | Jiangang Long | Edward Sharman | Jiankang Liu | W. Shen | E. Sharman | C. Tian | Jinmin Ren
[1] M. Belvisi,et al. Peroxisome proliferator-activated receptor gamma agonists as therapy for chronic airway inflammation. , 2006, European journal of pharmacology.
[2] C. Palmeira,et al. Alterations of liver mitochondrial bioenergetics in diabetic Goto-Kakizaki rats. , 1999, Metabolism: clinical and experimental.
[3] G. Steele,et al. Intracellular heterogeneity in mitochondrial membrane potentials revealed by a J-aggregate-forming lipophilic cation JC-1. , 1991, Proceedings of the National Academy of Sciences of the United States of America.
[4] T. Mosmann. Rapid colorimetric assay for cellular growth and survival: application to proliferation and cytotoxicity assays. , 1983, Journal of immunological methods.
[5] P. Oliveira,et al. Diabetes induces metabolic adaptations in rat liver mitochondria: role of coenzyme Q and cardiolipin contents. , 2003, Biochimica et biophysica acta.
[6] C. Palmeira,et al. Brain and liver mitochondria isolated from diabeticGoto‐Kakizaki rats show different susceptibility to induced oxidative stress , 2001, Diabetes/metabolism research and reviews.
[7] Jiankang Liu,et al. Stress, Aging, and Brain Oxidative Damage , 1999, Neurochemical Research.
[8] M. Kuwajima,et al. A role of nicotinamide-induced increase in pancreatic beta-cell mass on blood glucose control after discontinuation of the treatment in partially pancreatectomized OLETF rats. , 1998, Diabetes research and clinical practice.
[9] M. De la Fuente,et al. The immune system in the oxidative stress conditions of aging and hypertension: favorable effects of antioxidants and physical exercise. , 2005, Antioxidants & redox signaling.
[10] M. Gronski,et al. Death pathways in T cell homeostasis and their role in autoimmune diabetes. , 2006, The review of diabetic studies : RDS.
[11] Ken-ichi Isobe,et al. An inhibitor of mitochondrial complex I, rotenone, inactivates proteasome by oxidative modification and induces aggregation of oxidized proteins in SH‐SY5Y cells , 2003, Journal of neuroscience research.
[12] R. Hotchkiss,et al. Apoptosis and caspases regulate death and inflammation in sepsis , 2006, Nature Reviews Immunology.
[13] B. Ames,et al. (R)-alpha-lipoic acid protects retinal pigment epithelial cells from oxidative damage. , 2005, Investigative ophthalmology & visual science.
[14] G. Mingrone. Carnitine in Type 2 Diabetes , 2004, Annals of the New York Academy of Sciences.
[15] Jiankang Liu. The Effects and Mechanisms of Mitochondrial Nutrient α-Lipoic Acid on Improving Age-Associated Mitochondrial and Cognitive Dysfunction: An Overview , 2007, Neurochemical Research.
[16] B. Ames,et al. (R)-α-Lipoic Acid Protects Retinal Pigment Epithelial Cells from Oxidative Damage , 2005 .
[17] F. Russo,et al. Estrogenic regulation of cholesterol biosynthesis and cell growth in DLD-1 human colon cancer cells , 2005, Scandinavian journal of gastroenterology.
[18] C. Palmeira,et al. Higher efficiency of the liver phosphorylative system in diabetic Goto‐Kakizaki (GK) rats , 1999, FEBS letters.
[19] M. Mccarty,et al. Nutraceutical resources for diabetes prevention--an update. , 2005, Medical hypotheses.
[20] D. Chan. Mitochondria: Dynamic Organelles in Disease, Aging, and Development , 2006, Cell.
[21] J. Satoh,et al. Improvement of glucose tolerance with immunomodulators in type 2 diabetic animals , 1996, Biotherapy.
[22] B. Ames,et al. Reducing mitochondrial decay with mitochondrial nutrients to delay and treat cognitive dysfunction, Alzheimer's disease, and Parkinson's disease , 2005, Nutritional neuroscience.
[23] David W. Killilea,et al. Age-associated mitochondrial oxidative decay: Improvement of carnitine acetyltransferase substrate-binding affinity and activity in brain by feeding old rats acetyl-l- carnitine and/or R-α-lipoic acid , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[24] A. Abbas,et al. Cellular and Molecular Immunology , 1991 .
[25] C. Cotman,et al. R-α-Lipoic acid and acetyl-l-carnitine complementarily promote mitochondrial biogenesis in murine 3T3-L1 adipocytes , 2007, Diabetologia.
[26] N. Wiernsperger,et al. Metformin delays the manifestation of diabetes and vascular dysfunction in Goto–Kakizaki rats by reduction of mitochondrial oxidative stress , 2006, Diabetes/metabolism research and reviews.
[27] M. S. Santos,et al. Diabetes and mitochondrial bioenergetics: Alterations with age , 2003, Journal of biochemical and molecular toxicology.
[28] K. Yagi,et al. Assay for lipid peroxides in animal tissues by thiobarbituric acid reaction. , 1979, Analytical biochemistry.
[29] E. Donadi,et al. Effect of metabolic control on the in vitro proliferation of peripheral blood mononuclear cells in type 1 and type 2 diabetic patients. , 2006, Sao Paulo medical journal = Revista paulista de medicina.
[30] H. Kaneto,et al. Involvement of oxidative stress in the pathogenesis of diabetes. , 2006, Antioxidants & redox signaling.
[31] F. Chang,et al. Decreased cell-mediated immunity in patients with non-insulin-dependent diabetes mellitus. , 1995, Diabetes research and clinical practice.
[32] B. Ames,et al. Acetyl‐L‐carnitine and α‐lipoic acid supplementation of aged beagle dogs improves learning in two landmark discrimination tests , 2007, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[33] James C Bartholomew,et al. Feeding acetyl-l-carnitine and lipoic acid to old rats significantly improves metabolic function while decreasing oxidative stress , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[34] B. Portha. Programmed disorders of β‐cell development and function as one cause for type 2 diabetes? The GK rat paradigm , 2005, Diabetes/metabolism research and reviews.
[35] S. Khandelwal,et al. Role of oxidative stress and apoptosis in cadmium induced thymic atrophy and splenomegaly in mice. , 2007, Toxicology letters.
[36] K. Maiese,et al. Oxidative stress biology and cell injury during type 1 and type 2 diabetes mellitus. , 2007, Current neurovascular research.
[37] B. Ames,et al. Oxidative damage and mitochondrial decay in aging. , 1994, Proceedings of the National Academy of Sciences of the United States of America.
[38] A. De Gaetano,et al. Acetyl-L-carnitine infusion increases glucose disposal in type 2 diabetic patients. , 2000, Metabolism: clinical and experimental.
[39] M. Hanefeld,et al. Treatment of symptomatic diabetic peripheral neuropathy with the anti-oxidant alpha-lipoic acid. A 3-week multicentre randomized controlled trial (ALADIN Study). , 1995, Diabetologia.
[40] S. Riley,et al. The quest for a model of type II diabetes with nephropathy: the Goto Kakizaki rat. , 2004, Journal of nephrology.
[41] S. Yamamoto,et al. Exercise training restores decreased cellular immune functions in obese Zucker rats. , 1998, Journal of applied physiology.
[42] W. Habig,et al. Glutathione S-transferase A. A novel kinetic mechanism in which the major reaction pathway depends on substrate concentration. , 1974, The Journal of biological chemistry.
[43] J. Fanzo,et al. p53 protein and p21 mRNA levels and caspase-3 activity are altered by zinc status in aortic endothelial cells. , 2002, American journal of physiology. Cell physiology.
[44] L. Packer,et al. Cytosolic and Mitochondrial Systems for NADH- and NADPH-dependent Reduction of α-Lipoic Acid , 1997 .
[45] M. Kirsch,et al. NAD(P)H, a directly operating antioxidant? , 2001, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.
[46] E. Stadtman,et al. Carbonyl assays for determination of oxidatively modified proteins. , 1994, Methods in enzymology.
[47] C. Östenson,et al. Diabetic type II Goto-Kakizaki rats show progressively decreasing exploratory activity and learning impairments in fixed and progressive ratios of a lever-press task , 2007, Behavioural Brain Research.
[48] B. Ames,et al. Memory loss in old rats is associated with brain mitochondrial decay and RNA/DNA oxidation: Partial reversal by feeding acetyl-l-carnitine and/or R-α-lipoic acid , 2002, Proceedings of the National Academy of Sciences of the United States of America.
[49] R. Mccaman,et al. Carnitine acetyltransferase in nervous tissue. , 1966, The Journal of biological chemistry.
[50] J. Hayes,et al. Glutathione S‐transferases , 2002 .